Latonduine Analogs Restore F508del-Cystic Fibrosis Transmembrane Conductance Regulator Trafficking through the Modulation of Poly-ADP Ribose Polymerase 3 and Poly-ADP Ribose Polymerase 16 Activity.

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CFTR Modulators: Shedding Light on Precision Medicine for Cystic Fibrosis CFTR pharmacology.

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P2860

Latonduine Analogs Restore F508del-Cystic Fibrosis Transmembrane Conductance Regulator Trafficking through the Modulation of Poly-ADP Ribose Polymerase 3 and Poly-ADP Ribose Polymerase 16 Activity.

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2016 nî lūn-bûn

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2016年の論文

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2016年論文

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Latonduine Analogs Restore F50 ...... Ribose Polymerase 16 Activity.

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Latonduine Analogs Restore F50 ...... Ribose Polymerase 16 Activity.

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Latonduine Analogs Restore F50 ...... Ribose Polymerase 16 Activity

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Colin M Edge

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Elizabeth Matthes

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Graeme W Carlile

http://www.w3.org/2001/XMLSchema#string

John W Hanrahan

http://www.w3.org/2001/XMLSchema#string

Qi Yang

http://www.w3.org/2001/XMLSchema#string

Raymond Andersen

http://www.w3.org/2001/XMLSchema#string

Renaud Robert

http://www.w3.org/2001/XMLSchema#string

Richard Hatley

http://www.w3.org/2001/XMLSchema#string

Roberto Solari

http://www.w3.org/2001/XMLSchema#string

Véronique Birault

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P2860

PARP16 is a tail-anchored endoplasmic reticulum protein required for the PERK- and IRE1α-mediated unfolded protein response

Hsp90 cochaperone Aha1 downregulation rescues misfolding of CFTR in cystic fibrosis

The Protein Data Bank

Structural basis for inhibitor specificity in human poly(ADP-ribose) polymerase-3

Identification of the cystic fibrosis gene: cloning and characterization of complementary DNA

CFTR delivery to 25% of surface epithelial cells restores normal rates of mucus transport to human cystic fibrosis airway epithelium

Correctors of protein trafficking defects identified by a novel high-throughput screening assay.

Using combined computational techniques to predict the glass transition temperatures of aromatic polybenzoxazines.

IRE1 inhibition perturbs the unfolded protein response in a pancreatic β-cell line expressing mutant proinsulin, but does not sensitize the cells to apoptosis.

Correction of the F508del-CFTR protein processing defect in vitro by the investigational drug VX-809.

P304

65-79

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scholarly article

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10.1124/MOL.115.102418

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David Y. Thomas

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2016-05-18T00:00:00Z

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27193581

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cystic fibrosis

transmembrane protein

Latonduine Analogs Restore F508del-Cystic Fibrosis Transmembrane Conductance Regulator Trafficking through the Modulation of Poly-ADP Ribose Polymerase 3 and Poly-ADP Ribose Polymerase 16 Activity.

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P2860

P2860

Latonduine Analogs Restore F508del-Cystic Fibrosis Transmembrane Conductance Regulator Trafficking through the Modulation of Poly-ADP Ribose Polymerase 3 and Poly-ADP Ribose Polymerase 16 Activity.

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