MiR-135a promotes renal fibrosis in diabetic nephropathy by regulating TRPC1.
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Mini-review: emerging roles of microRNAs in the pathophysiology of renal diseasesThe role of microRNAs in diabetic nephropathy.BAFF promotes proliferation of human mesangial cells through interaction with BAFF-REpigenetic mechanisms of endothelial dysfunction in type 2 diabetes.Quantitative Genetics of Food Intake in Drosophila melanogaster.miR-135 family members mediate podocyte injury through the activation of Wnt/β-catenin signaling.Circulating TGF-β1-Regulated miRNAs and the Risk of Rapid Progression to ESRD in Type 1 Diabetes.MicroRNAs in diabetic nephropathy: functions, biomarkers, and therapeutic targets.Podocyte injury in diabetic nephropathy: implications of angiotensin II-dependent activation of TRPC channelsUp-regulation of microRNA-135a protects against myocardial ischemia/reperfusion injury by decreasing TXNIP expression in diabetic mice.Calcium-permeable ion channels in the kidney.MicroRNAs as novel therapeutic targets to treat kidney injury and fibrosis.MicroRNA-27a promotes renal tubulointerstitial fibrosis via suppressing PPARγ pathway in diabetic nephropathy.Gankyrin sustains PI3K/GSK-3β/β-catenin signal activation and promotes colorectal cancer aggressiveness and progression.RNA-binding protein HuR enhances mineralocorticoid signaling in renal KC3AC1 cells under hypotonicity.Canonical Transient Receptor Potential 6 Channel: A New Target of Reactive Oxygen Species in Renal Physiology and Pathology.Epigenetics of kidney disease.Emerging Roles for MicroRNAs in Diabetic Microvascular Disease: Novel Targets for Therapy.An endoplasmic reticulum stress-regulated lncRNA hosting a microRNA megacluster induces early features of diabetic nephropathy.Non-genetic mechanisms of diabetic nephropathy.Reciprocal regulation of miR-214 and PTEN by high glucose regulates renal glomerular mesangial and proximal tubular epithelial cell hypertrophy and matrix expansion.Increased Abundance of Plasmacytoid Dendritic Cells and Interferon-Alpha Induces Plasma Cell Differentiation in Patients of IgA Nephropathy.Triptolide prevents extracellular matrix accumulation in experimental diabetic kidney disease by targeting microRNA-137/Notch1 pathway.Schisandrin B inhibits the proliferation of airway smooth muscle cells via microRNA-135a suppressing the expression of transient receptor potential channel 1.miR302a-3p May Modulate Renal Epithelial-Mesenchymal Transition in Diabetic Kidney Disease by Targeting ZEB1.MicroRNA, Proteins, and Metabolites as Novel Biomarkers for Prediabetes, Diabetes, and Related Complications.Renal inhibition of miR-181a ameliorates 5-fluorouracil-induced mesangial cell apoptosis and nephrotoxicity.MicroRNA and Microvascular Complications of Diabetes.Validation of differentially methylated microRNAs identified from an epigenome-wide association study; Sanger and next generation sequencing approachesNoncoding RNAs as therapeutic targets in early stage diabetic kidney disease
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P2860
MiR-135a promotes renal fibrosis in diabetic nephropathy by regulating TRPC1.
description
2014 nî lūn-bûn
@nan
2014年の論文
@ja
2014年学术文章
@wuu
2014年学术文章
@zh-cn
2014年学术文章
@zh-hans
2014年学术文章
@zh-my
2014年学术文章
@zh-sg
2014年學術文章
@yue
2014年學術文章
@zh
2014年學術文章
@zh-hant
name
MiR-135a promotes renal fibrosis in diabetic nephropathy by regulating TRPC1.
@en
type
label
MiR-135a promotes renal fibrosis in diabetic nephropathy by regulating TRPC1.
@en
prefLabel
MiR-135a promotes renal fibrosis in diabetic nephropathy by regulating TRPC1.
@en
P2093
P2860
P1433
P1476
MiR-135a promotes renal fibrosis in diabetic nephropathy by regulating TRPC1.
@en
P2093
Fenfen Peng
Fengxian Huang
Weiming Guan
Xueqing Yu
Zhijian Li
P2860
P2888
P304
P356
10.1007/S00125-014-3282-0
P577
2014-06-09T00:00:00Z
P6179
1034108908