Amyloid-beta and tau synergistically impair the oxidative phosphorylation system in triple transgenic Alzheimer's disease mice. - Wikidata - awgldk - TriplyDB

Amyloid-beta and tau synergistically impair the oxidative phosphorylation system in triple transgenic Alzheimer's disease mice.

Amyloid-beta and tau synergistically impair the oxidative phosphorylation system in triple transgenic Alzheimer's disease mice.

http://www.wikidata.org/entity/Q37414108

about

Amyloidosis in Retinal Neurodegenerative Diseases Intracellular Calcium Dysregulation: Implications for Alzheimer's Disease Killing Me Softly: Connotations to Unfolded Protein Response and Oxidative Stress in Alzheimer's Disease The Amyloid Precursor Protein-A Novel Player within the Molecular Array of Presynaptic Nanomachines Unfolded Protein Response and Macroautophagy in Alzheimer's, Parkinson's and Prion Diseases A review on mitochondrial restorative mechanism of antioxidants in Alzheimer's disease and other neurological conditions Redox control of protein degradation Mitochondrial Dysfunction Contributes to the Pathogenesis of Alzheimer's Disease Shift in brain metabolism in late onset Alzheimer's disease: implications for biomarkers and therapeutic interventions Mitochondria and the autophagy-inflammation-cell death axis in organismal aging Connecting the dots between tau dysfunction and neurodegeneration Human wild-type full-length tau accumulation disrupts mitochondrial dynamics and the functions via increasing mitofusins.Estrogen amelioration of Aβ-induced defects in mitochondria is mediated by mitochondrial signaling pathway involving ERβ, AKAP and Drp1.Intracellular cleavage of amyloid β by a viral protease NIa prevents amyloid β-mediated cytotoxicity Improved mitochondrial function in brain aging and Alzheimer disease - the new mechanism of action of the old metabolic enhancer piracetam Dynamics of Human Mitochondrial Complex I Assembly: Implications for Neurodegenerative Diseases Tau physiology and pathomechanisms in frontotemporal lobar degeneration Spreading of pathology in neurodegenerative diseases: a focus on human studies Tau promotes neurodegeneration via DRP1 mislocalization in vivo Inhibition of the mitochondrial enzyme ABAD restores the amyloid-β-mediated deregulation of estradiol eIF4A inhibition allows translational regulation of mRNAs encoding proteins involved in Alzheimer's disease Amyloid-beta and mitochondria in aging and Alzheimer's disease: implications for synaptic damage and cognitive decline Mitochondria in Alzheimer's Disease and Diabetes-Associated Neurodegeneration: License to Heal!Early neurovascular dysfunction in a transgenic rat model of Alzheimer's disease.Estrogen: a master regulator of bioenergetic systems in the brain and body Docking Studies and Biological Evaluation of a Potential β-Secretase Inhibitor of 3-Hydroxyhericenone F from Hericium erinaceus.Ginkgo biloba extract ameliorates oxidative phosphorylation performance and rescues abeta-induced failure RCAN1 regulates mitochondrial function and increases susceptibility to oxidative stress in mammalian cells.Effects of dietary supplementation of carnosine on mitochondrial dysfunction, amyloid pathology, and cognitive deficits in 3xTg-AD mice.Convergence of amyloid-beta and tau pathologies on mitochondria in vivo.Alzheimer's disease: diverse aspects of mitochondrial malfunctioning.Is Alzheimer's disease a systemic disease?Altered proteostasis in aging and heat shock response in C. elegans revealed by analysis of the global and de novo synthesized proteome A synergistic dysfunction of mitochondrial fission/fusion dynamics and mitophagy in Alzheimer's disease.Abeta and human amylin share a common toxicity pathway via mitochondrial dysfunction.Data mining of high density genomic variant data for prediction of Alzheimer's disease risk Molecular signatures in post-mortem brain tissue of younger individuals at high risk for Alzheimer's disease as based on APOE genotype.Vascular and metabolic dysfunction in Alzheimer's disease: a review.Systemic mitochondrial dysfunction and the etiology of Alzheimer's disease and down syndrome dementia.Tau-mediated nuclear depletion and cytoplasmic accumulation of SFPQ in Alzheimer's and Pick's disease.

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Amyloid-beta and tau synergistically impair the oxidative phosphorylation system in triple transgenic Alzheimer's disease mice.

http://www.wikidata.org/entity/Q37414108

description

2009 nî lūn-bûn

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2009年の論文

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年學術文章

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2009年學術文章

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2009年學術文章

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name

Amyloid-beta and tau synergist ...... enic Alzheimer's disease mice.

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Amyloid-beta and tau synergist ...... enic Alzheimer's disease mice.

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type

label

Amyloid-beta and tau synergist ...... enic Alzheimer's disease mice.

@en

Amyloid-beta and tau synergist ...... enic Alzheimer's disease mice.

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prefLabel

Amyloid-beta and tau synergist ...... enic Alzheimer's disease mice.

@en

Amyloid-beta and tau synergist ...... enic Alzheimer's disease mice.

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PNAS.0905529106

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Proceedings of the National Academy of Sciences of the United States of America

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Amyloid-beta and tau synergist ...... enic Alzheimer's disease mice.

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Andreas Wiesner

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Egemen Savaskan

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Fides Meier

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Ginette Baysang

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Horst Bluethmann

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Laurence Ozmen

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Stefan Dröse

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Virginie Rhein

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Xiaomin Song

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P2860

Oxidative Stress Signaling in Alzheimers Disease

Abeta and tau form soluble complexes that may promote self aggregation of both into the insoluble forms observed in Alzheimer's disease

Molecular taxonomy of major neuronal classes in the adult mouse forebrain

Triple-transgenic model of Alzheimer's disease with plaques and tangles: intracellular Abeta and synaptic dysfunction

Neurodegenerative tauopathies

Tau filament formation in transgenic mice expressing P301L tau.

Transgenic animal models of Alzheimer's disease and related disorders: histopathology, behavior and therapy.

Maternal family history of Alzheimer's disease predisposes to reduced brain glucose metabolism

A neuronal model of Alzheimer's disease: an insight into the mechanisms of oxidative stress-mediated mitochondrial injury.

The amyloid beta-peptide is imported into mitochondria via the TOM import machinery and localized to mitochondrial cristae

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20057-20062

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scholarly article

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10.1073/PNAS.0905529106

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47

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106

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Christian Czech

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2009-11-06T00:00:00Z

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38074940

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19897719

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Alzheimer's disease

phosphorylation

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2774257

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