Fatty acid amide hydrolase inhibition heightens anandamide signaling without producing reinforcing effects in primates. - Wikidata - awgldk - TriplyDB

Fatty acid amide hydrolase inhibition heightens anandamide signaling without producing reinforcing effects in primates.

Fatty acid amide hydrolase inhibition heightens anandamide signaling without producing reinforcing effects in primates.

http://www.wikidata.org/entity/Q37239653

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Hub and switches: endocannabinoid signalling in midbrain dopamine neurons Cannabinoid-based drugs targeting CB1 and TRPV1, the sympathetic nervous system, and arthritis Chemical probes of endocannabinoid metabolism The pharmacological landscape and therapeutic potential of serine hydrolases Monoacylglycerol lipase - a target for drug development?Modulation of the endocannabinoid system: vulnerability factor and new treatment target for stimulant addiction Fatty acid amide hydrolase inhibitors: a patent review (2009-2014)Inhibition of FAAH and activation of PPAR: new approaches to the treatment of cognitive dysfunction and drug addiction Fatty acid amide hydrolase as a potential therapeutic target for the treatment of pain and CNS disorders Elevating endocannabinoid levels: pharmacological strategies and potential therapeutic applications Methanandamide attenuates cocaine-induced hyperthermia in rats by a cannabinoid CB1-dopamine D2 receptor mechanism Blockade of nicotine reward and reinstatement by activation of alpha-type peroxisome proliferator-activated receptors Dopaminergic augmentation of delta-9-tetrahydrocannabinol (THC) discrimination: possible involvement of D2-induced formation of anandamide Endocannabinoid signalling in reward and addiction.In vivo pharmacology of endocannabinoids and their metabolic inhibitors: therapeutic implications in Parkinson's disease and abuse liability Intracranial self-stimulation to evaluate abuse potential of drugs.Cannabinoid Regulation of Brain Reward Processing with an Emphasis on the Role of CB1 Receptors: A Step Back into the Future Enhancement of endocannabinoid signaling by fatty acid amide hydrolase inhibition: a neuroprotective therapeutic modality Animal models of cannabinoid reward.The role of fatty acid amide hydrolase inhibition in nicotine reward and dependence.Endocannabinoid 2-Arachidonoylglycerol Self-Administration by Sprague-Dawley Rats and Stimulation of in vivo Dopamine Transmission in the Nucleus Accumbens Shell.Inhibitor of fatty acid amide hydrolase normalizes cardiovascular function in hypertension without adverse metabolic effects A second generation of carbamate-based fatty acid amide hydrolase inhibitors with improved activity in vivo Discovery of potent and reversible monoacylglycerol lipase inhibitors.Drug addiction.Acetaminophen inhibits status epilepticus in cultured hippocampal neurons.Differential effects of endocannabinoid catabolic inhibitors on morphine withdrawal in mice.Inhibition of monoacylglycerol lipase reduces nicotine withdrawal Reinforcing and neurochemical effects of cannabinoid CB1 receptor agonists, but not cocaine, are altered by an adenosine A2A receptor antagonist.The endogenous cannabinoid 2-arachidonoylglycerol is intravenously self-administered by squirrel monkeys Fatty acid amide hydrolase blockade attenuates the development of collagen-induced arthritis and related thermal hyperalgesia in mice.Effects of fatty acid amide hydrolase inhibition on neuronal responses to nicotine, cocaine and morphine in the nucleus accumbens shell and ventral tegmental area: involvement of PPAR-alpha nuclear receptors.Chronic stimulation of the tone of endogenous anandamide reduces cue- and stress-induced relapse in rats.Blockade of endocannabinoid hydrolytic enzymes attenuates precipitated opioid withdrawal symptoms in mice.The fatty acid amide hydrolase inhibitor URB 597: interactions with anandamide in rhesus monkeys Simultaneous inhibition of fatty acid amide hydrolase and monoacylglycerol lipase shares discriminative stimulus effects with Δ9-tetrahydrocannabinol in mice.Endocannabinoid influence in drug reinforcement, dependence and addiction-related behaviors.Attenuation of persistent pain-related behavior by fatty acid amide hydrolase (FAAH) inhibitors in a rat model of HIV sensory neuropathy.Effects of Fatty Acid Amide Hydrolase (FAAH) Inhibitors in Non-Human Primate Models of Nicotine Reward and Relapse A polymorphism in the gene of the endocannabinoid-degrading enzyme FAAH (FAAH C385A) is associated with emotional-motivational reactivity.

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Fatty acid amide hydrolase inhibition heightens anandamide signaling without producing reinforcing effects in primates.

http://www.wikidata.org/entity/Q37239653

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scientific article published on 23 September 2008

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vedecký článok

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Fatty acid amide hydrolase inh ...... inforcing effects in primates.

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Fatty acid amide hydrolase inh ...... inforcing effects in primates.

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Fatty acid amide hydrolase inh ...... inforcing effects in primates.

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Fatty acid amide hydrolase inh ...... inforcing effects in primates.

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Fatty acid amide hydrolase inh ...... inforcing effects in primates.

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Fatty acid amide hydrolase inh ...... inforcing effects in primates.

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J.BIOPSYCH.2008.08.008

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Biological Psychiatry

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Fatty acid amide hydrolase inh ...... inforcing effects in primates.

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Alexey G Mukhin

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Alvin R King

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Daniele Piomelli

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Godfrey H Redhi

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Jason R Clapper

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Sevil Yasar

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Steven R Goldberg

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Svetlana I Chefer

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P2860

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A comprehensive profile of brain enzymes that hydrolyze the endocannabinoid 2-arachidonoylglycerol

Cyclohexylcarbamic acid 3'- or 4'-substituted biphenyl-3-yl esters as fatty acid amide hydrolase inhibitors: synthesis, quantitative structure-activity relationships, and molecular modeling studies

Meta-analysis of cannabis based treatments for neuropathic and multiple sclerosis-related pain

Molecular characterization of an enzyme that degrades neuromodulatory fatty-acid amides

The fatty acid amide hydrolase inhibitor URB597 (cyclohexylcarbamic acid 3'-carbamoylbiphenyl-3-yl ester) reduces neuropathic pain after oral administration in mice

The molecular logic of endocannabinoid signalling

Modulation of anxiety through blockade of anandamide hydrolysis

Cannabinoids: reward, dependence, and underlying neurochemical mechanisms--a review of recent preclinical data.

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930-937

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scholarly article

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10.1016/J.BIOPSYCH.2008.08.008

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11

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64

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Zuzana Justinova

Marco Bortolato

Regina A Mangieri

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2008-09-23T00:00:00Z

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23280388

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