Paracrine and autocrine signals induce and maintain mesenchymal and stem cell states in the breast. - Wikidata - awgldk - TriplyDB

Paracrine and autocrine signals induce and maintain mesenchymal and stem cell states in the breast.

Paracrine and autocrine signals induce and maintain mesenchymal and stem cell states in the breast.

http://www.wikidata.org/entity/Q37595164

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Transcription factors OVOL1 and OVOL2 induce the mesenchymal to epithelial transition in human cancer What does the concept of the stem cell niche really mean today?A noncanonical Frizzled2 pathway regulates epithelial-mesenchymal transition and metastasis Protein kinase C α is a central signaling node and therapeutic target for breast cancer stem cells Disrupting the interaction of BRD4 with diacetylated Twist suppresses tumorigenesis in basal-like breast cancer The hedgehog pathway in triple-negative breast cancer Pancreatic cancer stem cell markers and exosomes - the incentive push Anti-Cancer Stem-like Cell Compounds in Clinical Development - An Overview and Critical Appraisal Mechanisms of TGFβ-Induced Epithelial-Mesenchymal Transition Wnt Signaling in Cancer Stem Cell Biology Stem cell technology in breast cancer: current status and potential applications Epithelial-mesenchymal transition in gastric cancer Increased Oxidative Stress as a Selective Anticancer Therapy The cancer stem cell niche: how essential is the niche in regulating stemness of tumor cells?Cancer heterogeneity--a multifaceted view Breast cancer epithelial-to-mesenchymal transition: examining the functional consequences of plasticity Identifying and targeting tumor-initiating cells in the treatment of breast cancer The Roles of Epithelial-to-Mesenchymal Transition (EMT) and Mesenchymal-to-Epithelial Transition (MET) in Breast Cancer Bone Metastasis: Potential Targets for Prevention and Treatment CD95 maintains stem cell-like and non-classical EMT programs in primary human glioblastoma cells In vitro models of cancer stem cells and clinical applications Transforming growth factor β as regulator of cancer stemness and metastasis Targeting the VEGF-C/VEGFR3 axis suppresses Slug-mediated cancer metastasis and stemness via inhibition of KRAS/YAP1 signaling Epithelial-Mesenchymal Plasticity: A Central Regulator of Cancer Progression MicroRNAs in breast cancer: oncogene and tumor suppressors with clinical potential TGFβ Signaling in Tumor Initiation, Epithelial-to-Mesenchymal Transition, and Metastasis TGF-β family signaling in stem cells TGFβ signalling in context On the origin and destination of cancer stem cells: a conceptual evaluation Grainyhead-like 2 Reverses the Metabolic Changes Induced by the Oncogenic Epithelial-Mesenchymal Transition: Effects on Anoikis Both canonical and non-canonical Wnt signaling independently promote stem cell growth in mammospheres De-differentiation confers multidrug resistance via noncanonical PERK-Nrf2 signaling Microenvironmental Modulation of Decorin and Lumican in Temozolomide-Resistant Glioblastoma and Neuroblastoma Cancer Stem-Like Cells Epithelial-mesenchymal transition and tumor suppression are controlled by a reciprocal feedback loop between ZEB1 and Grainyhead-like-2 Upregulated LASP-1 correlates with a malignant phenotype and its potential therapeutic role in human cholangiocarcinoma.Activin upregulation by NF-κB is required to maintain mesenchymal features of cancer stem-like cells in non-small cell lung cancer.Loss of BRMS1 promotes a mesenchymal phenotype through NF-κB-dependent regulation of Twist1.Pigment Epithelium-Derived Factor (PEDF) Expression Induced by EGFRvIII Promotes Self-renewal and Tumor Progression of Glioma Stem Cells.Silencing of miR-148a in cancer-associated fibroblasts results in WNT10B-mediated stimulation of tumor cell motility.NF-κB regulates mesenchymal transition for the induction of non-small cell lung cancer initiating cells.VEGF/neuropilin-2 regulation of Bmi-1 and consequent repression of IGF-IR define a novel mechanism of aggressive prostate cancer

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Paracrine and autocrine signals induce and maintain mesenchymal and stem cell states in the breast.

http://www.wikidata.org/entity/Q37595164

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on June 2011

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

@cs

name

Paracrine and autocrine signal ...... tem cell states in the breast.

@en

Paracrine and autocrine signal ...... tem cell states in the breast.

@nl

type

label

Paracrine and autocrine signal ...... tem cell states in the breast.

@en

Paracrine and autocrine signal ...... tem cell states in the breast.

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prefLabel

Paracrine and autocrine signal ...... tem cell states in the breast.

@en

Paracrine and autocrine signal ...... tem cell states in the breast.

@nl

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J.CELL.2011.04.029

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P1476

Paracrine and autocrine signal ...... tem cell states in the breast.

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Andrea L Richardson

http://www.w3.org/2001/XMLSchema#string

Christina Scheel

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Christine L Chaffer

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Elinor Ng Eaton

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Jeffrey Rubin

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Kong-Jie Kah

http://www.w3.org/2001/XMLSchema#string

Wenjun Guo

http://www.w3.org/2001/XMLSchema#string

P2860

Autocrine and paracrine angiopoietin 1/Tie-2 signaling promotes muscle satellite cell self-renewal

The epithelial-mesenchymal transition generates cells with properties of stem cells

Induction by transforming growth factor-beta1 of epithelial to mesenchymal transition is a rare event in vitro

Epithelial-mesenchymal transitions in development and disease

Twist, a master regulator of morphogenesis, plays an essential role in tumor metastasis

Epigenetic inactivation of SFRP genes allows constitutive WNT signaling in colorectal cancer

Opinion: migrating cancer stem cells - an integrated concept of malignant tumour progression

Stem cell-derived neural stem/progenitor cell supporting factor is an autocrine/paracrine survival factor for adult neural stem/progenitor cells

Loss of E-cadherin promotes metastasis via multiple downstream transcriptional pathways

EMT, cancer stem cells and drug resistance: an emerging axis of evil in the war on cancer

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926-940

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scholarly article

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10.1016/J.CELL.2011.04.029

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6

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145

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Ferenc Reinhardt

Sophia Hsin-Jung Li

Robert Weinberg

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2011-06-01T00:00:00Z

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51210201

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21663795

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3930331

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