TGF-[beta]1 limits plaque growth, stabilizes plaque structure, and prevents aortic dilation in apolipoprotein E-null mice. - Wikidata - awgldk - TriplyDB

TGF-[beta]1 limits plaque growth, stabilizes plaque structure, and prevents aortic dilation in apolipoprotein E-null mice.

TGF-[beta]1 limits plaque growth, stabilizes plaque structure, and prevents aortic dilation in apolipoprotein E-null mice.

http://www.wikidata.org/entity/Q37339703

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Inhibition of microRNA-29b reduces murine abdominal aortic aneurysm development Accentuating and Opposing Factors Leading to Development of Thoracic Aortic Aneurysms Not Due to Genetic or Inherited Conditions IGF-1, oxidative stress and atheroprotection TGFB1 genetic polymorphisms and coronary heart disease risk: a meta-analysis Platelet TGF-β1 contributions to plasma TGF-β1, cardiac fibrosis, and systolic dysfunction in a mouse model of pressure overload.Overexpression of TGF-ß1 in macrophages reduces and stabilizes atherosclerotic plaques in ApoE-deficient mice Elevated systemic TGF-beta impairs aortic vasomotor function through activation of NADPH oxidase-driven superoxide production and leads to hypertension, myocardial remodeling, and increased plaque formation in apoE(-/-) mice Macrophage death and defective inflammation resolution in atherosclerosis.99mTc-labeled bevacizumab for detecting atherosclerotic plaque linked to plaque neovascularization and monitoring antiangiogenic effects of atorvastatin treatment in ApoE-/- mice Smooth muscle cell-specific insulin-like growth factor-1 overexpression in Apoe-/- mice does not alter atherosclerotic plaque burden but increases features of plaque stability Interaction between insulin-like growth factor-1 and atherosclerosis and vascular aging.Efficacy and mechanism of angiotensin II receptor blocker treatment in experimental abdominal aortic aneurysms.Regulation of ADAMTS-1, -4 and -5 expression in human macrophages: differential regulation by key cytokines implicated in atherosclerosis and novel synergism between TL1A and IL-17.T cell-specific overexpression of TGFß1 fails to influence atherosclerosis in ApoE-deficient mice A mechanism by which dietary trans fats cause atherosclerosis.Attenuated atherosclerotic lesions in apoE-Fcγ-chain-deficient hyperlipidemic mouse model is associated with inhibition of Th17 cells and promotion of regulatory T cells.Dihydroaustrasulfone Alcohol (WA-25) Impedes Macrophage Foam Cell Formation by Regulating the Transforming Growth Factor-β1 Pathway.Cytokines in atherosclerosis: Key players in all stages of disease and promising therapeutic targets.Aging, atherosclerosis, and IGF-1 Deletion of Periostin Protects Against Atherosclerosis in Mice by Altering Inflammation and Extracellular Matrix Remodeling Vascular complications of diabetes: mechanisms of injury and protective factors Postnatal Deletion of the Type II Transforming Growth Factor-β Receptor in Smooth Muscle Cells Causes Severe Aortopathy in Mice Possible mechanical roles of glycosaminoglycans in thoracic aortic dissection and associations with dysregulated transforming growth factor-β.Prevention of TGFβ induction attenuates angII-stimulated vascular biglycan and atherosclerosis in Ldlr-/- mice Abrogated transforming growth factor beta receptor II (TGFβRII) signalling in dendritic cells promotes immune reactivity of T cells resulting in enhanced atherosclerosis.Tgfbr2 disruption in postnatal smooth muscle impairs aortic wall homeostasis.Transforming growth factor (TGF)-β signaling in cardiac remodeling.Transforming growth factor-β and atherosclerosis: interwoven atherogenic and atheroprotective aspects.Molecular biology of atherosclerosis.T lymphocytes and aortic aneurysms.Regulatory T cells in atherosclerosis: critical immune regulatory function and therapeutic potential.The immunology of atherosclerosis.Growth differentiation factor-15 deficiency inhibits atherosclerosis progression by regulating interleukin-6-dependent inflammatory response to vascular injury.The role of shear-induced transforming growth factor-β signaling in the endothelium.Transforming growth factor-β: transforming plaque to stability Transforming Growth Factor-β1 Inhibits Pseudoaneurysm Formation After Aortic Patch Angioplasty.Regulatory T cells as a new therapeutic target for atherosclerosis.7-Dehydrocholesterol (7-DHC), But Not Cholesterol, Causes Suppression of Canonical TGF-β Signaling and Is Likely Involved in the Development of Atherosclerotic Cardiovascular Disease (ASCVD).TGF-β1 content in atherosclerotic plaques, TGF-β1 serum concentrations and incident coronary events

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TGF-[beta]1 limits plaque growth, stabilizes plaque structure, and prevents aortic dilation in apolipoprotein E-null mice.

http://www.wikidata.org/entity/Q37339703

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 26 March 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

TGF-[beta]1 limits plaque grow ...... in apolipoprotein E-null mice.

@en

TGF-[beta]1 limits plaque grow ...... in apolipoprotein E-null mice.

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type

label

TGF-[beta]1 limits plaque grow ...... in apolipoprotein E-null mice.

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TGF-[beta]1 limits plaque grow ...... in apolipoprotein E-null mice.

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prefLabel

TGF-[beta]1 limits plaque grow ...... in apolipoprotein E-null mice.

@en

TGF-[beta]1 limits plaque grow ...... in apolipoprotein E-null mice.

@nl

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ATVBAHA.109.186593

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Arteriosclerosis, Thrombosis, and Vascular Biology

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TGF-[beta]1 limits plaque grow ...... in apolipoprotein E-null mice

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April Stempien-Otero

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Caroline J Pennington

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Casilde Sesti

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Daniel Minter

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David A Dichek

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Goro Otsuka

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Helén L Dichek

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Jie Hong Hu

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Julien C Marie

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Liang Du

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P2860

Emilin1 links TGF-beta maturation to blood pressure homeostasis

Losartan, an AT1 antagonist, prevents aortic aneurysm in a mouse model of Marfan syndrome

A novel mechanism of action of tetracyclines: effects on nitric oxide synthases

Smad-dependent and Smad-independent pathways in TGF-beta family signalling

Transforming growth factor beta and atherosclerosis: so far, so good for the protective cytokine hypothesis

A syndrome of altered cardiovascular, craniofacial, neurocognitive and skeletal development caused by mutations in TGFBR1 or TGFBR2

Vascular respiratory uncoupling increases blood pressure and atherosclerosis

Hepatic lipase overexpression lowers remnant and LDL levels by a noncatalytic mechanism in LDL receptor-deficient mice.

Production of transforming growth factor beta 1 during repair of arterial injury.

In vivo expression of a conditional TGF-beta1 transgene: no evidence for TGF-beta1 transgene expression in SM22alpha-tTA transgenic mice

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1251-1257

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scholarly article

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10.1161/ATVBAHA.109.186593

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9

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29

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Dylan R. Edwards

Andrew D Frutkin

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2009-03-26T00:00:00Z

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19325140

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