Induction of HIF-1alpha expression by intermittent hypoxia: involvement of NADPH oxidase, Ca2+ signaling, prolyl hydroxylases, and mTOR.
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Hypoxia-inducible factors and the response to hypoxic stressIncreased Oxidative Stress as a Selective Anticancer TherapyPeripheral chemoreception and arterial pressure responses to intermittent hypoxiaUnexpected benefits of intermittent hypoxia: enhanced respiratory and nonrespiratory motor functionArrhythmia risk associated with sleep disordered breathing in chronic heart failureNeuromolecular mechanisms mediating the effects of chronic intermittent hypoxia on adrenal medullaHypoxia-inducible factors and hypertension: lessons from sleep apnea syndromeImpact of the phosphatidylinositide 3-kinase signaling pathway on the cardioprotection induced by intermittent hypoxiaPost-translational modification of glutamic acid decarboxylase 67 by intermittent hypoxia: evidence for the involvement of dopamine D1 receptor signalingChronic nicotine induces hypoxia inducible factor-2α in perinatal rat adrenal chromaffin cells: role in transcriptional upregulation of KATP channel subunit Kir6.2Hypoxia-inducible factor 1 mediates increased expression of NADPH oxidase-2 in response to intermittent hypoxiaEffects of stem cell factor on hypoxia-inducible factor 1 alpha accumulation in human acute myeloid leukaemia and LAD2 mast cellsMolecular mechanisms of HIF-1alpha modulation induced by oxygen tension and BMP2 in glioblastoma derived cells.Pattern-specific sustained activation of tyrosine hydroxylase by intermittent hypoxia: role of reactive oxygen species-dependent downregulation of protein phosphatase 2A and upregulation of protein kinasesThe polymorphic and contradictory aspects of intermittent hypoxiaGlobal gene expression profiling in three tumor cell lines subjected to experimental cycling and chronic hypoxia.CaMKII Inhibitor KN-62 Blunts Tumor Response to Hypoxia by Inhibiting HIF-1α in Hepatoma Cells.NADPH oxidase 2 mediates intermittent hypoxia-induced mitochondrial complex I inhibition: relevance to blood pressure changes in ratsHypoxia-inducible factor 2α (HIF-2α) heterozygous-null mice exhibit exaggerated carotid body sensitivity to hypoxia, breathing instability, and hypertensionIntermittent hypoxia augments acute hypoxic sensing via HIF-mediated ROSRedox signaling in cardiac myocytes.Molecular biomarkers of vascular dysfunction in obstructive sleep apnea.Hypoxia. 4. Hypoxia and ion channel functionXanthine oxidase mediates hypoxia-inducible factor-2α degradation by intermittent hypoxia.Increased risk of benign prostate hyperplasia in sleep apnea patients: a nationwide population-based study.Simulating obstructive sleep apnea patients' oxygenation characteristics into a mouse model of cyclical intermittent hypoxia.HIF-1α activation by intermittent hypoxia requires NADPH oxidase stimulation by xanthine oxidaseSensory plasticity of the carotid body: role of reactive oxygen species and physiological significance.Spinal vascular endothelial growth factor induces phrenic motor facilitation via extracellular signal-regulated kinase and Akt signaling.Adenosine A₂a receptors and O₂ sensing in developmentRole of compartmentalization on HiF-1α degradation dynamics during changing oxygen conditions: a computational approach.Obstructive sleep apnea: an emerging risk factor for atherosclerosis.NNC 55-0396, a T-type Ca2+ channel inhibitor, inhibits angiogenesis via suppression of hypoxia-inducible factor-1α signal transduction.Sleep-Disordered Breathing in Patients with Polycystic Liver and Kidney Disease Referred for Transcatheter Arterial EmbolizationEfficacy of Acute Intermittent Hypoxia on Physical Function and Health Status in Humans with Spinal Cord Injury: A Brief Review.Humans In Hypoxia: A Conspiracy Of Maladaptation?!Proteolytic Cleavage of AMPKα and Intracellular MMP9 Expression Are Both Required for TLR4-Mediated mTORC1 Activation and HIF-1α Expression in Leukocytes.Palmitoylethanolamide Modulates Inflammation-Associated Vascular Endothelial Growth Factor (VEGF) Signaling via the Akt/mTOR Pathway in a Selective Peroxisome Proliferator-Activated Receptor Alpha (PPAR-α)-Dependent Manner.Hypoxia-inducible factor 1 transcriptional activity in endothelial cells is required for acute phase cardioprotection induced by ischemic preconditioning.Transcriptional responses to intermittent hypoxia
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P2860
Induction of HIF-1alpha expression by intermittent hypoxia: involvement of NADPH oxidase, Ca2+ signaling, prolyl hydroxylases, and mTOR.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on December 2008
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Induction of HIF-1alpha expres ...... prolyl hydroxylases, and mTOR.
@en
Induction of HIF-1alpha expres ...... prolyl hydroxylases, and mTOR.
@nl
type
label
Induction of HIF-1alpha expres ...... prolyl hydroxylases, and mTOR.
@en
Induction of HIF-1alpha expres ...... prolyl hydroxylases, and mTOR.
@nl
prefLabel
Induction of HIF-1alpha expres ...... prolyl hydroxylases, and mTOR.
@en
Induction of HIF-1alpha expres ...... prolyl hydroxylases, and mTOR.
@nl
P2093
P2860
P356
P1476
Induction of HIF-1alpha expres ...... prolyl hydroxylases, and mTOR.
@en
P2093
Guoxiang Yuan
Jayasri Nanduri
Nanduri R Prabhakar
Shakil Khan
P2860
P304
P356
10.1002/JCP.21537
P577
2008-12-01T00:00:00Z