Opposite regulation of myc and p21waf1 transcription by STAT3 proteins.
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Crif1 is a novel transcriptional coactivator of STAT3The ERBB4/HER4 receptor tyrosine kinase regulates gene expression by functioning as a STAT5A nuclear chaperoneErbB2-mediated Src and signal transducer and activator of transcription 3 activation leads to transcriptional up-regulation of p21Cip1 and chemoresistance in breast cancer cellsInterplay Between Metabolism and Oncogenic Process: Role of microRNAsMaltose-binding protein fusion allows for high level bacterial expression and purification of bioactive mammalian cytokine derivativesGene expression and biological processes influenced by deletion of Stat3 in pulmonary type II epithelial cells.Heregulin Co-opts PR Transcriptional Action Via Stat3 Role As a Coregulator to Drive Cancer Growth.Interleukin-6 (IL-6) and IL-17 synergistically promote viral persistence by inhibiting cellular apoptosis and cytotoxic T cell functionExpression of metabolic, tissue remodeling, oxidative stress, and inflammatory pathways in mammary tissue during involution in lactating dairy cowsClinical implications of phosphorylated STAT3 expression in De Novo diffuse large B-cell lymphomaActivation of miR-21 by STAT3 induces proliferation and suppresses apoptosis in nasopharyngeal carcinoma by targeting PTEN geneProgress towards the development of SH2 domain inhibitors.The antihypertension drug doxazosin suppresses JAK/STATs phosphorylation and enhances the effects of IFN-α/γ-induced apoptosis.Regulation of glucose metabolism in hepatocarcinogenesis by microRNAs.Constitutive activation of JAK3/STAT3 in colon carcinoma tumors and cell lines: inhibition of JAK3/STAT3 signaling induces apoptosis and cell cycle arrest of colon carcinoma cells.Signal transducer and activator of transcription-3, inflammation, and cancer: how intimate is the relationship?.STAT3 is required for proliferation and maintenance of multipotency in glioblastoma stem cells.Landscape of EGFR signaling network in human cancers: biology and therapeutic response in relation to receptor subcellular locationsSTAT1 is Constitutively Activated in the T/C28a2 Immortalized Juvenile Human Chondrocyte Line and Stimulated by IL-6 Plus Soluble IL-6RGene Network Analysis of Glucose Linked Signaling Pathways and Their Role in Human Hepatocellular Carcinoma Cell Growth and Survival in HuH7 and HepG2 Cell Lines.Transcriptional upregulation of microtubule-associated protein 2 is involved in the protein kinase A-induced decrease in the invasiveness of glioma cellsImpaired liver regeneration is associated with reduced cyclin B1 in natural killer T cell-deficient miceSTAT3 inhibition in prostate and pancreatic cancer lines by STAT3 binding sequence oligonucleotides: differential activity between 5' and 3' ends.IL-6 upregulation contributes to the reduction of miR-26a expression in hepatocellular carcinoma cells.The two faces of IL-6 in the tumor microenvironment.STAT1 and STAT3 in tumorigenesis: A matter of balance.How should we define STAT3 as an oncogene and as a potential target for therapy?The Role of STAT3 in Non-Small Cell Lung Cancer.Leptin activates STAT3 and ERK1/2 pathways and induces endometrial cancer cell proliferation.Interleukin-6 induces expression of Ifi202, an interferon-inducible candidate gene for lupus susceptibility.Low-Dose DMC Significantly Enhances the Effect of TMZ on Glioma Cells by Targeting Multiple Signaling Pathways Both In Vivo and In Vitro.Histone deacetylase inhibitor valproic acid sensitizes B16F10 melanoma cells to cucurbitacin B treatment.The cdk5 kinase regulates the STAT3 transcription factor to prevent DNA damage upon topoisomerase I inhibition.Diesel particle-induced transcriptional expression of p21 involves activation of EGFR, Src, and Stat3.Constitutively activated STAT3 frequently coexpresses with epidermal growth factor receptor in high-grade gliomas and targeting STAT3 sensitizes them to Iressa and alkylatorsRac1 GTPase and the Rac1 exchange factor Tiam1 associate with Wnt-responsive promoters to enhance beta-catenin/TCF-dependent transcription in colorectal cancer cells.The cell cycle inhibitor p21waf1 binds to the myc and cdc25A promoters upon DNA damage and induces transcriptional repression.Implication of BRG1 and cdk9 in the STAT3-mediated activation of the p21waf1 gene.NCoA-1/SRC-1 is an essential coactivator of STAT5 that binds to the FDL motif in the alpha-helical region of the STAT5 transactivation domain.5-lipoxygenase-activating protein gene expression. Key role of CCAAT/enhancer-binding proteins (C/EBP) in constitutive and tumor necrosis factor (TNF) alpha-induced expression in THP-1 cells.
P2860
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P2860
Opposite regulation of myc and p21waf1 transcription by STAT3 proteins.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh
2002年學術文章
@zh-hant
name
Opposite regulation of myc and p21waf1 transcription by STAT3 proteins.
@en
type
label
Opposite regulation of myc and p21waf1 transcription by STAT3 proteins.
@en
prefLabel
Opposite regulation of myc and p21waf1 transcription by STAT3 proteins.
@en
P2093
P2860
P356
P1476
Opposite regulation of myc and p21waf1 transcription by STAT3 proteins.
@en
P2093
Benjamin Barré
Olivier Coqueret
Sylvie Avril
P2860
P304
P356
10.1074/JBC.M210422200
P407
P577
2002-11-15T00:00:00Z