Population structure of Toxoplasma gondii: clonal expansion driven by infrequent recombination and selective sweeps.
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Mice infected with low-virulence strains of Toxoplasma gondii lose their innate aversion to cat urine, even after extensive parasite clearancePhosphorylation of immunity-related GTPases by a Toxoplasma gondii-secreted kinase promotes macrophage survival and virulenceA nuclear factor of high mobility group box protein in Toxoplasma gondiiStructure of the Toxoplasma gondii ROP18 Kinase Domain Reveals a Second Ligand Binding Pocket Required for Acute VirulencePhosphorylation of mouse immunity-related GTPase (IRG) resistance proteins is an evasion strategy for virulent Toxoplasma gondiiAdmixture and recombination among Toxoplasma gondii lineages explain global genome diversityWhole genome sequencing of a natural recombinant Toxoplasma gondii strain reveals chromosome sorting and local allelic variantsSexual recombination punctuated by outbreaks and clonal expansions predicts Toxoplasma gondii population geneticsPopulation Genetic Analysis Reveals a High Genetic Diversity in the Brazilian Cryptococcus gattii VGII Population and Shifts the Global Origin from the Amazon Rainforest to the Semi-arid Desert in the Northeast of BrazilToxoplasma gondii Infection Induces High Mobility Group Box 1 Released from Mouse Macrophages.Comparative studies of macrophage-biased responses in mice to infection with Toxoplasma gondii ToxoDB #9 strains of different virulence isolated from China.Efficient gene disruption in diverse strains of Toxoplasma gondii using CRISPR/CAS9.Virulent Toxoplasma gondii evade immunity-related GTPase-mediated parasite vacuole disruption within primed macrophages.The Toxoplasma pseudokinase ROP5 forms complexes with ROP18 and ROP17 kinases that synergize to control acute virulence in mice.Genotyping of Toxoplasma gondii from Rats (Rattus rattus) in Riyadh, Saudi Arabia.Polyparasitism is associated with increased disease severity in Toxoplasma gondii-infected marine sentinel speciesToxoplasma gondii rhoptry kinase ROP16 activates STAT3 and STAT6 resulting in cytokine inhibition and arginase-1-dependent growth controlModulation of innate immunity by Toxoplasma gondii virulence effectors.Development of CRISPR/Cas9 for Efficient Genome Editing in Toxoplasma gondii.Structural and evolutionary divergence of eukaryotic protein kinases in Apicomplexa.Geographic separation of domestic and wild strains of Toxoplasma gondii in French Guiana correlates with a monomorphic version of chromosome1a.Epidemiology of and diagnostic strategies for toxoplasmosisToxoplasma gondii: immune response and protective efficacy induced by ROP16/GRA7 multicomponent DNA vaccine with a genetic adjuvant B7-2Single-nucleotide polymorphisms of two closely related microsporidian parasites suggest a clonal population expansion after the last glaciation.Subversion of host cellular functions by the apicomplexan parasites.Microsatellite and single-nucleotide polymorphisms indicate recurrent transitions to asexuality in a microsporidian parasite.Western Australian marsupials are multiply infected with genetically diverse strains of Toxoplasma gondii.Self-mating in the definitive host potentiates clonal outbreaks of the apicomplexan parasites Sarcocystis neurona and Toxoplasma gondiiComprehensive proteomic analysis of membrane proteins in Toxoplasma gondii.De novo reconstruction of the Toxoplasma gondii transcriptome improves on the current genome annotation and reveals alternatively spliced transcripts and putative long non-coding RNAsOptimizing small molecule inhibitors of calcium-dependent protein kinase 1 to prevent infection by Toxoplasma gondiiGuanylate-binding protein 1 (Gbp1) contributes to cell-autonomous immunity against Toxoplasma gondii.Genetic basis for phenotypic differences between different Toxoplasma gondii type I strainsGenetic analyses of atypical Toxoplasma gondii strains reveal a fourth clonal lineage in North America.Modulation of early β-defensin-2 production as a mechanism developed by type I Toxoplasma gondii to evade human intestinal immunityToxoplasma gondii protein disulfide isomerase (TgPDI) is a novel vaccine candidate against toxoplasmosisVirulence differences in Toxoplasma mediated by amplification of a family of polymorphic pseudokinases.Severe South American ocular toxoplasmosis is associated with decreased Ifn-γ/Il-17a and increased Il-6/Il-13 intraocular levels.Toxoplasma polymorphic effectors determine macrophage polarization and intestinal inflammation.Toxoplasma gondii superinfection and virulence during secondary infection correlate with the exact ROP5/ROP18 allelic combination.
P2860
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P2860
Population structure of Toxoplasma gondii: clonal expansion driven by infrequent recombination and selective sweeps.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on January 2008
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Population structure of Toxopl ...... bination and selective sweeps.
@en
Population structure of Toxopl ...... bination and selective sweeps.
@nl
type
label
Population structure of Toxopl ...... bination and selective sweeps.
@en
Population structure of Toxopl ...... bination and selective sweeps.
@nl
prefLabel
Population structure of Toxopl ...... bination and selective sweeps.
@en
Population structure of Toxopl ...... bination and selective sweeps.
@nl
P1476
Population structure of Toxopl ...... bination and selective sweeps.
@en
P2093
James W Ajioka
P304
P356
10.1146/ANNUREV.MICRO.62.081307.162925
P577
2008-01-01T00:00:00Z