A wild and an attenuated strain of Francisella tularensis differ in susceptibility to hypochlorous acid: a possible explanation of their different handling by polymorphonuclear leukocytes.
about
TularemiaMultiple mechanisms of NADPH oxidase inhibition by type A and type B Francisella tularensis.Neutrophils are critical for host defense against primary infection with the facultative intracellular bacterium Francisella tularensis in mice and participate in defense against reinfectionTransfer of immunity against lethal murine Francisella infection by specific antibody depends on host gamma interferon and T cells.In vitro susceptibility of fungi to killing by neutrophil granulocytes discriminates between primary pathogenicity and opportunism.Growth of Francisella tularensis LVS in macrophages: the acidic intracellular compartment provides essential iron required for growthFrancisella tularensis LVS evades killing by human neutrophils via inhibition of the respiratory burst and phagosome escapeResistance of Francisella tularensis strains against reactive nitrogen and oxygen species with special reference to the role of KatG.Increased synthesis of DnaK, GroEL, and GroES homologs by Francisella tularensis LVS in response to heat and hydrogen peroxideFrancisella tularensis: taxonomy, genetics, and Immunopathogenesis of a potential agent of biowarfare.Initial delay in the immune response to Francisella tularensis is followed by hypercytokinemia characteristic of severe sepsis and correlating with upregulation and release of damage-associated molecular patterns.Activation of macrophages for destruction of Francisella tularensis: identification of cytokines, effector cells, and effector molecules.A capsule-deficient mutant of Francisella tularensis LVS exhibits enhanced sensitivity to killing by serum but diminished sensitivity to killing by polymorphonuclear leukocytes.Distinct roles of reactive nitrogen and oxygen species to control infection with the facultative intracellular bacterium Francisella tularensis.Basis for the failure of Francisella tularensis lipopolysaccharide to prime human polymorphonuclear leukocytes.Multifaceted effects of Francisella tularensis on human neutrophil function and lifespan.An attenuated strain of the facultative intracellular bacterium Francisella tularensis can escape the phagosome of monocytic cellsAnaerobic phagocytosis, killing, and degradation of Streptococcus pneumoniae by human peripheral blood leukocytes.
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P2860
A wild and an attenuated strain of Francisella tularensis differ in susceptibility to hypochlorous acid: a possible explanation of their different handling by polymorphonuclear leukocytes.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on February 1984
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
A wild and an attenuated strai ...... polymorphonuclear leukocytes.
@en
A wild and an attenuated strai ...... polymorphonuclear leukocytes.
@nl
type
label
A wild and an attenuated strai ...... polymorphonuclear leukocytes.
@en
A wild and an attenuated strai ...... polymorphonuclear leukocytes.
@nl
prefLabel
A wild and an attenuated strai ...... polymorphonuclear leukocytes.
@en
A wild and an attenuated strai ...... polymorphonuclear leukocytes.
@nl
P2093
P2860
P1476
A wild and an attenuated strai ...... y polymorphonuclear leukocytes
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P2093
P2860
P304
P407
P577
1984-02-01T00:00:00Z