Linking Abeta and tau in late-onset Alzheimer's disease: a dual pathway hypothesis - Wikidata - awgldk - TriplyDB

Linking Abeta and tau in late-onset Alzheimer's disease: a dual pathway hypothesis

Linking Abeta and tau in late-onset Alzheimer's disease: a dual pathway hypothesis

http://www.wikidata.org/entity/Q37336139

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Phosphoprotein phosphatase 2A: a novel druggable target for Alzheimer's disease A/T/N: An unbiased descriptive classification scheme for Alzheimer disease biomarkers The amyloid hypothesis of Alzheimer's disease at 25 years Role of the Retromer Complex in Neurodegenerative Diseases Tau Oligomers: The Toxic Player at Synapses in Alzheimer's Disease Alzheimer's Disease: Mechanism and Approach to Cell Therapy Targeting the proper amyloid-beta neuronal toxins: a path forward for Alzheimer's disease immunotherapeutics Effect of the inhaled anesthetics isoflurane, sevoflurane and desflurane on the neuropathogenesis of Alzheimer's disease (review)APP intracellular domain impairs adult neurogenesis in transgenic mice by inducing neuroinflammation Tau Protein Mediates APP Intracellular Domain (AICD)-Induced Alzheimer's-Like Pathological Features in Mice Immunotherapeutic approaches for Alzheimer's disease eIF4A inhibition allows translational regulation of mRNAs encoding proteins involved in Alzheimer's disease Tracking pathophysiological processes in Alzheimer's disease: an updated hypothetical model of dynamic biomarkers PET amyloid-beta imaging in preclinical Alzheimer's disease Intranasal BMP9 Ameliorates Alzheimer Disease-Like Pathology and Cognitive Deficits in APP/PS1 Transgenic Mice.Allele-specific RNAi mitigates phenotypic progression in a transgenic model of Alzheimer's disease.Apolipoprotein E4 causes age- and Tau-dependent impairment of GABAergic interneurons, leading to learning and memory deficits in mice.Possible modification of Alzheimer's disease by statins in midlife: interactions with genetic and non-genetic risk factors Propagation of pathology through brain networks in neurodegenerative diseases: from molecules to clinical phenotypes.Rates of β-amyloid accumulation are independent of hippocampal neurodegeneration.Alzheimer Abeta peptide induces chromosome mis-segregation and aneuploidy, including trisomy 21: requirement for tau and APP.Identification of oligomers at early stages of tau aggregation in Alzheimer's disease.Structural neuroimaging in Altheimer's disease: do white matter hyperintensities matter?Targeting Abeta and tau in Alzheimer's disease, an early interim report Recent insights into the molecular genetics of dementia Modeling familial Danish dementia in mice supports the concept of the amyloid hypothesis of Alzheimer's disease.Genetics of Alzheimer disease in the pre- and post-GWAS era Syntheses, neural protective activities, and inhibition of glycogen synthase kinase-3β of substituted quinolines.Dissecting the involvement of tropomyosin-related kinase A and p75 neurotrophin receptor signaling in NGF deficit-induced neurodegeneration Hemichannels in neurodegenerative diseases: is there a link to pathology?Microarray analysis of CA1 pyramidal neurons in a mouse model of tauopathy reveals progressive synaptic dysfunction.Testosterone deficiency accelerates neuronal and vascular aging of SAMP8 mice: protective role of eNOS and SIRT1 Anti-aβ therapeutics in Alzheimer's disease: the need for a paradigm shift Prion strain interactions are highly selective.Sevoflurane induces tau phosphorylation and glycogen synthase kinase 3β activation in young mice Linking cardiometabolic disorders to sporadic Alzheimer's disease: a perspective on potential mechanisms and mediators.Cytoplasmic retention of protein phosphatase 2A inhibitor 2 (I2PP2A) induces Alzheimer-like abnormal hyperphosphorylation of Tau.AlzPathway: a comprehensive map of signaling pathways of Alzheimer's disease.Anesthetic isoflurane increases phosphorylated tau levels mediated by caspase activation and Aβ generation.Evaluating glymphatic pathway function utilizing clinically relevant intrathecal infusion of CSF tracer

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Linking Abeta and tau in late-onset Alzheimer's disease: a dual pathway hypothesis

http://www.wikidata.org/entity/Q37336139

description

2008 nî lūn-bûn

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2008年の論文

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2008年学术文章

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2008年学术文章

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2008年學術文章

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2008年學術文章

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2008年學術文章

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name

Linking Abeta and tau in late-onset Alzheimer's disease: a dual pathway hypothesis

@en

Linking Abeta and tau in late-onset Alzheimer's disease: a dual pathway hypothesis.

@nl

type

label

Linking Abeta and tau in late-onset Alzheimer's disease: a dual pathway hypothesis

@en

Linking Abeta and tau in late-onset Alzheimer's disease: a dual pathway hypothesis.

@nl

prefLabel

Linking Abeta and tau in late-onset Alzheimer's disease: a dual pathway hypothesis

@en

Linking Abeta and tau in late-onset Alzheimer's disease: a dual pathway hypothesis.

@nl

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J.NEURON.2008.11.007

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Linking Abeta and tau in late-onset Alzheimer's disease: a dual pathway hypothesis

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Karen Duff

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Scott A Small

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P2860

Alzheimer amyloid protein precursor in the rat hippocampus: transport and processing through the perforant path

Human orthologs of yeast vacuolar protein sorting proteins Vps26, 29, and 35: assembly into multimeric complexes

The vacuolar-ATPase inhibitor bafilomycin and mutant VPS35 inhibit canonical Wnt signaling

The low density lipoprotein receptor-related protein 6 interacts with glycogen synthase kinase 3 and attenuates activity

Interaction of the cytosolic domains of sorLA/LR11 with the amyloid precursor protein (APP) and beta-secretase beta-site APP-cleaving enzyme

The retromer complex influences Wnt secretion by recycling wntless from endosomes to the trans-Golgi network

The low density lipoprotein receptor-1, LRP1, interacts with the human frizzled-1 (HFz1) and down-regulates the canonical Wnt signaling pathway

Wingless secretion requires endosome-to-Golgi retrieval of Wntless/Evi/Sprinter by the retromer complex

ApoE3 binding to tau tandem repeat I is abolished by tau serine262 phosphorylation

Cloning of a gene bearing missense mutations in early-onset familial Alzheimer's disease

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