Inhibition of NADPH oxidase is neuroprotective after ischemia-reperfusion. - Wikidata - awgldk - TriplyDB

Inhibition of NADPH oxidase is neuroprotective after ischemia-reperfusion.

Inhibition of NADPH oxidase is neuroprotective after ischemia-reperfusion.

http://www.wikidata.org/entity/Q37322537

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The role of Nox2-derived ROS in the development of cognitive impairment after sepsis A combined pre-clinical meta-analysis and randomized confirmatory trial approach to improve data validity for therapeutic target validation Cerebral Small Vessel Disease: Targeting Oxidative Stress as a Novel Therapeutic Strategy?1,4-Dihydropyridine Derivatives: Dihydronicotinamide Analogues-Model Compounds Targeting Oxidative Stress Oxidative Stress and the Use of Antioxidants in Stroke NADPH oxidase in stroke and cerebrovascular disease Mitochondrial reactive oxygen species: a double edged sword in ischemia/reperfusion vs preconditioning Neuroprotection after stroke by targeting NOX4 as a source of oxidative stress Mechanisms and potential therapeutic applications of microglial activation after brain injury Pathophysiology and Treatments of Oxidative Injury in Ischemic Stroke: Focus on the Phagocytic NADPH Oxidase 2 Immune mechanisms in cerebral ischemic tolerance Deficiency in serine protease inhibitor neuroserpin exacerbates ischemic brain injury by increased postischemic inflammation Post-stroke inhibition of induced NADPH oxidase type 4 prevents oxidative stress and neurodegeneration The yin and yang of microglia Superoxide dismutase 1 limits renal microvascular remodeling and attenuates arteriole and blood pressure responses to angiotensin II via modulation of nitric oxide bioavailability Oxygen-sensitive outcomes and gene expression in acute ischemic stroke Estrogen attenuates ischemic oxidative damage via an estrogen receptor alpha-mediated inhibition of NADPH oxidase activation Therapeutic effect of apocynin through antioxidant activity and suppression of apoptosis and inflammation after spinal cord injury.Role of Rac1 GTPase in NADPH oxidase activation and cognitive impairment following cerebral ischemia in the rat Total salvianolic acid improves ischemia-reperfusion-induced microcirculatory disturbance in rat mesentery Reperfusion and neurovascular dysfunction in stroke: from basic mechanisms to potential strategies for neuroprotection.Intermittent hypoxia-induced cognitive deficits are mediated by NADPH oxidase activity in a murine model of sleep apnea.Innate inflammatory responses in stroke: mechanisms and potential therapeutic targets.Neuroprotective effects of consuming bovine colostrum after focal brain ischemia/reperfusion injury in rat model Anandamide protects HT22 cells exposed to hydrogen peroxide by inhibiting CB1 receptor-mediated type 2 NADPH oxidase.Genetic dissection of the ity3 locus identifies a role for ncf2 co-expression modules and suggests selp as a candidate gene underlying the ity3.2 locus.Nox2 oxidase activity accounts for the oxidative stress and vasomotor dysfunction in mouse cerebral arteries following ischemic stroke.Cynandione A mitigates ischemic injuries in rats with cerebral ischemia.Glucocorticoids as an emerging pharmacologic agent for cardiopulmonary resuscitation.Critical role of NADPH oxidase in neuronal oxidative damage and microglia activation following traumatic brain injury.The role of the microglia in acute CNS injury.Targeting NOX enzymes in the central nervous system: therapeutic opportunities.Hyperglycemia as a Risk Factor of Ischemic Stroke Activation of microglia depends on Na+/H+ exchange-mediated H+ homeostasis.NADPH oxidase mediates striatal neuronal injury after transient global cerebral ischemia.Nox2 knockout delays infarct progression and increases vascular recovery through angiogenesis in mice following ischaemic stroke with reperfusion.Oxidative stress in ischemic brain damage: mechanisms of cell death and potential molecular targets for neuroprotection Progressive neurodegeneration after experimental brain trauma: association with chronic microglial activation.Dietary Sutherlandia and elderberry mitigate cerebral ischemia-induced neuronal damage and attenuate p47phox and phospho-ERK1/2 expression in microglial cells Inhibition of gp91(phox) contributes towards normobaric hyperoxia afforded neuroprotection in focal cerebral ischemia.

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P2860

Inhibition of NADPH oxidase is neuroprotective after ischemia-reperfusion.

http://www.wikidata.org/entity/Q37322537

description

article científic

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article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

@tr

scientific article published on 06 May 2009

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Inhibition of NADPH oxidase is neuroprotective after ischemia-reperfusion.

@en

Inhibition of NADPH oxidase is neuroprotective after ischemia-reperfusion.

@nl

type

label

Inhibition of NADPH oxidase is neuroprotective after ischemia-reperfusion.

@en

Inhibition of NADPH oxidase is neuroprotective after ischemia-reperfusion.

@nl

prefLabel

Inhibition of NADPH oxidase is neuroprotective after ischemia-reperfusion.

@en

Inhibition of NADPH oxidase is neuroprotective after ischemia-reperfusion.

@nl

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Journal of Cerebral Blood Flow & Metabolism

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Inhibition of NADPH oxidase is neuroprotective after ischemia-reperfusion.

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Hai Chen

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Pak H Chan

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Yun Seon Song

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P2860

The NOX family of ROS-generating NADPH oxidases: physiology and pathophysiology

Mouse model of X-linked chronic granulomatous disease, an inherited defect in phagocyte superoxide production

Cytochrome c release and caspase activation in traumatic axonal injury

Ischemic cell death in brain neurons

Inflammatory cell adhesion molecules in ischemic cerebrovascular disease

Copper/zinc superoxide dismutase attenuates neuronal cell death by preventing extracellular signal-regulated kinase activation after transient focal cerebral ischemia in mice

Immunohistochemical study of p47Phox and gp91Phox distributions in rat brain

Exacerbation of delayed cell injury after transient global ischemia in mutant mice with CuZn superoxide dismutase deficiency

Transgenic mice with increased Cu/Zn-superoxide dismutase activity: animal model of dosage effects in Down syndrome

Leukocyte-endothelial cell adhesion: avenues for therapeutic intervention

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1262-1272

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scholarly article

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10.1038/JCBFM.2009.47

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7

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29

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2009-05-06T00:00:00Z

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19417757

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