Real-time in vivo imaging of p16Ink4a reveals cross talk with p53. - Wikidata - awgldk - TriplyDB

Real-time in vivo imaging of p16Ink4a reveals cross talk with p53.

Real-time in vivo imaging of p16Ink4a reveals cross talk with p53.

http://www.wikidata.org/entity/Q37309123

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Cellular senescence and tumor suppressor gene p16 Roles and mechanisms of cellular senescence in regulation of tissue homeostasis Redox control of senescence and age-related disease The second hit of DNA methylation Human Papillomavirus (HPV) Prevalence in Nasal and Antrochoanal Polyps and Association with Clinical Data.AMP-activated protein kinase reduces inflammatory responses and cellular senescence in pulmonary emphysema Exosomes maintain cellular homeostasis by excreting harmful DNA from cells.Iscador Qu inhibits doxorubicin-induced senescence of MCF7 cells Four faces of cellular senescence.Aging, cellular senescence, and cancer The essence of senescence.Monitoring tumorigenesis and senescence in vivo with a p16(INK4a)-luciferase model.An essential role for senescent cells in optimal wound healing through secretion of PDGF-AA Increased Arf/p53 activity in stem cells, aging and cancer.Ablation of the p16(INK4a) tumour suppressor reverses ageing phenotypes of klotho mice Dysregulation of the Bmi-1/p16(Ink⁴a) pathway provokes an aging-associated decline of submandibular gland function Senescence in tumours: evidence from mice and humans Mitochondrial DNA damage induces apoptosis in senescent cells Oncogene-induced senescence and its role in tumor suppression.Bioluminescence imaging: progress and applications Cellular senescence: a double-edged sword in the fight against cancer.Cellular senescence and its effector programs.The emerging role of senescent cells in tissue homeostasis and pathophysiology.Premature aging/senescence in cancer cells facing therapy: good or bad?Senescent cells harbour features of the cancer epigenome.Senescence in Health and Disease.Tumor suppressor p16INK4a controls oncogenic K-Ras function in human pancreatic cancer cells.A simple stochastic model for the feedback circuit between p16INK4a and p53 mediated by p38MAPK: implications for senescence and apoptosis.Small extracellular vesicles secreted from senescent cells promote cancer cell proliferation through EphA2.Targeted p16(Ink4a) epimutation causes tumorigenesis and reduces survival in mice.Role of p16(INK4A) in Replicative Senescence and DNA Damage-Induced Premature Senescence in p53-Deficient Human Cells.Sustained p16INK4a expression is required to prevent IR-induced tumorigenesis in mice Real-time in vivo imaging of p16gene expression: a new approach to study senescence stress signaling in living animals.Manipulating senescence in health and disease: emerging tools.p16Ink4a and p21Cip1/Waf1 promote tumour growth by enhancing myeloid-derived suppressor cells chemotaxis.Cellular Senescence in Mouse Hippocampus After Irradiation and the Role of p53 and p21.Senescence and aging: Causes, consequences, and therapeutic avenues.Mechanisms and functions of cellular senescence.Downregulation of cytoplasmic DNases is implicated in cytoplasmic DNA accumulation and SASP in senescent cells.Different roles for p16(INK) (4a) -Rb pathway and INK4a/ARF methylation between adenocarcinomas of gastric cardia and distal stomach.

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Real-time in vivo imaging of p16Ink4a reveals cross talk with p53.

http://www.wikidata.org/entity/Q37309123

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on August 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Real-time in vivo imaging of p16Ink4a reveals cross talk with p53.

@en

Real-time in vivo imaging of p16Ink4a reveals cross talk with p53.

@nl

type

label

Real-time in vivo imaging of p16Ink4a reveals cross talk with p53.

@en

Real-time in vivo imaging of p16Ink4a reveals cross talk with p53.

@nl

prefLabel

Real-time in vivo imaging of p16Ink4a reveals cross talk with p53.

@en

Real-time in vivo imaging of p16Ink4a reveals cross talk with p53.

@nl

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Journal of Cell Biology

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Real-time in vivo imaging of p16Ink4a reveals cross talk with p53.

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Akiko Takahashi

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Atsushi Hirao

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David J Mann

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Eiji Hara

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Fumiko Hirota

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Hideyuki Saya

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Kimi Yamakoshi

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Masako Ohmura

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Mitsuru Matsumoto

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Naoko Ohtani

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P2860

Premature senescence involving p53 and p16 is activated in response to constitutive MEK/MAPK mitogenic signaling

Feedback circuit among INK4 tumor suppressors constrains human glioblastoma development

Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a

A biomarker that identifies senescent human cells in culture and in aging skin in vivo

Regulation of p16CDKN2 expression and its implications for cell immortalization and senescence

Histone demethylase JMJD3 contributes to epigenetic control of INK4a/ARF by oncogenic RAS

Direct interaction between DNMT1 and G9a coordinates DNA and histone methylation during replication

Epstein-Barr virus LMP1 blocks p16INK4a-RB pathway by promoting nuclear export of E2F4/5

The Polycomb group proteins bind throughout the INK4A-ARF locus and are disassociated in senescent cells

Oxidants, oxidative stress and the biology of ageing

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393-407

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10.1083/JCB.200904105

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186

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2728398

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