Neuronal LR11/sorLA expression is reduced in mild cognitive impairment - Wikidata - awgldk - TriplyDB

Neuronal LR11/sorLA expression is reduced in mild cognitive impairment

Neuronal LR11/sorLA expression is reduced in mild cognitive impairment

http://www.wikidata.org/entity/Q37158949

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Rho kinase II phosphorylation of the lipoprotein receptor LR11/SORLA alters amyloid-beta production GGA1-mediated endocytic traffic of LR11/SorLA alters APP intracellular distribution and amyloid-β production Apolipoprotein E receptors in the nervous system Mild cognitive impairment: pathology and mechanisms.Trafficking regulation of proteins in Alzheimer's disease SORL1 Gene is Associated with the Conversion from Mild Cognitive Impairment to Alzheimer's Disease Loss of LR11/SORLA enhances early pathology in a mouse model of amyloidosis: evidence for a proximal role in Alzheimer's disease Omega-3 fatty acids and dementia.U1 small nuclear ribonucleoproteins (snRNPs) aggregate in Alzheimer's disease due to autosomal dominant genetic mutations and trisomy 21.Abnormal gephyrin immunoreactivity associated with Alzheimer disease pathologic changes.Genome-wide association reveals genetic effects on human Aβ42 and τ protein levels in cerebrospinal fluids: a case control study.Use of genetic variation as biomarkers for mild cognitive impairment and progression of mild cognitive impairment to dementia.Neuronal LR11 expression does not differentiate between clinically-defined Alzheimer's disease and control brains Alzheimer's disease risk genes and mechanisms of disease pathogenesis The role of apolipoprotein E in Alzheimer's disease.Meta-analysis of the association between variants in SORL1 and Alzheimer disease.DHA diet reduces AD pathology in young APPswe/PS1 Delta E9 transgenic mice: possible gender effects Genome-Wide Analyses of Working-Memory Ability: A Review.The role of the retromer complex in aging-related neurodegeneration: a molecular and genomic review.Diabetes-associated SorCS1 regulates Alzheimer's amyloid-beta metabolism: evidence for involvement of SorL1 and the retromer complex.Unexpected partial correction of metabolic and behavioral phenotypes of Alzheimer's APP/PSEN1 mice by gene targeting of diabetes/Alzheimer's-related Sorcs1 Vps10 family proteins and the retromer complex in aging-related neurodegeneration and diabetes.Impact of SORL1 single nucleotide polymorphisms on Alzheimer's disease cerebrospinal fluid markers Alzheimer's disease: advances in trafficking Genome-wide association study implicates a chromosome 12 risk locus for late-onset Alzheimer disease.Neuronal protein trafficking associated with Alzheimer disease: from APP and BACE1 to glutamate receptors Linking Abeta and tau in late-onset Alzheimer's disease: a dual pathway hypothesis Reduction of SorLA/LR11, a sorting protein limiting beta-amyloid production, in Alzheimer disease cerebrospinal fluid.Impact of SORL1 genetic variations on MRI markers in non-demented elders.Chipping away at diagnostics for neurodegenerative diseases Expression of SORL1 and a novel SORL1 splice variant in normal and Alzheimers disease brain.The SORL1 gene and convergent neural risk for Alzheimer's disease across the human lifespan.Advances and perspectives from genetic research: development of biological markers in Alzheimer's disease.Gene expression profiling and therapeutic interventions in neurodegenerative diseases: a comprehensive study on potentiality and limits.The Role of SORL1 in Alzheimer's Disease.SorLA complement-type repeat domains protect the amyloid precursor protein against processing.Clearance of cerebral Aβ in Alzheimer's disease: reassessing the role of microglia and monocytes.Unraveling the genes implicated in Alzheimer's disease.Retrieval of the Alzheimer's amyloid precursor protein from the endosome to the TGN is S655 phosphorylation state-dependent and retromer-mediated.SorLA in glia: shared subcellular distribution patterns with caveolin-1

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Neuronal LR11/sorLA expression is reduced in mild cognitive impairment

http://www.wikidata.org/entity/Q37158949

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

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scientific article published on December 2007

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Neuronal LR11/sorLA expression is reduced in mild cognitive impairment

@en

Neuronal LR11/sorLA expression is reduced in mild cognitive impairment.

@nl

type

label

Neuronal LR11/sorLA expression is reduced in mild cognitive impairment

@en

Neuronal LR11/sorLA expression is reduced in mild cognitive impairment.

@nl

prefLabel

Neuronal LR11/sorLA expression is reduced in mild cognitive impairment

@en

Neuronal LR11/sorLA expression is reduced in mild cognitive impairment.

@nl

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Annals of Neurology

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Neuronal LR11/sorLA expression is reduced in mild cognitive impairment

@en

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Allan I Levey

http://www.w3.org/2001/XMLSchema#string

Elliott J Mufson

http://www.w3.org/2001/XMLSchema#string

Howard D Rees

http://www.w3.org/2001/XMLSchema#string

James J Lah

http://www.w3.org/2001/XMLSchema#string

Joanne Wuu

http://www.w3.org/2001/XMLSchema#string

Kristen L Sager

http://www.w3.org/2001/XMLSchema#string

Marla Gearing

http://www.w3.org/2001/XMLSchema#string

Susan E Leurgans

http://www.w3.org/2001/XMLSchema#string

P2860

Neuronal sorting protein-related receptor sorLA/LR11 regulates processing of the amyloid precursor protein.

The neuronal sortilin-related receptor SORL1 is genetically associated with Alzheimer disease

“Mini-mental state”

Neuropathological stageing of Alzheimer-related changes

The amyloid hypothesis of Alzheimer's disease: progress and problems on the road to therapeutics

Reelin and ApoE receptors cooperate to enhance hippocampal synaptic plasticity and learning

Neuropathologic features of amnestic mild cognitive impairment

Loss of apolipoprotein E receptor LR11 in Alzheimer disease

The lipoprotein receptor LR11 regulates amyloid beta production and amyloid precursor protein traffic in endosomal compartments

Physical basis of cognitive alterations in Alzheimer's disease: synapse loss is the major correlate of cognitive impairment

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640-647

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scholarly article

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10.1002/ANA.21190

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6

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62

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2007-12-01T00:00:00Z

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6118223

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17721864

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2669325

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