Resolution of primary severe acute respiratory syndrome-associated coronavirus infection requires Stat1. - Wikidata - awgldk - TriplyDB

Resolution of primary severe acute respiratory syndrome-associated coronavirus infection requires Stat1.

Resolution of primary severe acute respiratory syndrome-associated coronavirus infection requires Stat1.

http://www.wikidata.org/entity/Q37567955

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ACE2 receptor expression and severe acute respiratory syndrome coronavirus infection depend on differentiation of human airway epithelia.Animal models for SARS and MERS coronaviruses Molecular pathology of emerging coronavirus infections Lack of innate interferon responses during SARS coronavirus infection in a vaccination and reinfection ferret model Functional genomics highlights differential induction of antiviral pathways in the lungs of SARS-CoV-infected macaques Severe acute respiratory syndrome coronavirus as an agent of emerging and reemerging infection A decade after SARS: strategies for controlling emerging coronaviruses The role of epidermal growth factor receptor (EGFR) signaling in SARS coronavirus-induced pulmonary fibrosis.Cynomolgus macaque as an animal model for severe acute respiratory syndrome.SARS-CoV pathogenesis is regulated by a STAT1 dependent but a type I, II and III interferon receptor independent mechanism.Cellular immune responses to severe acute respiratory syndrome coronavirus (SARS-CoV) infection in senescent BALB/c mice: CD4+ T cells are important in control of SARS-CoV infection.A mouse model of lethal infection for evaluating prophylactics and therapeutics against Monkeypox virus.Lambda interferon renders epithelial cells of the respiratory and gastrointestinal tracts resistant to viral infections Coronavirus pathogenesis and the emerging pathogen severe acute respiratory syndrome coronavirus.An immunosuppressed Syrian golden hamster model for SARS-CoV infection.Emerging respiratory viruses: challenges and vaccine strategies Severe acute respiratory syndrome coronavirus infection of mice transgenic for the human Angiotensin-converting enzyme 2 virus receptor.BoHV-4-Based Vector Single Heterologous Antigen Delivery Protects STAT1(-/-) Mice from Monkeypoxvirus Lethal Challenge.Development of animal models against emerging coronaviruses: From SARS to MERS coronavirus A review of genetic methods and models for analysis of coronavirus-induced severe pneumonitis.Vaccines to prevent severe acute respiratory syndrome coronavirus-induced disease.Genomic analysis reveals age-dependent innate immune responses to severe acute respiratory syndrome coronavirus Noncytolytic clearance of sindbis virus infection from neurons by gamma interferon is dependent on Jak/STAT signaling.The discovery of angiotensin-converting enzyme 2 and its role in acute lung injury in mice.Toll-like receptor 4 deficiency increases disease and mortality after mouse hepatitis virus type 1 infection of susceptible C3H mice SARS vaccines: where are we?Monkeypox virus infections in small animal models for evaluation of anti-poxvirus agents.Animal models in virus research: their utility and limitations.STAT2 and IRF9: Beyond ISGF3.Using siRNA in prophylactic and therapeutic regimens against SARS coronavirus in Rhesus macaque.The SARS-CoV ferret model in an infection-challenge study.Unraveling the complexities of the interferon response during SARS-CoV infection Severe acute respiratory syndrome coronavirus evades antiviral signaling: role of nsp1 and rational design of an attenuated strain.Severe acute respiratory syndrome coronavirus gene 7 products contribute to virus-induced apoptosis.Severe acute respiratory syndrome coronavirus open reading frame (ORF) 3b, ORF 6, and nucleocapsid proteins function as interferon antagonists.Pathology of immunodeficient mice with naturally occurring murine norovirus infection.Severe acute respiratory syndrome coronavirus ORF6 antagonizes STAT1 function by sequestering nuclear import factors on the rough endoplasmic reticulum/Golgi membrane.Animal models and vaccines for SARS-CoV infection.Modeling pathogenesis of emergent and pre-emergent human coronaviruses in mice

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Resolution of primary severe acute respiratory syndrome-associated coronavirus infection requires Stat1.

http://www.wikidata.org/entity/Q37567955

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article científic

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bilimsel makale

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scientific article published on October 2004

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vědecký článek

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Resolution of primary severe a ...... irus infection requires Stat1.

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Resolution of primary severe a ...... irus infection requires Stat1.

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Resolution of primary severe a ...... irus infection requires Stat1.

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JVI.78.20.11416-11421.2004

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Journal of Virology

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Resolution of primary severe a ...... irus infection requires Stat1.

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Douglas Flieder

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Guangping Gao

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Jason Paragas

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Julie Boyer

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Nelson A Wivel

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Peter Bell

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Robert J Hogan

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Ronald G Crystal

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Thomas G Voss

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Thomas Rowe

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P2860

Coronavirus as a possible cause of severe acute respiratory syndrome

Targeted disruption of the mouse Stat1 gene results in compromised innate immunity to viral disease

Targeted disruption of the Stat1 gene in mice reveals unexpected physiologic specificity in the JAK-STAT signaling pathway

A novel coronavirus associated with severe acute respiratory syndrome

Mechanisms and enzymes involved in SARS coronavirus genome expression.

Pulmonary pathological features in coronavirus associated severe acute respiratory syndrome (SARS).

Prior infection and passive transfer of neutralizing antibody prevent replication of severe acute respiratory syndrome coronavirus in the respiratory tract of mice.

Characterization of the budding compartment of mouse hepatitis virus: evidence that transport from the RER to the Golgi complex requires only one vesicular transport step.

Two types of virus-related particles are found during transmissible gastroenteritis virus morphogenesis.

A DNA vaccine induces SARS coronavirus neutralization and protective immunity in mice.

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11416-11421

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scholarly article

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10.1128/JVI.78.20.11416-11421.2004

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Gary P. Kobinger

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2004-10-01T00:00:00Z

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8263638

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15452265

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