The mitochondrial permeability transition pore as a target for preconditioning and postconditioning.
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Role of the MPTP in conditioning the heart - translatability and mechanismMitochondrial reactive oxygen species: a double edged sword in ischemia/reperfusion vs preconditioningProspects for Creation of Cardioprotective and Antiarrhythmic Drugs Based on Opioid Receptor AgonistsIschaemic conditioning and targeting reperfusion injury: a 30 year voyage of discoveryPeri-operative anaesthetic myocardial preconditioning and protection - cellular mechanisms and clinical relevance in cardiac anaesthesiaLate Post-Conditioning with Sevoflurane after Cardiac Surgery--Are Surrogate Markers Associated with Clinical Outcome?Protein S-nitrosylation and cardioprotectionIs there a role for ischaemic conditioning in cardiac surgery?NGFI-B targets mitochondria and induces cardiomyocyte apoptosis in restraint-stressed rats by mediating energy metabolism disorderPreconditioning involves selective mitophagy mediated by Parkin and p62/SQSTM1.Post-conditioning with gradually increased reperfusion provides better cardioprotection in rats.Perconditioning and postconditioning: current knowledge, knowledge gaps, barriers to adoption, and future directions.Calcium-sensing receptor: a sensor and mediator of ischemic preconditioning in the heart.Catestatin improves post-ischemic left ventricular function and decreases ischemia/reperfusion injury in heart.Life without oxygen: gene regulatory responses of the crucian carp (Carassius carassius) heart subjected to chronic anoxiaDodecafluoropentane emulsion elicits cardiac protection against myocardial infarction through an ATP-Sensitive K+ channel dependent mechanismAnaesthetic Postconditioning at the Initiation of CPR Improves Myocardial and Mitochondrial Function in a Pig Model of Prolonged Untreated Ventricular Fibrillation.Blockade of electron transport during ischemia preserves bcl-2 and inhibits opening of the mitochondrial permeability transition poreA2B adenosine receptors inhibit superoxide production from mitochondrial complex I in rabbit cardiomyocytes via a mechanism sensitive to Pertussis toxinIschemia/reperfusion injury and cardioprotective mechanisms: Role of mitochondria and reactive oxygen speciesProtective role of transient pore openings in calcium handling by cardiac mitochondria.Redox signalling and cardioprotection: translatability and mechanismCharacterization of a critical role for CFTR chloride channels in cardioprotection against ischemia/reperfusion injury.Distinct mPTP activation mechanisms in ischaemia-reperfusion: contributions of Ca2+, ROS, pH, and inorganic polyphosphate.Disruption of caveolae blocks ischemic preconditioning-mediated S-nitrosylation of mitochondrial proteins.Cardioprotection by H2S engages a cGMP-dependent protein kinase G/phospholamban pathway.Hyperglycemia raises the threshold of levosimendan- but not milrinone-induced postconditioning in rat heartsExogenous hydrogen sulfide restores cardioprotection of ischemic post-conditioning via inhibition of mPTP opening in the aging cardiomyocytes.Blockade of electron transport at the onset of reperfusion decreases cardiac injury in aged hearts by protecting the inner mitochondrial membrane.Targeted Modification of Mitochondrial ROS Production Converts High Glucose-Induced Cytotoxicity to Cytoprotection: Effects on Anesthetic Preconditioning.T lymphocytes and fractalkine contribute to myocardial ischemia/reperfusion injury in patients.Cardioprotective Properties of the Platelet P2Y12 Receptor Inhibitor, Cangrelor: Protective in Diabetics and Reliant Upon the Presence of BloodAstragaloside IV inhibits oxidative stress-induced mitochondrial permeability transition pore opening by inactivating GSK-3β via nitric oxide in H9c2 cardiac cells.Metformin in non-diabetic patients presenting with ST elevation myocardial infarction: rationale and design of the glycometabolic intervention as adjunct to primary percutaneous intervention in ST elevation myocardial infarction (GIPS)-III trialThe age-associated loss of ischemic preconditioning in the kidney is accompanied by mitochondrial dysfunction, increased protein acetylation and decreased autophagy.Later phase cardioprotection of ischemic post-conditioning against ischemia/reperfusion injury depends on iNOS and PI3K-Akt pathway.Naoxintong Protects Primary Neurons from Oxygen-Glucose Deprivation/Reoxygenation Induced Injury through PI3K-Akt Signaling Pathway.Cardioprotective Role of Caveolae in Ischemia-Reperfusion Injury.Extent of mitochondrial hexokinase II dissociation during ischemia correlates with mitochondrial cytochrome c release, reactive oxygen species production, and infarct size on reperfusionFTY720 postconditions isolated perfused heart by a mechanism independent of sphingosine kinase 2 and different from S1P or ischemic postconditioning.
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The mitochondrial permeability transition pore as a target for preconditioning and postconditioning.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 26 February 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
The mitochondrial permeability ...... itioning and postconditioning.
@en
The mitochondrial permeability ...... itioning and postconditioning.
@nl
type
label
The mitochondrial permeability ...... itioning and postconditioning.
@en
The mitochondrial permeability ...... itioning and postconditioning.
@nl
prefLabel
The mitochondrial permeability ...... itioning and postconditioning.
@en
The mitochondrial permeability ...... itioning and postconditioning.
@nl
P2860
P50
P1476
The mitochondrial permeability ...... ditioning and postconditioning
@en
P2860
P2888
P304
P356
10.1007/S00395-009-0010-X
P577
2009-02-26T00:00:00Z