A dominant-negative strategy for studying roles of G proteins in vivo.
about
Galpha12/13 regulate epiboly by inhibiting E-cadherin activity and modulating the actin cytoskeletonMechanism of activation of protein kinase D2(PKD2) by the CCK(B)/gastrin receptorGenetic analysis of the Drosophila Gs(alpha) geneAntagonist minigenes identify genes regulated by parathyroid hormone through G protein-selective and G protein co-regulated mechanisms in osteoblastic cellsThe G protein-coupled receptor GPR157 regulates neuronal differentiation of radial glial progenitors through the Gq-IP3 pathwayDistinct phospholipase C-β isozymes mediate lysophosphatidic acid receptor 1 effects on intestinal epithelial homeostasis and wound closure.Role of G-proteins in odor-sensing and CO2-sensing neurons in Drosophila.SDF1-induced antagonism of axonal repulsion requires multiple G-protein coupled signaling components that work in parallelRegulation of Gβγi-dependent PLC-β3 activity in smooth muscle: inhibitory phosphorylation of PLC-β3 by PKA and PKG and stimulatory phosphorylation of Gαi-GTPase-activating protein RGS2 by PKG.cAMP-induced expression of neuropilin1 promotes retinal axon crossing in the zebrafish optic chiasm.Dancing with multiple partners.Distinctive G Protein-Dependent Signaling by Protease-Activated Receptor 2 (PAR2) in Smooth Muscle: Feedback Inhibition of RhoA by cAMP-Independent PKA.The extreme C-terminal region of Gαs differentially couples to the luteinizing hormone and beta2-adrenergic receptors.NEURONAL DEVELOPMENT. Glycerophospholipid regulation of modality-specific sensory axon guidance in the spinal cord.The GTP-binding protein RhoA mediates Na,K-ATPase exocytosis in alveolar epithelial cellsRegulation of receptor-coupling to (multiple) G proteins. A challenge for basic research and drug discovery.Essential roles of G{alpha}12/13 signaling in distinct cell behaviors driving zebrafish convergence and extension gastrulation movements.Galpha13 mediates a signal that is essential for proliferation and survival of thymocyte progenitors.Accessibility of cysteine residues substituted into the cytoplasmic regions of the alpha-factor receptor identifies the intracellular residues that are available for G protein interaction.The gep proto-oncogene Gα13 mediates lysophosphatidic acid-mediated migration of pancreatic cancer cells.Regulation of heterotrimeric G protein signaling in airway smooth muscle.Characterization of signaling pathways coupled to melatonin receptors in gastrointestinal smooth muscle.Targeted G-protein inhibition as a novel approach to decrease vagal atrial fibrillation by selective parasympathetic attenuationThe gep proto-oncogene Gα12 mediates LPA-stimulated activation of CREB in ovarian cancer cells.Signal protein-derived peptides as functional probes and regulators of intracellular signaling.A perspective on more effective GPCR-targeted drug discovery efforts.Selective inhibition of heterotrimeric Gs signaling. Targeting the receptor-G protein interface using a peptide minigene encoding the Galpha(s) carboxyl terminus.Inhibitory and stimulatory regulation of Rac and cell motility by the G12/13-Rho and Gi pathways integrated downstream of a single G protein-coupled sphingosine-1-phosphate receptor isoform.Direct involvement of G protein alpha(q/11) subunit in regulation of muscarinic receptor-mediated sAPPalpha release.G alpha COOH-terminal minigene vectors dissect heterotrimeric G protein signaling.Activation of protein kinase D by signaling through the alpha subunit of the heterotrimeric G protein G(q).Effect of n-Alkanols on G-Protein α Subunits.Activation of protein kinase D by signaling through Rho and the alpha subunit of the heterotrimeric G protein G13.CCK-A receptor activates RhoA through G alpha 12/13 in NIH3T3 cells.Alpha-thrombin-mediated phosphatidylinositol 3-kinase activation through release of Gbetagamma dimers from Galphaq and Galphai2.Galphai1 and Galphai3 differentially interact with, and regulate, the G protein-activated K+ channel.Dominant negative effects of a Gbeta mutant on G-protein coupled inward rectifier K+ channel.
P2860
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P2860
A dominant-negative strategy for studying roles of G proteins in vivo.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
1999年學術文章
@zh
1999年學術文章
@zh-hant
name
A dominant-negative strategy for studying roles of G proteins in vivo.
@en
type
label
A dominant-negative strategy for studying roles of G proteins in vivo.
@en
prefLabel
A dominant-negative strategy for studying roles of G proteins in vivo.
@en
P2093
P2860
P356
P1476
A dominant-negative strategy for studying roles of G proteins in vivo
@en
P2093
P2860
P304
P356
10.1074/JBC.274.10.6610
P407
P577
1999-03-01T00:00:00Z