Axonal degeneration in multiple sclerosis: the mitochondrial hypothesis. - Wikidata - awgldk - TriplyDB

Axonal degeneration in multiple sclerosis: the mitochondrial hypothesis.

Axonal degeneration in multiple sclerosis: the mitochondrial hypothesis.

http://www.wikidata.org/entity/Q37576097

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Neurodegeneration in multiple sclerosis: novel treatment strategies Myelin damage and repair in pathologic CNS: challenges and prospects Optimizing therapeutics in the management of patients with multiple sclerosis: a review of drug efficacy, dosing, and mechanisms of action Nutrition facts in multiple sclerosis Oxidative stress and proinflammatory cytokines contribute to demyelination and axonal damage in a cerebellar culture model of neuroinflammation Diffusion fMRI detects white-matter dysfunction in mice with acute optic neuritis Axonal transport rate decreased at the onset of optic neuritis in EAE mice The contribution of immune and glial cell types in experimental autoimmune encephalomyelitis and multiple sclerosis Is mPTP the gatekeeper for necrosis, apoptosis, or both?Autophagy, inflammation and neurodegenerative disease.Adipocytokine profile, cytokine levels and foxp3 expression in multiple sclerosis: a possible link to susceptibility and clinical course of disease.Walking Speed and Brain Glucose Uptake are Uncoupled in Patients with Multiple Sclerosis Impaired cerebrovascular reactivity in multiple sclerosis Activation of the mitochondrial permeability transition pore modulates Ca2+ responses to physiological stimuli in adult neurons.Bioenergetics of the spinal cord in experimental autoimmune encephalitis of rats.Genetic inactivation of the p66 isoform of ShcA is neuroprotective in a murine model of multiple sclerosis.Characterizing brain oxygen metabolism in patients with multiple sclerosis with T2-relaxation-under-spin-tagging MRI.CXCR7 antagonism prevents axonal injury during experimental autoimmune encephalomyelitis as revealed by in vivo axial diffusivity Local translation of extranuclear lamin B promotes axon maintenance Genetic inactivation of mitochondria-targeted redox enzyme p66ShcA preserves neuronal viability and mitochondrial integrity in response to oxidative challenges.Transgenic inhibition of astroglial NF-κB protects from optic nerve damage and retinal ganglion cell loss in experimental optic neuritis.Oxidative stress modulates mitochondrial failure and cyclophilin D function in X-linked adrenoleukodystrophy Translocator protein 18 kDa (TSPO) expression in multiple sclerosis patients Ganglion cell loss in relation to visual disability in multiple sclerosis.PATHOLOGIES OF AXONAL TRANSPORT IN NEURODEGENERATIVE DISEASES Dimethyl fumarate may still have a role in progressive multiple sclerosis.White-matter astrocytes, axonal energy metabolism, and axonal degeneration in multiple sclerosis What can we learn about stroke from retinal ischemia models?Mitochondrial channels: ion fluxes and more.Remyelination and multiple sclerosis: therapeutic approaches and challenges.Remyelinating strategies in multiple sclerosis.Targeting mitochondrial function to protect against vision loss.Secondary Progressive Multiple Sclerosis: Definition and Measurement.SIRT1 and NAD+ precursors: Therapeutic targets in multiple sclerosis a review.Causal Effect of Genetic Variants Associated With Body Mass Index on Multiple Sclerosis Susceptibility.Modulating inflammation and neuroprotection in multiple sclerosis.Retinal Findings on OCT in Systemic Conditions.Restricted diffusion preceding gadolinium enhancement in large or tumefactive demyelinating lesions.Chronic progressive multiple sclerosis - pathogenesis of neurodegeneration and therapeutic strategies.Neuroprotective effects of crocin on the histopathological alterations following brain ischemia-reperfusion injury in rat.

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Axonal degeneration in multiple sclerosis: the mitochondrial hypothesis.

http://www.wikidata.org/entity/Q37576097

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on September 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Axonal degeneration in multiple sclerosis: the mitochondrial hypothesis.

@en

Axonal degeneration in multiple sclerosis: the mitochondrial hypothesis.

@nl

type

label

Axonal degeneration in multiple sclerosis: the mitochondrial hypothesis.

@en

Axonal degeneration in multiple sclerosis: the mitochondrial hypothesis.

@nl

prefLabel

Axonal degeneration in multiple sclerosis: the mitochondrial hypothesis.

@en

Axonal degeneration in multiple sclerosis: the mitochondrial hypothesis.

@nl

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S11910-009-0060-3

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Current Neurology and Neuroscience Reports

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Axonal degeneration in multiple sclerosis: the mitochondrial hypothesis.

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Dennis Bourdette

http://www.w3.org/2001/XMLSchema#string

Gary Banker

http://www.w3.org/2001/XMLSchema#string

Kimmy G Su

http://www.w3.org/2001/XMLSchema#string

Michael Forte

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P2860

APP binds DR6 to trigger axon pruning and neuron death via distinct caspases

The mechanism of Ca2+ -dependent regulation of kinesin-mediated mitochondrial motility

Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death

Cyclophilin D is a component of mitochondrial permeability transition and mediates neuronal cell death after focal cerebral ischemia

Axonal transection in the lesions of multiple sclerosis

Multiple sclerosis

Nitric oxide and the immune response

Candidate autoantigens in multiple sclerosis.

Unwrapping new layers of complexity in axon/glial relationships.

Molecular changes in neurons in multiple sclerosis: altered axonal expression of Nav1.2 and Nav1.6 sodium channels and Na+/Ca2+ exchanger

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s11910-009-0060-3

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411-417

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10.1007/S11910-009-0060-3

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multiple sclerosis

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