Bicarbonate Induces Vibrio cholerae virulence gene expression by enhancing ToxT activity.
about
A genome-wide approach to discovery of small RNAs involved in regulation of virulence in Vibrio choleraeA bistable switch and anatomical site control Vibrio cholerae virulence gene expression in the intestineVibrio cholerae Biofilms and Cholera PathogenesisCO2 - Intrinsic Product, Essential Substrate, and Regulatory Trigger of Microbial and Mammalian Production ProcessesThe crystal structure of AphB, a virulence gene activator from Vibrio cholerae, reveals residues that influence its response to oxygen and pHCrystal structure and kinetic studies of a tetrameric type II β-carbonic anhydrase from the pathogenic bacterium Vibrio choleraeSuppression of Virulence of Toxigenic Vibrio cholerae by Anethole through the Cyclic AMP (cAMP)-cAMP Receptor Protein Signaling SystemIdentification of Bicarbonate as a Trigger and Genes Involved with Extracellular DNA Export in Mycobacterial BiofilmsThe LysR-type virulence activator AphB regulates the expression of genes in Vibrio cholerae in response to low pH and anaerobiosisCholera toxin production during anaerobic trimethylamine N-oxide respiration is mediated by stringent response in Vibrio cholerae.Anethole inhibits growth of recently emerged multidrug resistant toxigenic Vibrio cholerae O1 El Tor variant strains in vitro.A Genome-Wide Screen Reveals that the Vibrio cholerae Phosphoenolpyruvate Phosphotransferase System Modulates Virulence Gene ExpressionDNA damage and reactive nitrogen species are barriers to Vibrio cholerae colonization of the infant mouse intestine.Stimulation of growth of the human gastric pathogen Helicobacter pylori by atmospheric level of oxygen under high carbon dioxide tensionBile acids and bicarbonate inversely regulate intracellular cyclic di-GMP in Vibrio cholerae.N-terminal residues of the Vibrio cholerae virulence regulatory protein ToxT involved in dimerization and modulation by fatty acids.Tissue-specific Salmonella Typhimurium gene expression during persistence in pigsRNA thermometer controls temperature-dependent virulence factor expression in Vibrio cholerae.A systems biology approach to modeling vibrio cholerae gene expression under virulence-inducing conditions.Transcriptional regulator PerA influences biofilm-associated, platelet binding, and metabolic gene expression in Enterococcus faecalisExpression of colonization factor CS5 of enterotoxigenic Escherichia coli (ETEC) is enhanced in vivo and by the bile component Na glycocholate hydrate.Intestinal Colonization Dynamics of Vibrio cholerae.Vibrio cholerae classical biotype is converted to the viable non-culturable state when cultured with the El Tor biotypeEffects of polyamines on Vibrio cholerae virulence properties.An Adult Mouse Model of Vibrio cholerae-induced Diarrhea for Studying Pathogenesis and Potential Therapy of Cholera.A small unstructured region in Vibrio cholerae ToxT mediates the response to positive and negative effectors and ToxT proteolysisOligopeptide permease A5 modulates vertebrate host-specific adaptation of Borrelia burgdorferi.Glucose- but not rice-based oral rehydration therapy enhances the production of virulence determinants in the human pathogen Vibrio choleraeIdentification of Novel Components Influencing Colonization Factor Antigen I Expression in Enterotoxigenic Escherichia coli.Involvement of PatE, a prophage-encoded AraC-like regulator, in the transcriptional activation of acid resistance pathways of enterohemorrhagic Escherichia coli strain EDL933.Intestinal GPS: bile and bicarbonate control cyclic di-GMP to provide Vibrio cholerae spatial cues within the small intestine.Conjugated Linoleic Acid Reduces Cholera Toxin Production In Vitro and In Vivo by Inhibiting Vibrio cholerae ToxT Activity.Bile salt-induced intermolecular disulfide bond formation activates Vibrio cholerae virulence.ToxR recognizes a direct repeat element in the toxT, ompU, ompT, and ctxA promoters of Vibrio cholerae to regulate transcription.RegR virulence regulon of rabbit-specific enteropathogenic Escherichia coli strain E22Staying Alive: Vibrio cholerae's Cycle of Environmental Survival, Transmission, and DisseminationSmall-molecule inhibitors of toxT expression in Vibrio cholerae.Control of bacterial virulence by the RalR regulator of the rabbit-specific enteropathogenic Escherichia coli strain E22.Bioinformatic analysis of beta carbonic anhydrase sequences from protozoans and metazoansCarbon dioxide-sensing in organisms and its implications for human disease.
P2860
Q21131415-958FF76E-69E8-4A97-9D3D-1714F278DFE9Q21559407-AF52DEC4-AD9F-4BDC-B720-B5CC54B47F2FQ26769025-2D445EEB-56B4-491F-9997-7341764B0BAFQ26797263-9A615252-4FF4-4196-947B-B419FDEA0970Q27675391-D6606979-5CEB-44D2-91EF-2956B9C565CDQ27702913-21FDCAC7-4B05-4927-85AE-45161D529793Q28547913-A577C125-CA08-47B8-B72E-FCF5841E5AD9Q28817790-76D2076C-76CF-4148-876B-8CA824FCD77BQ29346612-00C7B967-ACFE-4F0C-93D3-78F6EB1703A3Q30360446-194CEF35-55CC-4B01-9D5E-A6F252BD9863Q30371416-05B75EB8-8135-46FD-B7FE-2251AA26CD27Q30389680-3C3F606C-A91F-475B-9E9C-BE24E125F824Q33838288-4F51AA3F-1679-4252-9722-8B443A23037EQ33898649-F9FDB80A-AB51-4466-9A5E-0F72D4A150DDQ33899790-FE52E9B4-14C9-47D0-9C15-4CEFA6E504DAQ33932535-9E02ABB4-0935-4B09-A102-B86E425F48CBQ34009865-8A522C68-6BEB-4294-8996-354FF5FF79BAQ34042049-756B7722-8106-4D42-B3F8-D2E7B7B4A6C0Q34119152-2CCE3D8F-4891-44D8-8C37-EF1FC32501CAQ34230076-224AA83A-B86F-405D-B6D1-58DDB265572CQ34259770-BBC6821A-B29D-413A-9182-23730F5F70B9Q34477267-0D904F37-877A-4D0A-9067-8CB6D460204EQ34551450-C3C11A8F-9A49-4D89-AA19-6C7370EC7192Q34674051-D31D2DD1-5A93-4E13-B4EA-4DA6548E8153Q34798092-9D648B90-D080-415C-B1E1-7237C6EE4B2CQ34853660-15280295-C7EE-4091-9EE7-BF2801B55C0CQ35139390-9D6FA79D-C4D8-4819-996F-70F4353155D5Q35488746-C098B05F-41CE-4AC6-A9BB-C7FAEAD9E950Q35827432-5166F1E6-81C2-42BD-8E45-98735DB00C59Q36155783-316C207E-1766-408D-A681-1829EEE17384Q36191241-C49DEEAB-B55C-4087-9AC7-E25D702D50A6Q36290827-22CE4AB8-1398-4470-91FE-85790F56C0FBQ36598118-0C81BAA8-EAAF-4417-B948-E9B65E9EBB7EQ36646735-F2B73625-6487-4A26-BD96-9F1D0378CB48Q36804659-47035886-B0EB-4AEA-9012-F8364766A1F5Q36956153-50E8AD5C-8F16-4B2F-B6B6-61E7A6264906Q37074702-6FB2BD88-FA4C-4667-BFED-5712C7205C70Q37264776-C8CB34C6-24A9-4BEB-BBAE-55D87DB75B0FQ37534001-E9533A93-6459-44AE-8D4F-D52F247F4CAFQ37623230-FEA92D99-6754-46DF-B569-9BB198318797
P2860
Bicarbonate Induces Vibrio cholerae virulence gene expression by enhancing ToxT activity.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 29 June 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Bicarbonate Induces Vibrio cholerae virulence gene expression by enhancing ToxT activity.
@en
Bicarbonate Induces Vibrio cholerae virulence gene expression by enhancing ToxT activity.
@nl
type
label
Bicarbonate Induces Vibrio cholerae virulence gene expression by enhancing ToxT activity.
@en
Bicarbonate Induces Vibrio cholerae virulence gene expression by enhancing ToxT activity.
@nl
prefLabel
Bicarbonate Induces Vibrio cholerae virulence gene expression by enhancing ToxT activity.
@en
Bicarbonate Induces Vibrio cholerae virulence gene expression by enhancing ToxT activity.
@nl
P2860
P356
P1476
Bicarbonate Induces Vibrio cholerae virulence gene expression by enhancing ToxT activity
@en
P2093
Basel H Abuaita
P2860
P304
P356
10.1128/IAI.00409-09
P407
P577
2009-06-29T00:00:00Z