Murine gammaherpesvirus 68 infection of IFNgamma unresponsive mice: a small animal model for gammaherpesvirus-associated B-cell lymphoproliferative disease.
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Progress and problems in understanding and managing primary Epstein-Barr virus infectionsInterplay of Murine Gammaherpesvirus 68 with NF-kappaB Signaling of the HostViral cyclins mediate separate phases of infection by integrating functions of distinct mammalian cyclinsVirus-encoded microRNAs facilitate gammaherpesvirus latency and pathogenesis in vivoMurine gammaherpesvirus 68 has evolved gamma interferon and stat1-repressible promoters for the lytic switch gene 50.CD4 T-cell help programs a change in CD8 T-cell function enabling effective long-term control of murine gammaherpesvirus 68: role of PD-1-PD-L1 interactions.Histone deacetylases and the nuclear receptor corepressor regulate lytic-latent switch gene 50 in murine gammaherpesvirus 68-infected macrophagesCD8+ T Cell Response to Gammaherpesvirus Infection Mediates Inflammation and Fibrosis in Interferon Gamma Receptor-Deficient MiceMurine gammaherpesvirus 68 infection of gamma interferon-deficient mice on a BALB/c background results in acute lethal pneumonia that is dependent on specific viral genes.Pervasive transcription of a herpesvirus genome generates functionally important RNAs.Prospects of a novel vaccination strategy for human gamma-herpesviruses.CD4 and CD8 T cells directly recognize murine gammaherpesvirus 68-immortalized cells and prevent tumor outgrowth.Latent herpesvirus infection augments experimental pulmonary fibrosisATM supports gammaherpesvirus replication by attenuating type I interferon pathway.The anti-fibrotic effect of inhibition of TGFβ-ALK5 signalling in experimental pulmonary fibrosis in mice is attenuated in the presence of concurrent γ-herpesvirus infection.IFNγ and perforin cooperate to control infection and prevent fatal pathology during persistent gammaherpesvirus infection in mice.Fas ligand-mediated immune surveillance by T cells is essential for the control of spontaneous B cell lymphomas.Host factors associated with the kinetics of Epstein-Barr virus DNA load in patients with primary Epstein-Barr virus infection.Host Tumor Suppressor p18INK4c functions as a potent cell-intrinsic inhibitor of γHV68 reactivation and pathogenesis.Murine Gammaherpesvirus 68: A Small Animal Model for Gammaherpesvirus-Associated Diseases.
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Murine gammaherpesvirus 68 infection of IFNgamma unresponsive mice: a small animal model for gammaherpesvirus-associated B-cell lymphoproliferative disease.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 16 June 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Murine gammaherpesvirus 68 inf ...... l lymphoproliferative disease.
@en
Murine gammaherpesvirus 68 inf ...... l lymphoproliferative disease.
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type
label
Murine gammaherpesvirus 68 inf ...... l lymphoproliferative disease.
@en
Murine gammaherpesvirus 68 inf ...... l lymphoproliferative disease.
@nl
prefLabel
Murine gammaherpesvirus 68 inf ...... l lymphoproliferative disease.
@en
Murine gammaherpesvirus 68 inf ...... l lymphoproliferative disease.
@nl
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Murine gammaherpesvirus 68 inf ...... ll lymphoproliferative disease
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Bette K Kleinschmidt-DeMasters
Carlyne D Cool
Katherine S Lee
Steve D Groshong
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10.1158/0008-5472.CAN-09-0291
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P577
2009-06-16T00:00:00Z