Hepatic Ketogenesis Induced by Middle Cerebral Artery Occlusion in Mice.

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Hepatic Ketogenesis Induced by Middle Cerebral Artery Occlusion in Mice.

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2017 nî lūn-bûn

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2017年の論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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Hepatic Ketogenesis Induced by Middle Cerebral Artery Occlusion in Mice.

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Hepatic Ketogenesis Induced by Middle Cerebral Artery Occlusion in Mice.

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Hepatic Ketogenesis Induced by Middle Cerebral Artery Occlusion in Mice.

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P1476

Hepatic Ketogenesis Induced by Middle Cerebral Artery Occlusion in Mice.

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P2093

Anna Thinnes

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Dirk Berressem

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Gunter P Eckert

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Jan Konietzka

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Jochen Klein

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Konrad Koch

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P2860

Global and regional mortality from 235 causes of death for 20 age groups in 1990 and 2010: a systematic analysis for the Global Burden of Disease Study 2010

The neuroprotective properties of calorie restriction, the ketogenic diet, and ketone bodies

A simple method for the isolation and purification of total lipides from animal tissues

Stroke outcome in the ketogenic state--a systematic review of the animal data

Short-chain fatty acids and ketones directly regulate sympathetic nervous system via G protein-coupled receptor 41 (GPR41)

The contribution of ketone bodies to basal and activity-dependent neuronal oxidation in vivo

Metabolic and transmitter changes in core and penumbra after middle cerebral artery occlusion in mice

The succinate receptor as a novel therapeutic target for oxidative and metabolic stress-related conditions

[2,4-13 C2 ]-beta-Hydroxybutyrate metabolism in human brain.

Ischaemic accumulation of succinate controls reperfusion injury through mitochondrial ROS.

P31

scholarly article

P356

10.1161/JAHA.117.005556

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P433

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P478

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P577

2017-04-05T00:00:00Z

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P698

28381467

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P932

5533036

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