Review: experimental manipulations of microglia in mouse models of Alzheimer's pathology: activation reduces amyloid but hastens tau pathology.
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Amyloid-beta Alzheimer targets - protein processing, lipid rafts, and amyloid-beta poresThe flavonoid quercetin ameliorates Alzheimer's disease pathology and protects cognitive and emotional function in aged triple transgenic Alzheimer's disease model miceImmunotherapeutic approaches for Alzheimer's diseaseInnate Immunity Fights Alzheimer's DiseaseInvolvement of the Bradykinin B1 Receptor in Microglial Activation: In Vitro and In Vivo Studies.Amyloid β and Tau Alzheimer's disease related pathology is reduced by Toll-like receptor 9 stimulation.Linalool reverses neuropathological and behavioral impairments in old triple transgenic Alzheimer's mice.Identifying biomarkers of dementia prevalent among amnestic mild cognitively impaired ethnic female patients.CB₂ receptor deficiency increases amyloid pathology and alters tau processing in a transgenic mouse model of Alzheimer's disease.Innate Immunity Stimulation via Toll-Like Receptor 9 Ameliorates Vascular Amyloid Pathology in Tg-SwDI Mice with Associated Cognitive Benefits.CB2 receptor deficiency increases amyloid pathology and alters tau processing in a transgenic mouse model of Alzheimer's disease.Microglia, neuroinflammation, and beta-amyloid protein in Alzheimer's disease.Modulation of inflammation in transgenic models of Alzheimer's diseaseEnvironmental factors in the development and progression of late-onset Alzheimer's disease.The Role of TREM2 in Alzheimer's Disease and Other Neurological Disorders.Developing therapeutic vaccines against Alzheimer's disease.IFN-γ promotes τ phosphorylation without affecting mature tangles.Microglial internalization and degradation of pathological tau is enhanced by an anti-tau monoclonal antibody.Fractalkine activates NRF2/NFE2L2 and heme oxygenase 1 to restrain tauopathy-induced microgliosis.The roles of inflammation and immune mechanisms in Alzheimer's disease.Monophosphoryl lipid A is an lipopolysaccharide-derived Toll-like receptor 4 agonist which may improve Alzheimer's disease pathology.Influence of Allergy on Immunoglobulins and Amyloid-β in the Cerebrospinal Fluid of Patients with Alzheimer's Disease.The small molecule CA140 inhibits the neuroinflammatory response in wild-type mice and a mouse model of AD
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P2860
Review: experimental manipulations of microglia in mouse models of Alzheimer's pathology: activation reduces amyloid but hastens tau pathology.
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article científic
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article scientifique
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articol științific
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articolo scientifico
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artigo científico
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artigo científico
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artigo científico
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artikel ilmiah
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artikull shkencor
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artículo científico
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Review: experimental manipulat ...... oid but hastens tau pathology.
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type
label
Review: experimental manipulat ...... oid but hastens tau pathology.
@en
prefLabel
Review: experimental manipulat ...... oid but hastens tau pathology.
@en
P2093
P2860
P921
P356
P1476
Review: experimental manipulat ...... oid but hastens tau pathology.
@en
P2093
P2860
P356
10.1111/NAN.12002
P577
2013-02-01T00:00:00Z