Cutting Edge: Mycobacterium tuberculosis Induces Aerobic Glycolysis in Human Alveolar Macrophages That Is Required for Control of Intracellular Bacillary Replication.
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Does Elevated Glucose Promote Atherosclerosis? Pros and Cons.Rewiring cellular metabolism via the AKT/mTOR pathway contributes to host defence against Mycobacterium tuberculosis in human and murine cells.Immunometabolic Pathways in BCG-Induced Trained Immunity.The bacillary and macrophage response to hypoxia in tuberculosis and the consequences for T cell antigen recognition.Metabolic Remodeling, Inflammasome Activation, and Pyroptosis in Macrophages Stimulated by Porphyromonas gingivalis and Its Outer Membrane Vesicles.Innate Immune Function of Mitochondrial MetabolismIntegrating Lung Physiology, Immunology, and Tuberculosis.Energy metabolic pathways control the fate and function of myeloid immune cells.Nitric Oxide Modulates Macrophage Responses to Mycobacterium tuberculosis Infection through Activation of HIF-1α and Repression of NF-κB.Imaging the NADH:NAD(+) Homeostasis for Understanding the Metabolic Response of Mycobacterium to Physiologically Relevant Stresses.Phenolic Glycolipid Facilitates Mycobacterial Escape from Microbicidal Tissue-Resident Macrophages.Interleukin-17 limits hypoxia-inducible factor 1α and development of hypoxic granulomas during tuberculosis.Protective immunity against tuberculosis: what does it look like and how do we find it?Mycobacterium tuberculosis: Rewiring host cell signaling to promote infection.De Novo Fatty Acid Synthesis During Mycobacterial Infection Is a Prerequisite for the Function of Highly Proliferative T Cells, But Not for Dendritic Cells or Macrophages.Tissue metabolic changes drive cytokine responses to Mycobacterium tuberculosis.Growth of Mycobacterium tuberculosis in vivo segregates with host macrophage metabolism and ontogeny.Macrophage Heterogeneity in the Immunopathogenesis of Tuberculosis.Long-term reduction in hyperglycemia in advanced type 1 diabetes: the value of induced aerobic glycolysis with BCG vaccinations.Modulating Iron for Metabolic Support of TB Host Defense
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P2860
Cutting Edge: Mycobacterium tuberculosis Induces Aerobic Glycolysis in Human Alveolar Macrophages That Is Required for Control of Intracellular Bacillary Replication.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年学术文章
@wuu
2016年学术文章
@zh-cn
2016年学术文章
@zh-hans
2016年学术文章
@zh-my
2016年学术文章
@zh-sg
2016年學術文章
@yue
2016年學術文章
@zh
2016年學術文章
@zh-hant
name
Cutting Edge: Mycobacterium tu ...... ellular Bacillary Replication.
@en
type
label
Cutting Edge: Mycobacterium tu ...... ellular Bacillary Replication.
@en
prefLabel
Cutting Edge: Mycobacterium tu ...... ellular Bacillary Replication.
@en
P2860
P50
P356
P1476
Cutting Edge: Mycobacterium tu ...... ellular Bacillary Replication.
@en
P2093
Eva M Palsson-McDermott
Laura E Gleeson
P2860
P304
P356
10.4049/JIMMUNOL.1501612
P407
P577
2016-02-12T00:00:00Z