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Mitochondrial reactive oxygen species regulate hypoxic signaling.

Mitochondrial reactive oxygen species regulate hypoxic signaling.

http://www.wikidata.org/entity/Q37603588

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AMP-activated protein kinase, stress responses and cardiovascular diseases Targeting Cancer Metabolism - Revisiting the Warburg Effects Reactive oxygen species, nutrition, hypoxia and diseases: Problems solved?Targeting CSCs in tumor microenvironment: the potential role of ROS-associated miRNAs in tumor aggressiveness Mitochondrial oxidative stress in aging and healthspan Mitochondria as a target in treatment NADPH oxidase subunit 4-mediated reactive oxygen species contribute to cycling hypoxia-promoted tumor progression in glioblastoma multiforme A HIF-1 target, ATIA, protects cells from apoptosis by modulating the mitochondrial thioredoxin, TRX2 Mitochondrial genome instability and ROS enhance intestinal tumorigenesis in APC(Min/+) mice Recent Progress in Research on the Pathogenesis of Pulmonary Thromboembolism: An Old Story with New Perspectives.Mitochondrial reactive oxygen species promote epidermal differentiation and hair follicle development.Caloric Restriction-Induced Extension of Chronological Lifespan Requires Intact Respiration in Budding Yeast.Bioenergetic analysis of ovarian cancer cell lines: profiling of histological subtypes and identification of a mitochondria-defective cell line.The alternative medicine pawpaw and its acetogenin constituents suppress tumor angiogenesis via the HIF-1/VEGF pathway Enhancement of reactive oxygen species production and chlamydial infection by the mitochondrial Nod-like family member NLRX1.Hypoxia-adaptation involves mitochondrial metabolic depression and decreased ROS leakage ECM-dependent HIF induction directs trophoblast stem cell fate via LIMK1-mediated cytoskeletal rearrangement.SIRT3 opposes reprogramming of cancer cell metabolism through HIF1α destabilization Functional implications of mitochondrial reactive oxygen species generated by oncogenic viruses Binge alcohol promotes hypoxic liver injury through a CYP2E1-HIF-1α-dependent apoptosis pathway in mice and humans Regulation of yeast chronological life span by TORC1 via adaptive mitochondrial ROS signaling.Perinatal oxygen in the developing lung.Networked-based characterization of extracellular matrix proteins from adult mouse pulmonary and aortic valves.Mitochondria as signaling organelles.Reactive oxygen species alter autocrine and paracrine signaling.Cardiac aging: from molecular mechanisms to significance in human health and disease Promoter G-quadruplex sequences are targets for base oxidation and strand cleavage during hypoxia-induced transcription Forkhead Transcription Factor 3a (FOXO3a) Modulates Hypoxia Signaling via Up-regulation of the von Hippel-Lindau Gene (VHL).Oroxylin A inhibits glycolysis-dependent proliferation of human breast cancer via promoting SIRT3-mediated SOD2 transcription and HIF1α destabilization Oxygen versus Reactive Oxygen in the Regulation of HIF-1α: The Balance Tips Structures and mechanisms of antitumor agents: xestoquinones uncouple cellular respiration and disrupt HIF signaling in human breast tumor cells.VDAC1: from structure to cancer therapy.Aged garlic extract and S-allylcysteine prevent apoptotic cell death in a chemical hypoxia model.Targeting Tyrosine Kinase Inhibitor-Resistant Non-Small Cell Lung Cancer by Inducing Epidermal Growth Factor Receptor Degradation via Methionine 790 Oxidation Iron uptake controls the generation of Leishmania infective forms through regulation of ROS levels.Peroxisome-proliferator-activated receptors regulate redox signaling in the cardiovascular system Brusatol inhibits HIF-1 signaling pathway and suppresses glucose uptake under hypoxic conditions in HCT116 cells.Hypoxia and hypoxia-inducible factors as regulators of T cell development, differentiation, and function.Cancer metabolism: what we can learn from proteomic analysis by mass spectrometry.New role for an old rule: N-end rule-mediated degradation of ethylene responsive factor proteins governs low oxygen response in plants(F).

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Mitochondrial reactive oxygen species regulate hypoxic signaling.

http://www.wikidata.org/entity/Q37603588

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 24 September 2009

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Mitochondrial reactive oxygen species regulate hypoxic signaling.

@en

Mitochondrial reactive oxygen species regulate hypoxic signaling.

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type

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Mitochondrial reactive oxygen species regulate hypoxic signaling.

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Mitochondrial reactive oxygen species regulate hypoxic signaling.

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prefLabel

Mitochondrial reactive oxygen species regulate hypoxic signaling.

@en

Mitochondrial reactive oxygen species regulate hypoxic signaling.

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J.CEB.2009.08.005

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Current Opinion in Cell Biology

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Mitochondrial reactive oxygen species regulate hypoxic signaling.

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Navdeep S Chandel

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Robert B Hamanaka

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P2860

The tumour suppressor protein VHL targets hypoxia-inducible factors for oxygen-dependent proteolysis

HIFalpha targeted for VHL-mediated destruction by proline hydroxylation: implications for O2 sensing

C. elegans EGL-9 and mammalian homologs define a family of dioxygenases that regulate HIF by prolyl hydroxylation

A conserved family of prolyl-4-hydroxylases that modify HIF

A nuclear factor induced by hypoxia via de novo protein synthesis binds to the human erythropoietin gene enhancer at a site required for transcriptional activation

Hypoxia-inducible factor 1 is a basic-helix-loop-helix-PAS heterodimer regulated by cellular O2 tension

Endothelial PAS domain protein 1 (EPAS1), a transcription factor selectively expressed in endothelial cells

How mitochondria produce reactive oxygen species

Regulation of hypoxia-inducible factor 1alpha is mediated by an O2-dependent degradation domain via the ubiquitin-proteasome pathway

Targeting of HIF-alpha to the von Hippel-Lindau ubiquitylation complex by O2-regulated prolyl hydroxylation

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scholarly article

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10.1016/J.CEB.2009.08.005

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