Effects of 17-AAG on the cell cycle and apoptosis of H446 cells and the associated mechanisms.

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Effects of 17-AAG on the cell cycle and apoptosis of H446 cells and the associated mechanisms.

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2016 nî lūn-bûn

@nan

2016年の論文

@ja

2016年論文

@yue

2016年論文

@zh-hant

2016年論文

@zh-hk

2016年論文

@zh-mo

2016年論文

@zh-tw

2016年论文

@wuu

2016年论文

@zh

2016年论文

@zh-cn

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Effects of 17-AAG on the cell ...... and the associated mechanisms.

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Item

label

Effects of 17-AAG on the cell ...... and the associated mechanisms.

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Effects of 17-AAG on the cell ...... and the associated mechanisms.

@en

P1476

Effects of 17-AAG on the cell ...... and the associated mechanisms.

@en

P2093

Enhong Zhao

http://www.w3.org/2001/XMLSchema#string

Huachuan Zheng

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Jianping Wang

http://www.w3.org/2001/XMLSchema#string

Lijun Xiao

http://www.w3.org/2001/XMLSchema#string

Qian Xu

http://www.w3.org/2001/XMLSchema#string

Shi Ding

http://www.w3.org/2001/XMLSchema#string

Shuang Zhao

http://www.w3.org/2001/XMLSchema#string

Xin Zheng

http://www.w3.org/2001/XMLSchema#string

Xuerong Zhao

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P2860

Survivin - biology and potential as a therapeutic target in oncology

Heat shock protein 90 inhibition abrogates hepatocellular cancer growth through cdc2-mediated G2/M cell cycle arrest and apoptosis.

17-Allylamino-17-demethoxygeldanamycin induces downregulation of critical Hsp90 protein clients and results in cell cycle arrest and apoptosis of human urinary bladder cancer cells

Tanespimycin as antitumor therapy.

Targeting the molecular chaperone heat shock protein 90 (HSP90): lessons learned and future directions.

Upregulation of survivin in G2/M cells and inhibition of caspase 9 activity enhances resistance in staurosporine-induced apoptosis.

STAT3 Decoy Oligodeoxynucleotides-Loaded Solid Lipid Nanoparticles Induce Cell Death and Inhibit Invasion in Ovarian Cancer Cells

SNX-2112, a selective Hsp90 inhibitor, potently inhibits tumor cell growth, angiogenesis, and osteoclastogenesis in multiple myeloma and other hematologic tumors by abrogating signaling via Akt and ERK

Wentilactone A as a novel potential antitumor agent induces apoptosis and G2/M arrest of human lung carcinoma cells, and is mediated by HRas-GTP accumulation to excessively activate the Ras/Raf/ERK/p53-p21 pathway.

Current status of targeted therapy in non-small cell lung cancer.

P304

1067-1074

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P31

scholarly article

P356

10.3892/MMR.2016.5365

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P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2016-06-06T00:00:00Z

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27279418

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P932

4940086

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