Lung NF-kappaB activation and neutrophil recruitment require IL-1 and TNF receptor signaling during pneumococcal pneumonia.
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Common NFKBIL2 polymorphisms and susceptibility to pneumococcal disease: a genetic association studySystemic disease during Streptococcus pneumoniae acute lung infection requires 12-lipoxygenase-dependent inflammationTNF-alpha drives remodeling of blood vessels and lymphatics in sustained airway inflammation in mice.Differential activation of inflammatory pathways in A549 type II pneumocytes by Streptococcus pneumoniae strains with different adherence properties.Pneumolysin activates the NLRP3 inflammasome and promotes proinflammatory cytokines independently of TLR4.A cardinal role for cathepsin d in co-ordinating the host-mediated apoptosis of macrophages and killing of pneumococci.Mechanisms of neutrophil accumulation in the lungs against bacteria.Mice lacking both TNF and IL-1 receptors exhibit reduced lung inflammation and delay in onset of death following infection with a highly virulent H5N1 virus.TLR-mediated inflammatory responses to Streptococcus pneumoniae are highly dependent on surface expression of bacterial lipoproteinsMyeloid ZFP36L1 does not regulate inflammation or host defense in mouse models of acute bacterial infectionIL-36α exerts pro-inflammatory effects in the lungs of miceRegulation of neutrophilic inflammation by proteinase-activated receptor 1 during bacterial pulmonary infection.Role of an iron-dependent transcriptional regulator in the pathogenesis and host response to infection with Streptococcus pneumoniaeIkappaB genetic polymorphisms and invasive pneumococcal disease.NFKBIZ polymorphisms and susceptibility to pneumococcal disease in European and African populations.Mathematical model of a three-stage innate immune response to a pneumococcal lung infectionAirway epithelial MyD88 restores control of Pseudomonas aeruginosa murine infection via an IL-1-dependent pathway.Earliest innate immune responses require macrophage RelA during pneumococcal pneumonia.Critical roles of ASC inflammasomes in caspase-1 activation and host innate resistance to Streptococcus pneumoniae infection.Pulmonary inflammation triggered by ricin toxin requires macrophages and IL-1 signalingInterleukin-1β regulates CXCL8 release and influences disease outcome in response to Streptococcus pneumoniae, defining intercellular cooperation between pulmonary epithelial cells and macrophages.Tumor necrosis factor-alpha deficiency impairs host defense against Streptococcus pneumoniaeHepatocyte-specific mutation of both NF-κB RelA and STAT3 abrogates the acute phase response in miceElectroporation-mediated gene transfer of the Na+,K+ -ATPase rescues endotoxin-induced lung injury.Leukemia inhibitory factor signaling is required for lung protection during pneumoniaHyperbaric Oxygen Reduces Production of Reactive Oxygen Species in Neutrophils from Polytraumatized Patients Yielding in the Inhibition of p38 MAP Kinase and Downstream Pathways.Type I alveolar epithelial cells mount innate immune responses during pneumococcal pneumonia.Differential contribution of bacterial N-formyl-methionyl-leucyl- phenylalanine and host-derived CXC chemokines to neutrophil infiltration into pulmonary alveoli during murine pneumococcal pneumoniaType I interleukin-1 receptor is required for pulmonary responses to subacute ozone exposure in mice.Tumor necrosis factor-alpha from macrophages enhances LPS-induced clara cell expression of keratinocyte-derived chemokine.Electroporation- and mechanical ventilation-mediated gene transfer to the lung.Respiratory infection and the impact of pulmonary immunity on lung health and diseaseCritical involvement of pneumolysin in production of interleukin-1alpha and caspase-1-dependent cytokines in infection with Streptococcus pneumoniae in vitro: a novel function of pneumolysin in caspase-1 activationStreptococcus pneumoniae Endopeptidase O (PepO) Elicits a Strong Innate Immune Response in Mice via TLR2 and TLR4 Signaling Pathways.Alveolar epithelial STAT3, IL-6 family cytokines, and host defense during Escherichia coli pneumoniaRoles of STAT3 in protein secretion pathways during the acute-phase response.The role of the macrophage in lung disease mediated by bacteria.Animal models of human pneumonia.Acute lower respiratory tract infectionReactive oxygen species regulate neutrophil recruitment and survival in pneumococcal pneumonia
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P2860
Lung NF-kappaB activation and neutrophil recruitment require IL-1 and TNF receptor signaling during pneumococcal pneumonia.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on December 2005
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Lung NF-kappaB activation and ...... during pneumococcal pneumonia.
@en
Lung NF-kappaB activation and ...... during pneumococcal pneumonia.
@nl
type
label
Lung NF-kappaB activation and ...... during pneumococcal pneumonia.
@en
Lung NF-kappaB activation and ...... during pneumococcal pneumonia.
@nl
prefLabel
Lung NF-kappaB activation and ...... during pneumococcal pneumonia.
@en
Lung NF-kappaB activation and ...... during pneumococcal pneumonia.
@nl
P2093
P2860
P1476
Lung NF-kappaB activation and ...... during pneumococcal pneumonia.
@en
P2093
Benjamin T Simms
Joseph P Mizgerd
Mariya S Kogan
Matthew R Jones
Michal M Lupa
P2860
P304
P356
10.4049/JIMMUNOL.175.11.7530
P407
P577
2005-12-01T00:00:00Z