Nephropathy and elevated BP in mice with podocyte-specific NADPH oxidase 5 expression.
about
The balance of powers: Redox regulation of fibrogenic pathways in kidney injuryCalcium and ROS: A mutual interplayRedox signaling, Nox5 and vascular remodeling in hypertensionNADPH Oxidases: Insights into Selected Functions and Mechanisms of Action in Cancer and Stem Cells.Nox-4 deletion reduces oxidative stress and injury by PKC-α-associated mechanisms in diabetic nephropathy.Impact of Nox5 polymorphisms on basal and stimulus-dependent ROS generation.Upstream regulators and downstream effectors of NADPH oxidases as novel therapeutic targets for diabetic kidney disease.Targeting NADPH oxidase with a novel dual Nox1/Nox4 inhibitor attenuates renal pathology in type 1 diabetes.Endothelial Gata5 transcription factor regulates blood pressure.Metabolomics Reveals a Key Role for Fumarate in Mediating the Effects of NADPH Oxidase 4 in Diabetic Kidney DiseaseEnzymatic regulation and functional relevance of NOX5Oxidative stress and autophagy: crucial modulators of kidney injuryDiabetes and Kidney Disease: Role of Oxidative Stress.NADPH oxidase in brain injury and neurodegenerative disordersNox and renal disease.Nox5 stability and superoxide production is regulated by C-terminal binding of Hsp90 and CO-chaperones.APX-115, a first-in-class pan-NADPH oxidase (Nox) inhibitor, protects db/db mice from renal injury.Are reactive oxygen species still the basis for diabetic complications?Use of Biomarkers in the Evaluation and Treatment of Hypertensive Patients.Oxidative Stress and Hypertensive Diseases.Oxidative Stress in Atherosclerosis.Podocyte-specific Nox4 deletion affords renoprotection in a mouse model of diabetic nephropathy.Vascular injury in diabetic db/db mice is ameliorated by atorvastatin: role of Rac1/2-sensitive Nox-dependent pathways.NDRG2 contributes to cisplatin sensitivity through modulation of BAK-to-Mcl-1 ratio.Diabetes, Hypertension, and Cardiovascular Disease: Clinical Insights and Vascular Mechanisms.Oxidative Stress and Renal Fibrosis: Recent Insights for the Development of Novel Therapeutic Strategies.Recent novel approaches to limit oxidative stress and inflammation in diabetic complications.NADPH Oxidase 5 Is a Pro-Contractile Nox Isoform and a Point of Cross-Talk for Calcium and Redox Signaling-Implications in Vascular Function
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P2860
Nephropathy and elevated BP in mice with podocyte-specific NADPH oxidase 5 expression.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年学术文章
@wuu
2013年学术文章
@zh-cn
2013年学术文章
@zh-hans
2013年学术文章
@zh-my
2013年学术文章
@zh-sg
2013年學術文章
@yue
2013年學術文章
@zh
2013年學術文章
@zh-hant
name
Nephropathy and elevated BP in mice with podocyte-specific NADPH oxidase 5 expression.
@en
Nephropathy and elevated BP in mice with podocyte-specific NADPH oxidase 5 expression.
@nl
type
label
Nephropathy and elevated BP in mice with podocyte-specific NADPH oxidase 5 expression.
@en
Nephropathy and elevated BP in mice with podocyte-specific NADPH oxidase 5 expression.
@nl
prefLabel
Nephropathy and elevated BP in mice with podocyte-specific NADPH oxidase 5 expression.
@en
Nephropathy and elevated BP in mice with podocyte-specific NADPH oxidase 5 expression.
@nl
P2093
P2860
P356
P1476
Nephropathy and elevated BP in mice with podocyte-specific NADPH oxidase 5 expression.
@en
P2093
Alex Gutsol
Augusto C Montezano
Chelsea Towaij
Christopher R J Kennedy
Jean-François Thibodeau
Mark E Cooper
Robin J Parks
P2860
P304
P356
10.1681/ASN.2013040371
P577
2013-11-21T00:00:00Z