Virus-triggered acquired immunodeficiency by cytotoxic T-cell-dependent destruction of antigen-presenting cells and lymph follicle structure
about
Immune response and immunopathology during toxoplasmosisViral immune evasion due to persistence of activated T cells without effector functionDirect interaction of hepatitis C virus core protein with the cellular lymphotoxin-beta receptor modulates the signal pathway of the lymphotoxin-beta receptorSpecific remodeling of splenic architecture by cytomegalovirus.Cytotoxic granule secretion by lymphocytes and its link to immune homeostasisHepatitis C virus (HCV)-specific cytotoxic T lymphocytes recognize epitopes in the core and envelope proteins of HCVLaminins affect T cell trafficking and allograft fate.Immunodeficiency of alymphoplasia mice (aly/aly) in vivo: structural defect of secondary lymphoid organs and functional B cell defect.Anti-viral strategies of cytotoxic T lymphocytes are manifested through a variety of granule-bound pathways of apoptosis induction.Differences in affinity of binding of lymphocytic choriomeningitis virus strains to the cellular receptor alpha-dystroglycan correlate with viral tropism and disease kinetics.Apoptosis and the homeostatic control of immune responses.Destruction of lymphoid organ architecture and hepatitis caused by CD4+ T cells.Revisiting the B-cell compartment in mouse and humans: more than one B-cell subset exists in the marginal zone and beyond.Point mutation in the glycoprotein of lymphocytic choriomeningitis virus is necessary for receptor binding, dendritic cell infection, and long-term persistence.Uncertainties - discrepancies in immunology.Early virus-host interactions dictate the course of a persistent infectionRole of an intact splenic microarchitecture in early lymphocytic choriomeningitis virus production.Viral persistence redirects CD4 T cell differentiation toward T follicular helper cellsViral persistence in vivo through selection of neutralizing antibody-escape variants.Perforin is a critical physiologic regulator of T-cell activation.Direct stimulation of naive T cells by membrane vesicles from antigen-presenting cells: distinct roles for CD54 and B7 moleculesEnhanced CD8 T cell responses through GITR-mediated costimulation resolve chronic viral infection.Infection of dendritic cells by lymphocytic choriomeningitis virus.Blockade of interferon Beta, but not interferon alpha, signaling controls persistent viral infectionPersistence of lymphocytic choriomeningitis virus at very low levels in immune mice.A role for tumor necrosis factor-alpha in remodeling the splenic marginal zone during Leishmania donovani infection.Virus-induced immunosuppression: immune system-mediated destruction of virus-infected dendritic cells results in generalized immune suppression.Interleukin-6 exacerbates glomerulonephritis in (NZB x NZW)F1 mice.Increased Expression of CD169 on Blood Monocytes and Its Regulation by Virus and CD8 T Cells in Macaque Models of HIV Infection and AIDS.Murine infection with lymphocytic choriomeningitis virus following gastric inoculation.Viral targeting of fibroblastic reticular cells contributes to immunosuppression and persistence during chronic infection.Elimination of chronic viral infection by blocking CD27 signaling.Early antibodies specific for the neutralizing epitope on the receptor binding subunit of the lymphocytic choriomeningitis virus glycoprotein fail to neutralize the virus.Induction of long-lived germinal centers associated with persisting antigen after viral infection.Immunosuppression and resultant viral persistence by specific viral targeting of dendritic cellsExpression of the serpin serine protease inhibitor 6 protects dendritic cells from cytotoxic T lymphocyte-induced apoptosis: differential modulation by T helper type 1 and type 2 cells.On the key role of secondary lymphoid organs in antiviral immune responses studied in alymphoplastic (aly/aly) and spleenless (Hox11(-)/-) mutant mice.Repeated stimulation of CD4 effector T cells can limit their protective function.Enhanced establishment of a virus carrier state in adult CD4+ T-cell-deficient mice.Overexpression of lymphotoxin in T cells induces fulminant thymic involution.
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P2860
Virus-triggered acquired immunodeficiency by cytotoxic T-cell-dependent destruction of antigen-presenting cells and lymph follicle structure
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on September 1991
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Virus-triggered acquired immun ...... s and lymph follicle structure
@en
Virus-triggered acquired immun ...... and lymph follicle structure.
@nl
type
label
Virus-triggered acquired immun ...... s and lymph follicle structure
@en
Virus-triggered acquired immun ...... and lymph follicle structure.
@nl
prefLabel
Virus-triggered acquired immun ...... s and lymph follicle structure
@en
Virus-triggered acquired immun ...... and lymph follicle structure.
@nl
P2093
P2860
P356
P1476
Virus-triggered acquired immun ...... s and lymph follicle structure
@en
P2093
Hengartner H
Odermatt B
Zinkernagel RM
P2860
P304
P356
10.1073/PNAS.88.18.8252
P407
P577
1991-09-01T00:00:00Z