MiR-135a promotes renal fibrosis in diabetic nephropathy by regulating TRPC1. - Wikidata - awgldk - TriplyDB

MiR-135a promotes renal fibrosis in diabetic nephropathy by regulating TRPC1.

MiR-135a promotes renal fibrosis in diabetic nephropathy by regulating TRPC1.

http://www.wikidata.org/entity/Q38987204

about

Mini-review: emerging roles of microRNAs in the pathophysiology of renal diseases The role of microRNAs in diabetic nephropathy.BAFF promotes proliferation of human mesangial cells through interaction with BAFF-R Epigenetic mechanisms of endothelial dysfunction in type 2 diabetes.Quantitative Genetics of Food Intake in Drosophila melanogaster.miR-135 family members mediate podocyte injury through the activation of Wnt/β-catenin signaling.Circulating TGF-β1-Regulated miRNAs and the Risk of Rapid Progression to ESRD in Type 1 Diabetes.MicroRNAs in diabetic nephropathy: functions, biomarkers, and therapeutic targets.Podocyte injury in diabetic nephropathy: implications of angiotensin II-dependent activation of TRPC channels Up-regulation of microRNA-135a protects against myocardial ischemia/reperfusion injury by decreasing TXNIP expression in diabetic mice.Calcium-permeable ion channels in the kidney.MicroRNAs as novel therapeutic targets to treat kidney injury and fibrosis.MicroRNA-27a promotes renal tubulointerstitial fibrosis via suppressing PPARγ pathway in diabetic nephropathy.Gankyrin sustains PI3K/GSK-3β/β-catenin signal activation and promotes colorectal cancer aggressiveness and progression.RNA-binding protein HuR enhances mineralocorticoid signaling in renal KC3AC1 cells under hypotonicity.Canonical Transient Receptor Potential 6 Channel: A New Target of Reactive Oxygen Species in Renal Physiology and Pathology.Epigenetics of kidney disease.Emerging Roles for MicroRNAs in Diabetic Microvascular Disease: Novel Targets for Therapy.An endoplasmic reticulum stress-regulated lncRNA hosting a microRNA megacluster induces early features of diabetic nephropathy.Non-genetic mechanisms of diabetic nephropathy.Reciprocal regulation of miR-214 and PTEN by high glucose regulates renal glomerular mesangial and proximal tubular epithelial cell hypertrophy and matrix expansion.Increased Abundance of Plasmacytoid Dendritic Cells and Interferon-Alpha Induces Plasma Cell Differentiation in Patients of IgA Nephropathy.Triptolide prevents extracellular matrix accumulation in experimental diabetic kidney disease by targeting microRNA-137/Notch1 pathway.Schisandrin B inhibits the proliferation of airway smooth muscle cells via microRNA-135a suppressing the expression of transient receptor potential channel 1.miR302a-3p May Modulate Renal Epithelial-Mesenchymal Transition in Diabetic Kidney Disease by Targeting ZEB1.MicroRNA, Proteins, and Metabolites as Novel Biomarkers for Prediabetes, Diabetes, and Related Complications.Renal inhibition of miR-181a ameliorates 5-fluorouracil-induced mesangial cell apoptosis and nephrotoxicity.MicroRNA and Microvascular Complications of Diabetes.Validation of differentially methylated microRNAs identified from an epigenome-wide association study; Sanger and next generation sequencing approaches Noncoding RNAs as therapeutic targets in early stage diabetic kidney disease

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P2860

MiR-135a promotes renal fibrosis in diabetic nephropathy by regulating TRPC1.

http://www.wikidata.org/entity/Q38987204

description

2014 nî lūn-bûn

@nan

2014年の論文

@ja

2014年学术文章

@wuu

2014年学术文章

@zh-cn

2014年学术文章

@zh-hans

2014年学术文章

@zh-my

2014年学术文章

@zh-sg

2014年學術文章

@yue

2014年學術文章

@zh

2014年學術文章

@zh-hant

name

MiR-135a promotes renal fibrosis in diabetic nephropathy by regulating TRPC1.

@en

type

label

MiR-135a promotes renal fibrosis in diabetic nephropathy by regulating TRPC1.

@en

prefLabel

MiR-135a promotes renal fibrosis in diabetic nephropathy by regulating TRPC1.

@en

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P1476

MiR-135a promotes renal fibrosis in diabetic nephropathy by regulating TRPC1.

@en

P2093

Chen Zhao

http://www.w3.org/2001/XMLSchema#string

Fenfen Peng

http://www.w3.org/2001/XMLSchema#string

Feng He

http://www.w3.org/2001/XMLSchema#string

Fengxian Huang

http://www.w3.org/2001/XMLSchema#string

Qimei Luo

http://www.w3.org/2001/XMLSchema#string

Weiming Guan

http://www.w3.org/2001/XMLSchema#string

Xi Xia

http://www.w3.org/2001/XMLSchema#string

Xueqing Yu

http://www.w3.org/2001/XMLSchema#string

Zhijian Li

http://www.w3.org/2001/XMLSchema#string

P2860

Dynamic assembly of TRPC1-STIM1-Orai1 ternary complex is involved in store-operated calcium influx. Evidence for similarities in store-operated and calcium release-activated calcium channel components

Antagonism of microRNA-122 in mice by systemically administered LNA-antimiR leads to up-regulation of a large set of predicted target mRNAs in the liver

Circulating microRNAs as stable blood-based markers for cancer detection.

Non-stimulated, agonist-stimulated and store-operated Ca2+ influx in MDA-MB-468 breast cancer cells and the effect of EGF-induced EMT on calcium entry

MicroRNAs: small RNAs with a big role in gene regulation

RhoA interaction with inositol 1,4,5-trisphosphate receptor and transient receptor potential channel-1 regulates Ca2+ entry. Role in signaling increased endothelial permeability

STIM1 carboxyl-terminus activates native SOC, I(crac) and TRPC1 channels

TRPC6 is a glomerular slit diaphragm-associated channel required for normal renal function

3-Hydroxy-3-methylglutaryl CoA reductase inhibitors prevent high glucose-induced proliferation of mesangial cells via modulation of Rho GTPase/ p21 signaling pathway: Implications for diabetic nephropathy.

miRNA-135a promotes breast cancer cell migration and invasion by targeting HOXA10.

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8

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57

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