Dysregulated gene expression networks in human acute myelogenous leukemia stem cells.
about
The serotonin transporter promoter variant (5-HTTLPR), stress, and depression meta-analysis revisited: evidence of genetic moderationAberrant Wnt Signaling in LeukemiaRegulation of ovarian cancer stem cells or tumor-initiating cellsInternal Tandem Duplication in FLT3 Attenuates Proliferation and Regulates Resistance to the FLT3 Inhibitor AC220 by Modulating p21Cdkn1a and Pbx1 in Hematopoietic CellsNOTCH1 signaling promotes human T-cell acute lymphoblastic leukemia initiating cell regeneration in supportive nichesGene Expression Commons: an open platform for absolute gene expression profilingHematopoietic stem cell: self-renewal versus differentiationStem cells are units of natural selection for tissue formation, for germline development, and in cancer developmentMolecular signatures of quiescent, mobilized and leukemia-initiating hematopoietic stem cells.Cancer reduces transcriptome specialization.Re-adapting T cells for cancer therapy: from mouse models to clinical trials.Independent component analysis: mining microarray data for fundamental human gene expression modulesIdentifying dysfunctional crosstalk of pathways in various regions of Alzheimer's disease brainsClonal evolution of preleukemic hematopoietic stem cells precedes human acute myeloid leukemia.NOTCH inhibits osteoblast formation in inflammatory arthritis via noncanonical NF-κB.The Apc(min) mouse has altered hematopoietic stem cell function and provides a model for MPD/MDS.Alignment of gene expression profiles from test samples against a reference database: New method for context-specific interpretation of microarray data.Association of a leukemic stem cell gene expression signature with clinical outcomes in acute myeloid leukemia.Identification and targeting of cancer stem cells.A network-based approach to prioritize results from genome-wide association studies.Imprinted genes that regulate early mammalian growth are coexpressed in somatic stem cells.Decreased SFRP2 expression is associated with intermediate and poor karyotypes in de novo acute myeloid leukemiaPeripheral T-lymphocytes express WNT7A and its restoration in leukemia-derived lymphoblasts inhibits cell proliferation.Identification and targeting leukemia stem cells: The path to the cure for acute myeloid leukemia.Cyclin-A1 represents a new immunogenic targetable antigen expressed in acute myeloid leukemia stem cells with characteristics of a cancer-testis antigenMetaMapp: mapping and visualizing metabolomic data by integrating information from biochemical pathways and chemical and mass spectral similarity.Requirement for ssbp2 in hematopoietic stem cell maintenance and stress responseIdentification of therapeutic targets for quiescent, chemotherapy-resistant human leukemia stem cells.Survivin selectively modulates genes deregulated in human leukemia stem cells.Concise review: preleukemic stem cells: molecular biology and clinical implications of the precursors to leukemia stem cellsReduced NR4A gene dosage leads to mixed myelodysplastic/myeloproliferative neoplasms in mice.Prospective separation of normal and leukemic stem cells based on differential expression of TIM3, a human acute myeloid leukemia stem cell markerHeterogeneous sensitivity of human acute myeloid leukemia to β-catenin down-modulation.Proteomic profiling identifies distinct protein patterns in acute myelogenous leukemia CD34+CD38- stem-like cells.Dissecting the oncogenic and tumorigenic potential of differentiated human induced pluripotent stem cells and human embryonic stem cells.Differential niche and Wnt requirements during acute myeloid leukemia progressionIL8-CXCR2 pathway inhibition as a therapeutic strategy against MDS and AML stem cellsIdentification of epigenetic modifications that contribute to pathogenesis in therapy-related AML: Effective integration of genome-wide histone modification with transcriptional profiles.Genome Wide Mapping of NR4A Binding Reveals Cooperativity with ETS Factors to Promote Epigenetic Activation of Distal Enhancers in Acute Myeloid Leukemia CellsFunctional inhibition of osteoblastic cells in an in vivo mouse model of myeloid leukemia
P2860
Q24289552-C1D2A5A1-00D4-4CFB-86F5-93A4EAFA18C4Q26738411-68368ABF-67B9-4EC7-B617-7A71327357F0Q26853096-A0AB1D54-57A0-4E03-89F5-50CBA9B4681AQ28552425-A10507A7-EBFF-4A03-93A6-8A6A4EEF279DQ28727674-43096BD7-02BD-4A60-9E1E-C2A5ED9BDDE4Q28729064-31878D2A-3BF4-4767-9104-F78CCEC7BCCDQ29396498-4CFBD762-CDFD-46D5-9A6F-F5B4CB12232CQ30659198-B1A9EE19-F516-467E-B4C6-D5E005E71319Q33526322-8C43382E-ACB5-4CCE-B80B-FAAD55FC1575Q33573483-60227F49-33DC-4974-B2F7-53F81925F0A6Q33588711-082758B2-502C-46C4-83E1-236A85F3D145Q33629605-667A53F5-D08D-43DF-A9F8-614A222FA19AQ33692288-C194E207-9501-4DEC-993A-0C5F9952E342Q33711497-C429E0F9-7736-480A-84CC-F6AAD51A03C6Q33808220-61DC4D7F-56E9-499A-845E-5D5D04020B3AQ33839824-8F5E0627-BC7D-4D46-AD67-728D04C62521Q33859024-9F255D5E-9690-44DB-A318-96B5BDD0E13FQ33873844-27D3F9AE-060E-48C2-B302-812A396DAED1Q33921460-94CF7C76-682B-42AC-BFF2-4F3959E580F8Q34018953-65FF6241-E026-4D52-BC4F-F2DFD953F9FCQ34062409-320A59A1-C0E5-478C-9AB6-612569D4BB20Q34121336-31C25475-F485-43B5-9D08-E29325ECE66BQ34153063-C12AD4C8-86B1-4D29-8891-D0AFDFAA1D5DQ34232877-9204C897-33E1-4AEB-A7B7-4761E564C922Q34243434-7B049C6F-B606-40ED-8927-73AB0CC03D74Q34271370-DBC160A3-CCB7-4EBD-B0D4-3D88455EFEA3Q34364759-75C75FC2-1C4C-4C96-96AE-6615931975F8Q34421127-4A07D2E1-0C96-4809-9CE3-251E559A259DQ34495409-3911D5EA-E23B-4F75-A33C-1B2BFC2EED34Q34647637-CCAED758-6744-4222-9A54-22EA1290904DQ34707445-4E4BCD5C-255F-4F40-BD1C-67919B2C16CBQ34720554-6DC9577E-047F-4051-8275-FA3BBDAD809EQ34905557-BDB6F38B-A91F-4C8E-A639-DC1986D3802FQ35040064-72D57F14-AC03-45B4-A2FA-40F17AA0AA1BQ35112764-736C8D2B-F49B-4E12-8B31-27DD437E44F5Q35212397-61FE87FE-52A8-4714-9FCC-D658DE3FBE94Q35607293-B50D79D9-1E84-482D-9F5C-97823B756E77Q35653085-F445FA7F-8F39-478F-B8B8-938BFE12E182Q35944651-B2D275B7-3453-4CDA-8CB2-0CEFE09211E2Q36062301-14D45FCE-6B73-4CF4-9F88-5926D50608B4
P2860
Dysregulated gene expression networks in human acute myelogenous leukemia stem cells.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 13 February 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Dysregulated gene expression networks in human acute myelogenous leukemia stem cells.
@en
Dysregulated gene expression networks in human acute myelogenous leukemia stem cells.
@nl
type
label
Dysregulated gene expression networks in human acute myelogenous leukemia stem cells.
@en
Dysregulated gene expression networks in human acute myelogenous leukemia stem cells.
@nl
prefLabel
Dysregulated gene expression networks in human acute myelogenous leukemia stem cells.
@en
Dysregulated gene expression networks in human acute myelogenous leukemia stem cells.
@nl
P2093
P2860
P356
P1476
Dysregulated gene expression networks in human acute myelogenous leukemia stem cells.
@en
P2093
Irving L Weissman
Jung-Hsien Chiang
Michael F Clarke
Michael W Becker
Qiang Tian
Ravindra Majeti
Tsung-Lu Michael Lee
Xiaowei Yan
P2860
P304
P356
10.1073/PNAS.0900089106
P407
P50
P577
2009-02-13T00:00:00Z