IL-17 and therapeutic kynurenines in pathogenic inflammation to fungi.
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Fungal allergy in asthma-state of the art and research needsAntifungal Th Immunity: Growing up in FamilyThe Role of the Microbial Metabolites Including Tryptophan Catabolites and Short Chain Fatty Acids in the Pathophysiology of Immune-Inflammatory and Neuroimmune DiseaseAryl hydrocarbon receptor negatively regulates dendritic cell immunogenicity via a kynurenine-dependent mechanismContribution of Bordetella filamentous hemagglutinin and adenylate cyclase toxin to suppression and evasion of interleukin-17-mediated inflammation.Cryptococcus neoformans variants generated by phenotypic switching differ in virulence through effects on macrophage activation.Role of IL-17A on resolution of pulmonary C. neoformans infectionTryptophan/kynurenine metabolism in human leukocytes is independent of superoxide and is fully maintained in chronic granulomatous disease.M. tuberculosis induces potent activation of IDO-1, but this is not essential for the immunological control of infection.Indoleamine 2,3 dioxygenase and metabolic control of immune responses.IDO upregulates regulatory T cells via tryptophan catabolite and suppresses encephalitogenic T cell responses in experimental autoimmune encephalomyelitisDepletion of neutrophils in a protective model of pulmonary cryptococcosis results in increased IL-17A production by γδ T cells.Therapy of experimental type 1 diabetes by isolated Sertoli cell xenografts alone.Kynurenine 3-monooxygenase inhibition in blood ameliorates neurodegeneration.Indoleamine 2,3-dioxygenase controls fungal loads and immunity in Paracoccidioidomicosis but is more important to susceptible than resistant hosts.Opposing biological functions of tryptophan catabolizing enzymes during intracellular infection.Interleukin-17 drives pulmonary eosinophilia following repeated exposure to Aspergillus fumigatus conidia.Indoleamine 2,3-dioxygenase and metabolites protect murine lung allografts and impair the calcium mobilization of T cells.Phenotypic switching in Cryptococcus neoformans contributes to virulence by changing the immunological host responseToll-like receptor 9 modulates immune responses to Aspergillus fumigatus conidia in immunodeficient and allergic mice.SOCS3 drives proteasomal degradation of indoleamine 2,3-dioxygenase (IDO) and antagonizes IDO-dependent tolerogenesisThe Role of Interleukin-23 in the Early Development of Emphysema in HIV1(+) Smokers.Hypothetical review: thymic aberrations and type-I interferons; attempts to deduce autoimmunizing mechanisms from unexpected clues in monogenic and paraneoplastic syndromes.Interleukins 17 and 23 influence the host response to Histoplasma capsulatumInterplay between effector Th17 and regulatory T cells.Generation of mesenchymal stromal cells in the presence of platelet lysate: a phenotypic and functional comparison of umbilical cord blood- and bone marrow-derived progenitors.Manipulation of indoleamine 2,3 dioxygenase; a novel therapeutic target for treatment of diseases.Identification of cinnabarinic acid as a novel endogenous aryl hydrocarbon receptor ligand that drives IL-22 production.The biocontaminants and complexity of damp indoor spaces: more than what meets the eyes.Non-hematopoietic cells contribute to protective tolerance to Aspergillus fumigatus via a TRIF pathway converging on IDO.Variability of phenotypic traits in Cryptococcus varieties and species and the resulting implications for pathogenesis.Innate and adaptive antifungal immune responses: partners on an equal footing.Regulatory T cells in HIV infection: can immunotherapy regulate the regulator?Learning from other diseases: protection and pathology in chronic fungal infections.Redox Properties of Tryptophan Metabolism and the Concept of Tryptophan Use in Pregnancy.Th17/Treg imbalance in murine cystic fibrosis is linked to indoleamine 2,3-dioxygenase deficiency but corrected by kynurenines.Immunity and tolerance to fungi in hematopoietic transplantation: principles and perspectives.Commensal Propionibacterium strain UF1 mitigates intestinal inflammation via Th17 cell regulation.Toll-like receptor 4 signaling leads to severe fungal infection associated with enhanced proinflammatory immunity and impaired expansion of regulatory T cells.Anti-Aspergillus human host defence relies on type 1 T helper (Th1), rather than type 17 T helper (Th17), cellular immunity.
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IL-17 and therapeutic kynurenines in pathogenic inflammation to fungi.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on April 2008
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
IL-17 and therapeutic kynurenines in pathogenic inflammation to fungi.
@en
IL-17 and therapeutic kynurenines in pathogenic inflammation to fungi.
@nl
type
label
IL-17 and therapeutic kynurenines in pathogenic inflammation to fungi.
@en
IL-17 and therapeutic kynurenines in pathogenic inflammation to fungi.
@nl
prefLabel
IL-17 and therapeutic kynurenines in pathogenic inflammation to fungi.
@en
IL-17 and therapeutic kynurenines in pathogenic inflammation to fungi.
@nl
P50
P1476
IL-17 and therapeutic kynurenines in pathogenic inflammation to fungi.
@en
P2093
Francesca Fallarino
Paolo Puccetti
P304
P356
10.4049/JIMMUNOL.180.8.5157
P407
P577
2008-04-01T00:00:00Z