Local production of IFN-gamma by invariant NKT cells modulates acute Lyme carditis - Wikidata - awgldk - TriplyDB

Local production of IFN-gamma by invariant NKT cells modulates acute Lyme carditis

Local production of IFN-gamma by invariant NKT cells modulates acute Lyme carditis

http://www.wikidata.org/entity/Q37186506

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Recognition of Microbial Glycolipids by Natural Killer T Cells Phagosomal TLR signaling upon Borrelia burgdorferi infection Antagonistic Interplay between MicroRNA-155 and IL-10 during Lyme Carditis and Arthritis CD14 cooperates with complement receptor 3 to mediate MyD88-independent phagocytosis of Borrelia burgdorferi Lipid exchange between Borrelia burgdorferi and host cells Organ-specific protective role of NKT cells in virus-induced inflammatory demyelination and myocarditis depends on mouse strain.Raising the roof: the preferential pharmacological stimulation of Th1 and th2 responses mediated by NKT cells.Innate immune deficiency of extremely premature neonates can be reversed by interferon-γ.MicroRNA-146a provides feedback regulation of lyme arthritis but not carditis during infection with Borrelia burgdorferi.Inflammatory mechanisms in sepsis: elevated invariant natural killer T-cell numbers in mouse and their modulatory effect on macrophage function.Invariant NKT cell development requires a full complement of functional CD3 zeta immunoreceptor tyrosine-based activation motifs Of ticks, mice and men: understanding the dual-host lifestyle of Lyme disease spirochaetes.The tick saliva immunosuppressor, Salp15, contributes to Th17-induced pathology during Experimental Autoimmune Encephalomyelitis Different patterns of expression and of IL-10 modulation of inflammatory mediators from macrophages of Lyme disease-resistant and -susceptible mice.Cross-talk between cd1d-restricted nkt cells and γδ cells in t regulatory cell response.CD4+ T cells promote antibody production but not sustained affinity maturation during Borrelia burgdorferi infection.Regulation of oxidative stress by methylation-controlled J protein controls macrophage responses to inflammatory insults Ikaros mediates the DNA methylation-independent silencing of MCJ/DNAJC15 gene expression in macrophages.Macrophage p38 mitogen-activated protein kinase activity regulates invariant natural killer T-cell responses during Borrelia burgdorferi infection Slam haplotype 2 promotes NKT but suppresses Vγ4+ T-cell activation in coxsackievirus B3 infection leading to increased liver damage but reduced myocarditis The role of invariant natural killer T cells in microbial immunity T Cells Exacerbate Lyme Borreliosis in TLR2-Deficient Mice.Innate-like recognition of microbes by invariant natural killer T cells.Spatiotemporal evolution of erythema migrans, the hallmark rash of Lyme disease.Natural killer T cells and atherosclerosis: form and function meet pathogenesis Infection with the Lyme disease pathogen suppresses innate immunity in mice with diet-induced obesity.iNKT cells in microbial immunity: recognition of microbial glycolipids.Interplay between carbohydrate and lipid in recognition of glycolipid antigens by natural killer T cells.From the Deep Sea to Everywhere: Environmental Antigens for iNKT Cells.Interferon-γ influences the composition of leukocytic infiltrates in murine lyme carditis.Flagellar motility of the pathogenic spirochetes.Invariant natural killer T cells: front line fighters in the war against pathogenic microbes.Localized production of IL-10 suppresses early inflammatory cell infiltration and subsequent development of IFN-γ-mediated Lyme arthritis The role of CD1d-restricted NKT cells in the clearance of Pseudomonas aeruginosa from the lung is dependent on the host genetic background.Endothelial cells and fibroblasts amplify the arthritogenic type I IFN response in murine Lyme disease and are major sources of chemokines in Borrelia burgdorferi-infected joint tissue.CD14 targets complement receptor 3 to lipid rafts during phagocytosis of Borrelia burgdorferi.Slam haplotypes modulate the response to lipopolysaccharide in vivo through control of NKT cell number and function γδ T lymphocytes kill T regulatory cells through CD1d.Fructose-induced steatosis in mice: role of plasminogen activator inhibitor-1, microsomal triglyceride transfer protein and NKT cells.Molecular recognition of microbial lipid-based antigens by T cells.

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Local production of IFN-gamma by invariant NKT cells modulates acute Lyme carditis

http://www.wikidata.org/entity/Q37186506

description

article científic

@ca

article scientifique

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articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on March 2009

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Local production of IFN-gamma by invariant NKT cells modulates acute Lyme carditis

@en

Local production of IFN-gamma by invariant NKT cells modulates acute Lyme carditis.

@nl

type

label

Local production of IFN-gamma by invariant NKT cells modulates acute Lyme carditis

@en

Local production of IFN-gamma by invariant NKT cells modulates acute Lyme carditis.

@nl

prefLabel

Local production of IFN-gamma by invariant NKT cells modulates acute Lyme carditis

@en

Local production of IFN-gamma by invariant NKT cells modulates acute Lyme carditis.

@nl

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JIMMUNOL.0804111

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Journal of Immunology

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Local production of IFN-gamma by invariant NKT cells modulates acute Lyme carditis

@en

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Chris M Olson

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Hooman Izadi

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Jonathan E Boyson

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Justin D Radolf

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Sally A Huber

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Tonya C Bates

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P2860

p38 mitogen-activated protein kinase controls NF-kappaB transcriptional activation and tumor necrosis factor alpha production through RelA phosphorylation mediated by mitogen- and stress-activated protein kinase 1 in response to Borrelia burgdorferi

Interferon-gamma: an overview of signals, mechanisms and functions

Cellular responses to interferon-gamma

Invariant NKT cells sustain specific B cell responses and memory.

Induction of host matrix metalloproteinases by Borrelia burgdorferi differs in human and murine lyme arthritis.

Activation of gamma delta T cells by Borrelia burgdorferi is indirect via a TLR- and caspase-dependent pathway

Experimental lyme arthritis in the absence of interleukin-4 or gamma interferon.

Gamma interferon is not required for arthritis resistance in the murine Lyme disease model

CD4+ T helper 1 cells facilitate regression of murine Lyme carditis

Control of Borrelia burgdorferi-specific CD4+-T-cell effector function by interleukin-12- and T-cell receptor-induced p38 mitogen-activated protein kinase activity

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3728-3734

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scholarly article

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10.4049/JIMMUNOL.0804111

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2009-03-01T00:00:00Z

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24180987

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19265151

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