Sphingolipids: agents provocateurs in the pathogenesis of insulin resistance.
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Mechanisms of intrahepatic triglyceride accumulationDefect of insulin signal in peripheral tissues: Important role of ceramideRole of ceramide in diabetes mellitus: evidence and mechanisms.Sphingolipids in obesity, type 2 diabetes, and metabolic disease.Characterising the inhibitory actions of ceramide upon insulin signaling in different skeletal muscle cell models: a mechanistic insightMetabolic derangements mediate cognitive impairment and Alzheimer's disease: role of peripheral insulin-resistance diseasesEffects of inhibition of serine palmitoyltransferase (SPT) and sphingosine kinase 1 (SphK1) on palmitate induced insulin resistance in L6 myotubesPlasma metabolomic profiling of patients with diabetes-associated cognitive decline.Mechanisms underlying skeletal muscle insulin resistance induced by fatty acids: importance of the mitochondrial functionBrain metabolic dysfunction at the core of Alzheimer's disease.Triangulated mal-signaling in Alzheimer's disease: roles of neurotoxic ceramides, ER stress, and insulin resistance reviewedEnglerin A induces an acute inflammatory response and reveals lipid metabolism and ER stress as targetable vulnerabilities in renal cell carcinomaRegulation of plasma ceramide levels with fatty acid oversupply: evidence that the liver detects and secretes de novo synthesised ceramide.Plasma sphingolipids are biomarkers of metabolic syndrome in non-human primates maintained on a Western-style diet.Mitochondrial substrate availability and its role in lipid-induced insulin resistance and proinflammatory signaling in skeletal muscle.Sphingolipid metabolism and its role in the skeletal tissues.Lipid-mediated muscle insulin resistance: different fat, different pathways?Ceramide and insulin resistance: how should the issue be approached?β cell membrane remodelling and procoagulant events occur in inflammation-driven insulin impairment: a GLP-1 receptor dependent and independent control.Mevalonate inhibits acid sphingomyelinase activity, increases sphingomyelin levels and inhibits cell proliferation of HepG2 and Caco-2 cells.Sphingolipids in obesity and related complications.Effect of plasma free fatty acid supply on the rate of ceramide synthesis in different muscle types in the rat.Ceramide accumulation in L6 skeletal muscle cells due to increased activity of ceramide synthase isoforms has opposing effects on insulin action to those caused by palmitate treatment.GM3 ganglioside and phosphatidylethanolamine-containing lipids are adipose tissue markers of insulin resistance in obese women.Ceramide content is higher in type I compared to type II fibers in obesity and type 2 diabetes mellitus.
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P2860
Sphingolipids: agents provocateurs in the pathogenesis of insulin resistance.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 06 April 2011
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Sphingolipids: agents provocateurs in the pathogenesis of insulin resistance.
@en
Sphingolipids: agents provocateurs in the pathogenesis of insulin resistance.
@nl
type
label
Sphingolipids: agents provocateurs in the pathogenesis of insulin resistance.
@en
Sphingolipids: agents provocateurs in the pathogenesis of insulin resistance.
@nl
prefLabel
Sphingolipids: agents provocateurs in the pathogenesis of insulin resistance.
@en
Sphingolipids: agents provocateurs in the pathogenesis of insulin resistance.
@nl
P2860
P1433
P1476
Sphingolipids: agents provocateurs in the pathogenesis of insulin resistance.
@en
P2093
P2860
P2888
P304
P356
10.1007/S00125-011-2127-3
P577
2011-04-06T00:00:00Z