The CD160, BTLA, LIGHT/HVEM pathway: a bidirectional switch regulating T-cell activation.
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BTLA mediates inhibition of human tumor-specific CD8+ T cells that can be partially reversed by vaccinationCancer immunotherapy: the beginning of the end of cancer?The Immune System Is a Natural Target for Estrogen Action: Opposing Effects of Estrogen in Two Prototypical Autoimmune DiseasesMolecular mechanisms of T cell co-stimulation and co-inhibitionThe role of costimulatory receptors of the tumour necrosis factor receptor family in atherosclerosisNovel immunotherapies for hematologic malignanciesHow Rheumatoid Arthritis Can Result from Provocation of the Immune System by Microorganisms and VirusesRegulatory T Cell Modulation by CBP/EP300 Bromodomain InhibitionMechanistic Basis for Functional Promiscuity in the TNF and TNF Receptor Superfamilies: Structure of the LIGHT:DcR3 AssemblyIncreased Heterologous Protein Expression in Drosophila S2 Cells for Massive Production of Immune Ligands/Receptors and Structural Analysis of Human HVEMCutting edge: A monoclonal antibody specific for the programmed death-1 homolog prevents graft-versus-host disease in mouse modelsPutting the brakes on BTLA in T cell-mediated cancer immunotherapyDiffuse large B-cell lymphoma: can genomics improve treatment options for a curable cancer?Functional interaction between herpes simplex virus type 2 gD and HVEM transiently dampens local chemokine production after murine mucosal infectionPolymorphic variants of LIGHT (TNF superfamily-14) alter receptor avidity and bioavailability.Mechanisms of costimulation.Herpes virus entry mediator (HVEM) modulates proliferation and activation of regulatory T cells following HSV-1 infectionCostimulatory pathways in transplantation.Herpes simplex virus glycoprotein D interferes with binding of herpesvirus entry mediator to its ligands through downregulation and direct competitionCD160 and PD-1 co-expression on HIV-specific CD8 T cells defines a subset with advanced dysfunction.The effect of adjuvanting cancer vaccines with herpes simplex virus glycoprotein D on melanoma-driven CD8+ T cell exhaustionRegulation of inflammation, autoimmunity, and infection immunity by HVEM-BTLA signaling.Expression of a broad array of negative costimulatory molecules and Blimp-1 in T cells following priming by HIV-1 pulsed dendritic cells.Bicistronic DNA vaccines simultaneously encoding HIV, HSV and HPV antigens promote CD8⁺ T cell responses and protective immunity.HVEM gene polymorphisms are associated with sporadic breast cancer in Chinese women.Surface expression patterns of negative regulatory molecules identify determinants of virus-specific CD8+ T-cell exhaustion in HIV infectionCD8 T cell memory to a viral pathogen requires trans cosignaling between HVEM and BTLA.Herpesvirus entry mediator (TNFRSF14) regulates the persistence of T helper memory cell populations.CD160 is essential for NK-mediated IFN-γ productionTumor necrosis factor superfamily in innate immunity and inflammation.TNF Superfamily Networks: bidirectional and interference pathways of the herpesvirus entry mediator (TNFSF14)T cell exhaustion during persistent viral infections.The soluble amino-terminal region of HVEM mediates efficient herpes simplex virus type 1 infection of gD receptor-negative cells.Challenges and future perspectives of T cell immunotherapy in cancerPositive and negative regulation of cellular immune responses in physiologic conditions and diseasesHerpes virus entry mediator licenses Listeria infection induced immunopathology through control of type I interferon.Herpesvirus entry mediator on radiation-resistant cell lineages promotes ocular herpes simplex virus 1 pathogenesis in an entry-independent manner.Soluble B and T Lymphocyte Attenuator Correlates to Disease Severity in Sepsis and High Levels Are Associated with an Increased Risk of MortalityBTLA expression declines on B cells of the aged and is associated with low responsiveness to the trivalent influenza vaccineT-cell inhibitors: a bench-to-bedside review.
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P2860
The CD160, BTLA, LIGHT/HVEM pathway: a bidirectional switch regulating T-cell activation.
description
article científic
@ca
article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on May 2009
@en
vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
The CD160, BTLA, LIGHT/HVEM pathway: a bidirectional switch regulating T-cell activation.
@en
The CD160, BTLA, LIGHT/HVEM pathway: a bidirectional switch regulating T-cell activation.
@nl
type
label
The CD160, BTLA, LIGHT/HVEM pathway: a bidirectional switch regulating T-cell activation.
@en
The CD160, BTLA, LIGHT/HVEM pathway: a bidirectional switch regulating T-cell activation.
@nl
prefLabel
The CD160, BTLA, LIGHT/HVEM pathway: a bidirectional switch regulating T-cell activation.
@en
The CD160, BTLA, LIGHT/HVEM pathway: a bidirectional switch regulating T-cell activation.
@nl
P2860
P1476
The CD160, BTLA, LIGHT/HVEM pathway: a bidirectional switch regulating T-cell activation.
@en
P2093
Gordon J Freeman
Guifang Cai
P2860
P304
P356
10.1111/J.1600-065X.2009.00783.X
P577
2009-05-01T00:00:00Z