about
Nuclear organization and 3D chromatin architecture in cognition and neuropsychiatric disordersCommon molecular mechanisms in explicit and implicit memorycAMP-responsive element-binding protein (CREB) and cAMP co-regulate activator protein 1 (AP1)-dependent regeneration-associated gene expression and neurite growthTransient expansion of synaptically connected dendritic spines upon induction of hippocampal long-term potentiation.A meeting to remember: meeting on memory and related disordersGenetic selection of poliovirus 2Apro-binding peptidesA stable HeLa cell line that inducibly expresses poliovirus 2A(pro): effects on cellular and viral gene expression.Chromatin acetylation, memory, and LTP are impaired in CBP+/- mice: a model for the cognitive deficit in Rubinstein-Taybi syndrome and its amelioration.cAMP response element-binding protein-mediated gene expression increases the intrinsic excitability of CA1 pyramidal neurons.Enhanced CREB-dependent gene expression increases the excitability of neurons in the basal amygdala and primes the consolidation of contextual and cued fear memory.CREB, memory enhancement and the treatment of memory disorders: promises, pitfalls and prospects.Histone acetylation and CREB binding protein are required for neuronal resistance against ischemic injury.Synapse-specific stabilization of plasticity processes: the synaptic tagging and capture hypothesis revisited 10 years later.Inhibition of cAMP responsive element binding protein in striatal neurons enhances approach and avoidance responses toward morphine--and morphine withdrawal-related cues.A semi-persistent adult ocular dominance plasticity in visual cortex is stabilized by activated CREB.CREB's control of intrinsic and synaptic plasticity: implications for CREB-dependent memory models.Hippocampal gene profiling: toward a systems biology of the hippocampus.Genetic approaches to investigate the role of CREB in neuronal plasticity and memory.Lysine acetyltransferases CBP and p300 as therapeutic targets in cognitive and neurodegenerative disorders.Histone H3 lysine methylation in cognition and intellectual disability disorders.The neuronal activity-driven transcriptome.Can changes in histone acetylation contribute to memory formation?Epigenetic factors in intellectual disability: the Rubinstein-Taybi syndrome as a paradigm of neurodevelopmental disorder with epigenetic origin.Gene expression profiling of facilitated L-LTP in VP16-CREB mice reveals that BDNF is critical for the maintenance of LTP and its synaptic capture.CREB Regulates Distinct Adaptive Transcriptional Programs in Astrocytes and Neurons.Role of Dicer and the miRNA system in neuronal plasticity and brain function.Lack of IL-1R8 in neurons causes hyperactivation of IL-1 receptor pathway and induces MECP2-dependent synaptic defectsHistone acetylation deficits in lymphoblastoid cell lines from patients with Rubinstein-Taybi syndrome.Identification of regions of poliovirus 2BC protein that are involved in cytotoxicity.Ablation of CBP in forebrain principal neurons causes modest memory and transcriptional defects and a dramatic reduction of histone acetylation but does not affect cell viability.A human virus protein, poliovirus protein 2BC, induces membrane proliferation and blocks the exocytic pathway in the yeast Saccharomyces cerevisiae.Brain size regulations by cbp haploinsufficiency evaluated by in-vivo MRI based volumetry.Skill Learning Modulates RNA Pol II Poising at Immediate Early Genes in the Adult Striatum.Inhibition of cAMP response element-binding protein reduces neuronal excitability and plasticity, and triggers neurodegeneration.Capture of the late phase of long-term potentiation within and across the apical and basilar dendritic compartments of CA1 pyramidal neurons: synaptic tagging is compartment restricted.Blocking miRNA Biogenesis in Adult Forebrain Neurons Enhances Seizure Susceptibility, Fear Memory, and Food Intake by Increasing Neuronal Responsiveness.Fine-tuned SRF activity controls asymmetrical neuronal outgrowth: implications for cortical migration, neural tissue lamination and circuit assembly.Selective boosting of transcriptional and behavioral responses to drugs of abuse by histone deacetylase inhibition.Genomic landscape of transcriptional and epigenetic dysregulation in early onset polyglutamine disease.Expression of constitutively active CREB protein facilitates the late phase of long-term potentiation by enhancing synaptic capture.
P50
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P50
description
hulumtues
@sq
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Angel Barco
@ast
Angel Barco
@en
Angel Barco
@es
Angel Barco
@nl
Angel Barco
@sl
type
label
Angel Barco
@ast
Angel Barco
@en
Angel Barco
@es
Angel Barco
@nl
Angel Barco
@sl
prefLabel
Angel Barco
@ast
Angel Barco
@en
Angel Barco
@es
Angel Barco
@nl
Angel Barco
@sl
P1053
C-3062-2008
P106
P1153
8934538000
P21
P31
P3829
P496
0000-0002-0653-3751