Closely opposed apurinic/apyrimidinic sites are converted to double strand breaks in Escherichia coli even in the absence of exonuclease III, endonuclease IV, nucleotide excision repair and AP lyase cleavage.
about
Reactive oxygen species regulate DNA copy number in isolated yeast mitochondria by triggering recombination-mediated replication.Apex1 can cleave complex clustered DNA lesions in cells.Abasic sites and strand breaks in DNA cause transcriptional mutagenesis in Escherichia coli.Dynamic flexibility of DNA repair pathways in growth arrested Escherichia coliClustered DNA lesion repair in eukaryotes: relevance to mutagenesis and cell survivalDelayed repair of radiation induced clustered DNA damage: friend or foe?Base excision repair processing of abasic site/single-strand break lesions within clustered damage sites associated with XRCC1 deficiency.Human abasic endonuclease action on multilesion abasic clusters: implications for radiation-induced biological damage.DNA tandem lesion repair by strand displacement synthesis and nucleotide excision repair.Artemis is required to improve the accuracy of repair of double-strand breaks with 5'-blocked termini generated from non-DSB-clustered lesions.DNA repair of clustered lesions in mammalian cells: involvement of non-homologous end-joining.Processing of DNA damage clusters in human cells: current status of knowledge.The formation of double-strand breaks at multiply damaged sites is driven by the kinetics of excision/incision at base damage in eukaryotic cells.Replication fork collapse is a major cause of the high mutation frequency at three-base lesion clustersThe yield, processing, and biological consequences of clustered DNA damage induced by ionizing radiation.Increased mutability and decreased repairability of a three-lesion clustered DNA-damaged site comprised of an AP site and bi-stranded 8-oxoG lesions.Hierarchy of lesion processing governs the repair, double-strand break formation and mutability of three-lesion clustered DNA damage.Ape1 guides DNA repair pathway choice that is associated with drug tolerance in glioblastomaProcessing of thymine glycol in a clustered DNA damage site: mutagenic or cytotoxic.The roles of specific glycosylases in determining the mutagenic consequences of clustered DNA base damage.Specific and nonhomologous isofunctional enzymes of the genetic information processing pathways as potential therapeutical targets for tritryps.Genome-wide map of Apn1 binding sites under oxidative stress in Saccharomyces cerevisiae.Nucleotides containing variously modified sugars: energetics, structure, and mechanical properties.
P2860
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P2860
Closely opposed apurinic/apyrimidinic sites are converted to double strand breaks in Escherichia coli even in the absence of exonuclease III, endonuclease IV, nucleotide excision repair and AP lyase cleavage.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
Closely opposed apurinic/apyri ...... repair and AP lyase cleavage.
@en
type
label
Closely opposed apurinic/apyri ...... repair and AP lyase cleavage.
@en
prefLabel
Closely opposed apurinic/apyri ...... repair and AP lyase cleavage.
@en
P2093
P2860
P1433
P1476
Closely opposed apurinic/apyri ...... repair and AP lyase cleavage.
@en
P2093
April M Landry
Katherine L Brame
Laura E Geltz
Lynn Harrison
P2860
P304
P356
10.1016/J.DNAREP.2005.10.009
P577
2005-12-06T00:00:00Z