Exclusive expression of Epstein-Barr virus nuclear antigen 1 in Burkitt lymphoma arises from a third promoter, distinct from the promoters used in latently infected lymphocytes.
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Expression of Epstein-Barr virus nuclear antigen-1 induces B cell neoplasia in transgenic miceThe cellular proteins that bind specifically to the Epstein-Barr virus origin of plasmid DNA replication belong to a gene familyEpstein-Barr virus latency switch in human B-cells: a physico-chemical model.Viral response to chemotherapy in endemic burkitt lymphomaThe Epstein-Barr virus major latent promoter Qp is constitutively active, hypomethylated, and methylation sensitiveRedefining the Epstein-Barr virus-encoded nuclear antigen EBNA-1 gene promoter and transcription initiation site in group I Burkitt lymphoma cell linesEstablishment and characterization of a human Epstein-Barr virus-associated gastric carcinoma in SCID miceEpstein-Barr virus nuclear antigen 2 (EBNA2) gene deletion is consistently linked with EBNA3A, -3B, and -3C expression in Burkitt's lymphoma cells and with increased resistance to apoptosis.Host factors LR1 and Sp1 regulate the Fp promoter of Epstein-Barr virus.Murine gammaherpesvirus 68 LANA is essential for virus reactivation from splenocytes but not long-term carriage of viral genome.Constitutive binding of EBNA1 protein to the Epstein-Barr virus replication origin, oriP, with distortion of DNA structure during latent infection.Genome-wide DNA methylation as an epigenetic consequence of Epstein-Barr virus infection of immortalized keratinocytesIdentification of spliced gammaherpesvirus 68 LANA and v-cyclin transcripts and analysis of their expression in vivo during latent infectionGastric carcinoma: monoclonal epithelial malignant cells expressing Epstein-Barr virus latent infection protein.Detection of heterogeneous Epstein-Barr virus gene expression patterns within individual post-transplantation lymphoproliferative disorders.Immortalization of human B lymphocytes by a plasmid containing 71 kilobase pairs of Epstein-Barr virus DNA.Epstein-Barr virus latent messages with shuffled leader exons: remnants of circumgenomic transcription?Structural analyses of the Epstein-Barr virus BamHI A transcriptsA subpopulation of normal B cells latently infected with Epstein-Barr virus resembles Burkitt lymphoma cells in expressing EBNA-1 but not EBNA-2 or LMP1.Transcription start sites downstream of the Epstein-Barr virus (EBV) Fp promoter in early-passage Burkitt lymphoma cells define a fourth promoter for expression of the EBV EBNA-1 proteinMolecular genetic analysis of Epstein-Barr virus Cp promoter functionActivation of the Epstein-Barr virus DNA polymerase promoter by the BRLF1 immediate-early protein is mediated through USF and E2F.Transcription of the Epstein-Barr virus nuclear antigen 1 (EBNA1) gene occurs before induction of the BCR2 (Cp) EBNA gene promoter during the initial stages of infection in B cells.A simple reverse transcriptase PCR assay to distinguish EBNA1 gene transcripts associated with type I and II latency from those arising during induction of the viral lytic cycleThe Epstein-Barr virus EBNA-1 promoter Qp requires an initiator-like element.Transcriptional analysis of Epstein-Barr virus gene expression in EBV-positive gastric carcinoma: unique viral latency in the tumour cellsAn auto-regulatory loop for EBV LMP2A involves activation of Notch.Host-cell-determined methylation of specific Epstein-Barr virus promoters regulates the choice between distinct viral latency programsThe latency pattern of Epstein-Barr virus infection and viral IL-10 expression in cutaneous natural killer/T-cell lymphomas.Epstein-Barr virus latency in blood mononuclear cells: analysis of viral gene transcription during primary infection and in the carrier state.Separation of the complex DNA binding domain of EBNA-1 into DNA recognition and dimerization subdomains of novel structure.Methylation of discrete sites within the enhancer region regulates the activity of the Epstein-Barr virus BamHI W promoter in Burkitt lymphoma lines.Reciprocal regulation of the Epstein-Barr virus BamHI-F promoter by EBNA-1 and an E2F transcription factor.The Epstein-Barr virus (EBV) nuclear antigen 1 BamHI F promoter is activated on entry of EBV-transformed B cells into the lytic cycle.Epstein-Barr virus latent gene expression in uncultured peripheral blood lymphocytes.The Epstein-Barr virus nuclear protein 1 promoter active in type I latency is autoregulated.Methylation status and AP1 elements are involved in EBV-mediated miR-155 expression in EBV positive lymphoma cells.Alternatively initiated gene 50/RTA transcripts expressed during murine and human gammaherpesvirus reactivation from latencyLigation-anchored PCR: a simple amplification technique with single-sided specificity.The Epstein-Barr virus BamHI F promoter is an early lytic promoter: lack of correlation with EBNA 1 gene transcription in group 1 Burkitt's lymphoma cell lines
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Exclusive expression of Epstein-Barr virus nuclear antigen 1 in Burkitt lymphoma arises from a third promoter, distinct from the promoters used in latently infected lymphocytes.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on August 1991
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Exclusive expression of Epstei ...... latently infected lymphocytes.
@en
Exclusive expression of Epstei ...... latently infected lymphocytes.
@nl
type
label
Exclusive expression of Epstei ...... latently infected lymphocytes.
@en
Exclusive expression of Epstei ...... latently infected lymphocytes.
@nl
prefLabel
Exclusive expression of Epstei ...... latently infected lymphocytes.
@en
Exclusive expression of Epstei ...... latently infected lymphocytes.
@nl
P2093
P2860
P921
P356
P1476
Exclusive expression of Epstei ...... latently infected lymphocytes.
@en
P2093
B C Schaefer
M Woisetschlaeger
P2860
P304
P356
10.1073/PNAS.88.15.6550
P407
P577
1991-08-01T00:00:00Z