Gamma interferon blocks gammaherpesvirus reactivation from latency. - Wikidata - awgldk - TriplyDB

Gamma interferon blocks gammaherpesvirus reactivation from latency.

Gamma interferon blocks gammaherpesvirus reactivation from latency.

http://www.wikidata.org/entity/Q34233053

about

Enhanced response of T cells from murine gammaherpesvirus 68-infected mice lacking the suppressor of T cell receptor signaling molecules Sts-1 and Sts-2 Latent gammaherpesvirus 68 infection induces distinct transcriptional changes in different organs Effective control of chronic gamma-herpesvirus infection by unconventional MHC Class Ia-independent CD8 T cells.Antibody-independent control of gamma-herpesvirus latency via B cell induction of anti-viral T cell responses.Alternative capture of noncoding RNAs or protein-coding genes by herpesviruses to alter host T cell function.Murine gammaherpesvirus 68 has evolved gamma interferon and stat1-repressible promoters for the lytic switch gene 50.The murine gammaherpesvirus immediate-early Rta synergizes with IRF4, targeting expression of the viral M1 superantigen to plasma cells Inhibition of NF-kappaB signaling reduces virus load and gammaherpesvirus-induced pulmonary fibrosis Viral latency and its regulation: lessons from the gamma-herpesviruses.CD4 T-cell help programs a change in CD8 T-cell function enabling effective long-term control of murine gammaherpesvirus 68: role of PD-1-PD-L1 interactions.gamma-Herpesvirus-induced protection against bacterial infection is transient.Persistent gamma-herpesvirus infection induces a CD4 T cell response containing functionally distinct effector populations.CD80 and CD86 control antiviral CD8+ T-cell function and immune surveillance of murine gammaherpesvirus 68.Strain-dependent requirement for IFN-γ for respiratory control and immunotherapy in murine gammaherpesvirus infection Malignant catarrhal fever induced by Alcelaphine herpesvirus 1 is characterized by an expansion of activated CD3+CD8+CD4- T cells expressing a cytotoxic phenotype in both lymphoid and non-lymphoid tissues Effects of Sleep Fragmentation and Chronic Latent Viral Infection on Behavior and Inflammation in Mice.Control of virus reactivation arrests pulmonary herpesvirus-induced fibrosis in IFN-gamma receptor-deficient mice.Autophagy Genes Enhance Murine Gammaherpesvirus 68 Reactivation from Latency by Preventing Virus-Induced Systemic Inflammation.A gammaherpesvirus-secreted activator of Vbeta4+ CD8+ T cells regulates chronic infection and immunopathology.Cytoplasmic isoforms of Kaposi sarcoma herpesvirus LANA recruit and antagonize the innate immune DNA sensor cGAS Gamma interferon blocks gammaherpesvirus reactivation from latency in a cell type-specific manner.The small noncoding RNAs (sncRNAs) of murine gammaherpesvirus 68 (MHV-68) are involved in regulating the latent-to-lytic switch in vivo Murine gammaherpesvirus 68 infection of IFNgamma unresponsive mice: a small animal model for gammaherpesvirus-associated B-cell lymphoproliferative disease.Cross-regulation between herpesviruses and the TNF superfamily members Murine gammaherpesvirus 68 infection of gamma interferon-deficient mice on a BALB/c background results in acute lethal pneumonia that is dependent on specific viral genes.When autophagy meets viruses: a double-edged sword with functions in defense and offense.Interferon Regulatory Factor 1 and Type I Interferon Cooperate To Control Acute Gammaherpesvirus Infection.ATM supports gammaherpesvirus replication by attenuating type I interferon pathway.Immune activation suppresses initiation of lytic Epstein-Barr virus infection.IFNγ and perforin cooperate to control infection and prevent fatal pathology during persistent gammaherpesvirus infection in mice.Antiviral activity of 20(R)-ginsenoside Rh2 against murine gammaherpesvirus.Herpesvirus and Autophagy: "All Right, Everybody Be Cool, This Is a Robbery!"Host Tumor Suppressor p18INK4c functions as a potent cell-intrinsic inhibitor of γHV68 reactivation and pathogenesis.Recombinant IFN-γ from the bank vole Myodes glareolus: a novel tool for research on rodent reservoirs of zoonotic pathogens.Herpesviruses: Harmonious Pathogens but Relevant Cofactors in Other Diseases?Inhibition of murine herpesvirus-68 replication by IFN-gamma in macrophages is counteracted by the induction of SOCS1 expression Direct Antiviral Mechanisms of Interferon-Gamma

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Gamma interferon blocks gammaherpesvirus reactivation from latency.

http://www.wikidata.org/entity/Q34233053

description

2006 nî lūn-bûn

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2006 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2006 թվականի հունվարին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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name

Gamma interferon blocks gammaherpesvirus reactivation from latency.

@ast

Gamma interferon blocks gammaherpesvirus reactivation from latency.

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Gamma interferon blocks gammaherpesvirus reactivation from latency.

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type

label

Gamma interferon blocks gammaherpesvirus reactivation from latency.

@ast

Gamma interferon blocks gammaherpesvirus reactivation from latency.

@en

Gamma interferon blocks gammaherpesvirus reactivation from latency.

@nl

prefLabel

Gamma interferon blocks gammaherpesvirus reactivation from latency.

@ast

Gamma interferon blocks gammaherpesvirus reactivation from latency.

@en

Gamma interferon blocks gammaherpesvirus reactivation from latency.

@nl

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JVI.80.1.192-200.2006

P1433

Journal of Virology

P1476

Gamma interferon blocks gammaherpesvirus reactivation from latency.

@en

P2093

Ashley L Steed

http://www.w3.org/2001/XMLSchema#string

Daniel L Popkin

http://www.w3.org/2001/XMLSchema#string

Erik S Barton

http://www.w3.org/2001/XMLSchema#string

Mary L Lutzke

http://www.w3.org/2001/XMLSchema#string

Rosemary Rochford

http://www.w3.org/2001/XMLSchema#string

Scott A Tibbetts

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P2860

Complete sequence and genomic analysis of murine gammaherpesvirus 68

Primary Characterization of a Herpesvirus Agent Associated with Kaposi’s Sarcoma

Identification of herpesvirus-like DNA sequences in AIDS-associated Kaposi's sarcoma

Cellular responses to interferon-gamma

Disruption of the murine gammaherpesvirus 68 M1 open reading frame leads to enhanced reactivation from latency.

Rta of murine gammaherpesvirus 68 reactivates the complete lytic cycle from latency.

Murine gammaherpesvirus 68 cyclin D homologue is required for efficient reactivation from latency.

Ongoing viral replication is required for gammaherpesvirus 68-induced vascular damage.

Gamma interferon can prevent herpes simplex virus type 1 reactivation from latency in sensory neurons.

Kinetics of murine gammaherpesvirus 68 gene expression following infection of murine cells in culture and in mice.

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192-200

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scholarly article

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10.1128/JVI.80.1.192-200.2006

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1

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80

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Herbert W. "Skip" Virgin

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2006-01-01T00:00:00Z

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7416581

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16352543

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1317536

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