Tumor necrosis factor alpha induces senescence and chromosomal instability in human leukemic cells.
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Therapeutic targeting of replicative immortalityMultivitamin use and telomere length in womenA multi-targeted approach to suppress tumor-promoting inflammationInflammation, telomere length, and grip strength: a 10-year longitudinal study.NF-κB regulates DNA double-strand break repair in conjunction with BRCA1-CtIP complexesCD4(+) T cells contribute to the remodeling of the microenvironment required for sustained tumor regression upon oncogene inactivationTNFα facilitates clonal expansion of JAK2V617F positive cells in myeloproliferative neoplasms.Paracrine effects of haematopoietic cells on human mesenchymal stem cells.Epstein-Barr Virus BZLF1-Mediated Downregulation of Proinflammatory Factors Is Essential for Optimal Lytic Viral ReplicationIL-32θ gene expression in acute myeloid leukemia suppresses TNF-α production.Premature senescence of endothelial cells upon chronic exposure to TNFα can be prevented by N-acetyl cysteine and plumericin.TNF-α inhibitors: are they carcinogenic?BZLF1 Attenuates Transmission of Inflammatory Paracrine Senescence in Epstein-Barr Virus-Infected Cells by Downregulating Tumor Necrosis Factor Alpha.TNFα enhances cancer stem cell-like phenotype via Notch-Hes1 activation in oral squamous cell carcinoma cellsRepeated lipopolysaccharide stimulation promotes cellular senescence in human dental pulp stem cells (DPSCs).IL-32θ: a recently identified anti-inflammatory variant of IL-32 and its preventive role in various disorders and tumor suppressor activity.Deficient proliferation and expansion in vitro of two bone marrow cell populations from patients with acute myeloid leukemia in response to hematopoietic cytokines.Oncodriver inhibition and CD4+ Th1 cytokines cooperate through Stat1 activation to induce tumor senescence and apoptosis in HER2+ and triple negative breast cancer: implications for combining immune and targeted therapies.The Neuro-Immuno-Senescence Integrative Model (NISIM) on the Negative Association Between Parasympathetic Activity and Cellular Senescence
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Tumor necrosis factor alpha induces senescence and chromosomal instability in human leukemic cells.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh
2004年學術文章
@zh-hant
name
Tumor necrosis factor alpha in ...... ility in human leukemic cells.
@en
type
label
Tumor necrosis factor alpha in ...... ility in human leukemic cells.
@en
prefLabel
Tumor necrosis factor alpha in ...... ility in human leukemic cells.
@en
P2093
P2860
P356
P1433
P1476
Tumor necrosis factor alpha in ...... ility in human leukemic cells.
@en
P2093
Christian Recher
Cécile Demur
Guy Laurent
Géraldine Pottier
Nicole Dastugue
Odile Beyne-Rauzy
Véronique Mansat-De Mas
P2860
P2888
P304
P356
10.1038/SJ.ONC.1208024
P407
P577
2004-09-01T00:00:00Z
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P6179
1000734131