NADPH oxidases as therapeutic targets in ischemic stroke.
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A Review of the Mechanisms of Blood-Brain Barrier Permeability by Tissue-Type Plasminogen Activator Treatment for Cerebral IschemiaCardiovascular and Hepatic Toxicity of Cocaine: Potential Beneficial Effects of Modulators of Oxidative StressOxidative Stress and the Use of Antioxidants in StrokePathophysiology and Treatments of Oxidative Injury in Ischemic Stroke: Focus on the Phagocytic NADPH Oxidase 2Dual and Opposing Roles of MicroRNA-124 in Epilepsy Are Mediated through Inflammatory and NRSF-Dependent Gene Networks.Revisiting cerebral postischemic reperfusion injury: new insights in understanding reperfusion failure, hemorrhage, and edema.Ischemia-reperfusion injury in strokeProtective and antioxidant effects of PPARα in the ischemic retina.NADPH oxidase-2: linking glucose, acidosis, and excitotoxicity in stroke.Reactive oxygen species prevent imiquimod-induced psoriatic dermatitis through enhancing regulatory T cell functionMyeloperoxidase propagates damage and is a potential therapeutic target for subacute stroke.Attenuation of experimental colitis in glutathione peroxidase 1 and catalase double knockout mice through enhancing regulatory T cell functionActivation of α-7 nicotinic acetylcholine receptor reduces ischemic stroke injury through reduction of pro-inflammatory macrophages and oxidative stress.Acupuncture elicits neuroprotective effect by inhibiting NAPDH oxidase-mediated reactive oxygen species production in cerebral ischaemiaWhich NADPH oxidase isoform is relevant for ischemic stroke? The case for nox 2.Amputation-induced reactive oxygen species are required for successful Xenopus tadpole tail regeneration.Intracellular pH reduction prevents excitotoxic and ischemic neuronal death by inhibiting NADPH oxidase.Dimethyl Fumarate and Monomethyl Fumarate Promote Post-Ischemic Recovery in Mice.NADPH oxidase and the degeneration of dopaminergic neurons in parkinsonian mice.Redox signaling in cardiovascular health and disease.Pretreated quercetin protects gerbil hippocampal CA1 pyramidal neurons from transient cerebral ischemic injury by increasing the expression of antioxidant enzymesUnderstanding the biology of reactive oxygen species and their link to cancer: NADPH oxidases as novel pharmacological targets.Crosstalk Between Endoplasmic Reticulum Stress, Oxidative Stress, and Autophagy: Potential Therapeutic Targets for Acute CNS Injuries.Time-course and intensity-based classifications of oxidative stresses and their potential application in biomedical, comparative and environmental research.Gastrodin improves cognitive dysfunction and decreases oxidative stress in vascular dementia rats induced by chronic ischemia.Puerarin attenuates cognitive dysfunction and oxidative stress in vascular dementia rats induced by chronic ischemiaHigh-glucose-increased expression and activation of NADPH oxidase in human vascular smooth muscle cells is mediated by 4-hydroxynonenal-activated PPARα and PPARβ/δ.Nrf2-a Promising Therapeutic Target for Defensing Against Oxidative Stress in Stroke.Covalent adduct formation between the plasmalogen-derived modification product 2-chlorohexadecanal and phloretin.IL-17A Enhances Microglial Response to OGD by Regulating p53 and PI3K/Akt Pathways with Involvement of ROS/HMGB1.Downregulation of thioredoxin reductase 1 expression in the substantia nigra pars compacta of Parkinson's disease mice.Renal denervation prevents stroke and brain injury via attenuation of oxidative stress in hypertensive rats.Nox4 contributes to the hypoxia-mediated regulation of actin cytoskeleton in cerebrovascular smooth muscleAlterations in the time course of expression of the Nox family in the brain in a rat experimental cerebral ischemia and reperfusion model: effects of melatonin.Activation of T-LAK-cell-originated protein kinase-mediated antioxidation protects against focal cerebral ischemia-reperfusion injury.Suitable Concentrations of Uric Acid Can Reduce Cell Death in Models of OGD and Cerebral Ischemia-Reperfusion Injury.Role of NADPH oxidase in total salvianolic acid injection attenuating ischemia-reperfusion impaired cerebral microcirculation and neurons: implication of AMPK/Akt/PKC.PPAR-γ Ameliorates Neuronal Apoptosis and Ischemic Brain Injury via Suppressing NF-κB-Driven p22phox Transcription.Dietary supplementation of GrandFusion(®) mitigates cerebral ischemia-induced neuronal damage and attenuates inflammation.Deletion of Nrf2 Exacerbates Oxidative Stress After Traumatic Brain Injury in Mice.
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P2860
NADPH oxidases as therapeutic targets in ischemic stroke.
description
article científic
@ca
article scientifique
@fr
articol științific
@ro
articolo scientifico
@it
artigo científico
@gl
artigo científico
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artigo científico
@pt-br
artikel ilmiah
@id
artikull shkencor
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artículo científico
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name
NADPH oxidases as therapeutic targets in ischemic stroke.
@en
type
label
NADPH oxidases as therapeutic targets in ischemic stroke.
@en
prefLabel
NADPH oxidases as therapeutic targets in ischemic stroke.
@en
P2860
P1476
NADPH oxidases as therapeutic targets in ischemic stroke
@en
P2093
Timo Kahles
P2860
P2888
P304
P356
10.1007/S00018-012-1011-8
P577
2012-05-23T00:00:00Z