Recognition of DNA double strand breaks by the BRCA1 tumor suppressor network.
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BRCA1 is an essential regulator of heart function and survival following myocardial infarctionThe Rap80-BRCC36 de-ubiquitinating enzyme complex antagonizes RNF8-Ubc13-dependent ubiquitination events at DNA double strand breaksMERIT40 facilitates BRCA1 localization and DNA damage repairHigh-definition DNA methylation profiles from breast and ovarian carcinoma cell lines with differing doxorubicin resistanceHemizygosity for Atm and Brca1 influence the balance between cell transformation and apoptosis.Mutation screening of the MERIT40 gene encoding a novel BRCA1 and RAP80 interacting protein in breast cancer familiesIdentification of differentially methylated BRCA1 and CRISP2 DNA regions as blood surrogate markers for cardiovascular diseaseDifferential regulation of JAMM domain deubiquitinating enzyme activity within the RAP80 complex.Tumor characteristics as an analytic tool for classifying genetic variants of uncertain clinical significance.BRCA1 pathway function in basal-like breast cancer cells.Systematic screening reveals a role for BRCA1 in the response to transcription-associated DNA damageBRCC36A is epistatic to BRCA1 in DNA crosslink repair and homologous recombination in Arabidopsis thalianaUnraveling the chromosome 17 patterns of FISH in interphase nuclei: an in-depth analysis of the HER2 amplicon and chromosome 17 centromere by karyotyping, FISH and M-FISH in breast cancer cells.ABRAXAS (FAM175A) and Breast Cancer Susceptibility: No Evidence of Association in the Breast Cancer Family Registry.Effect of EME1 exon variant Ile350Thr on risk and early onset of breast cancer in southern Chinese women.Familial breast cancer screening reveals an alteration in the RAP80 UIM domain that impairs DNA damage response functionModification of ovarian cancer risk by BRCA1/2-interacting genes in a multicenter cohort of BRCA1/2 mutation carriers.Breast cancer-associated Abraxas mutation disrupts nuclear localization and DNA damage response functions.The ubiquitin landscape at DNA double-strand breaks.Histone tails: Directing the chromatin response to DNA damageLinks between genome integrity and BRCA1 tumor suppression.Mutations in a gene encoding a midbody protein in binucleated Reed-Sternberg cells of Hodgkin lymphomaBRCA1 and CtIP suppress long-tract gene conversion between sister chromatids.BRCA1 gene therapy reduces systemic inflammatory response and multiple organ failure and improves survival in experimental sepsis.Reactive oxygen species-dependent down-regulation of tumor suppressor genes PTEN, USP28, DRAM, TIGAR, and CYLD under oxidative stress.
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P2860
Recognition of DNA double strand breaks by the BRCA1 tumor suppressor network.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 28 March 2008
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Recognition of DNA double strand breaks by the BRCA1 tumor suppressor network.
@en
Recognition of DNA double strand breaks by the BRCA1 tumor suppressor network.
@nl
type
label
Recognition of DNA double strand breaks by the BRCA1 tumor suppressor network.
@en
Recognition of DNA double strand breaks by the BRCA1 tumor suppressor network.
@nl
prefLabel
Recognition of DNA double strand breaks by the BRCA1 tumor suppressor network.
@en
Recognition of DNA double strand breaks by the BRCA1 tumor suppressor network.
@nl
P2860
P1433
P1476
Recognition of DNA double strand breaks by the BRCA1 tumor suppressor network.
@en
P2093
Roger A Greenberg
P2860
P2888
P304
P356
10.1007/S00412-008-0154-8
P577
2008-03-28T00:00:00Z