ROS-generating oxidases Nox1 and Nox4 contribute to oncogenic Ras-induced premature senescence. - Wikidata - awgldk - TriplyDB

ROS-generating oxidases Nox1 and Nox4 contribute to oncogenic Ras-induced premature senescence.

ROS-generating oxidases Nox1 and Nox4 contribute to oncogenic Ras-induced premature senescence.

http://www.wikidata.org/entity/Q39231615

about

"Inflammaging" as a Druggable Target: A Senescence-Associated Secretory Phenotype-Centered View of Type 2 Diabetes Inflammation in gastric cancer: Interplay of the COX-2/prostaglandin E2 and Toll-like receptor/MyD88 pathways The ins and outs of muscle stem cell aging NADPH oxidases: key modulators in aging and age-related cardiovascular diseases?Interplay between oxidant species and energy metabolism Oncogene-induced reactive oxygen species fuel hyperproliferation and DNA damage response activation Cardiac Cell Senescence and Redox Signaling.European contribution to the study of ROS: A summary of the findings and prospects for the future from the COST action BM1203 (EU-ROS).Negative regulation of NADPH oxidase 4 by hydrogen peroxide-inducible clone 5 (Hic-5) protein E2F transcription factor 1 regulates cellular and organismal senescence by inhibiting Forkhead box O transcription factors NADPH oxidase 4 is a critical mediator in Ataxia telangiectasia disease.Emerging roles of the p38 MAPK and PI3K/AKT/mTOR pathways in oncogene-induced senescence.MacroH2A1 and ATM Play Opposing Roles in Paracrine Senescence and the Senescence-Associated Secretory Phenotype NOX1 and NOX4 are required for the differentiation of mouse F9 cells into extraembryonic endoderm EPA or DHA enhanced oxidative stress and aging protein expression in brain of d-galactose treated mice.Reactive oxygen species, vascular Noxs, and hypertension: focus on translational and clinical research Molecular mechanisms underlying chronic inflammation-associated cancers.NOX4 downregulation leads to senescence of human vascular smooth muscle cells.Areca nut exposure increases secretion of tumor-promoting cytokines in gingival fibroblasts that trigger DNA damage in oral keratinocytes.ROS in gastrointestinal inflammation: Rescue Or Sabotage?Therapeutic potential of NADPH oxidase 1/4 inhibitors.Molecular mechanisms of mTOR regulation by stress IFNγ induces oxidative stress, DNA damage and tumor cell senescence via TGFβ/SMAD signaling-dependent induction of Nox4 and suppression of ANT2.Increased susceptibility of H-Ras(G12V)-transformed human urothelial cells to the genotoxic effects of sodium arsenite.Changes in the oxidative stress/anti-oxidant system after exposure to sulfur mustard and antioxidant strategies in the therapy, a review.Interferon gamma/NADPH oxidase defense system in immunity and cancer.Reactive oxygen species: The good, the bad, and the enigma Dopamine induces the accumulation of insoluble prion protein and affects autophagic flux.Changing T-cell enigma: cancer killing or cancer control?Autophagy maintains stemness by preventing senescence.Temporal changes in K-ras mutant fraction in lung tissue of big blue B6C3F₁ mice exposed to ethylene oxide.Cytokine-induced 'bystander' senescence in DDR and immuno-surveillance.NADPH oxidase 4 mediates ROS production in radiation-induced senescent cells and promotes migration of inflammatory cells.Anti-neoplastic drugs increase caveolin-1-dependent migration, invasion and metastasis of cancer cells.NADPH Oxidases and Mitochondria in Vascular Senescence.The defense and signaling role of NADPH oxidases in eukaryotic cells : Review

P2860

Q26747251-895A9789-9E53-4C32-A6D6-CBEDBD02227F Q26748123-69F31A9D-21D9-4C30-967A-B85D062490FF Q26771242-B0261219-3F43-40AE-B28B-A128E31F23B8 Q28072348-77C913EA-99B0-4845-B121-F8E27C71DBF4 Q28383905-EE2EEE9C-400B-4DA3-B0E0-DC8785200D14 Q33580123-A1830611-2765-415E-85A2-B97648260B0E Q33734782-E3458F5E-F86F-4DED-99B2-9B60031CF9BD Q33761211-D203DD3E-58CA-48E0-9684-69BE16A02F40 Q34073807-6C73C7DC-A6BD-425F-8389-9E81DC5F3D13 Q34634278-5950074D-B033-46E7-A006-FF3DACFAEEDA Q35128895-71024A76-CF1B-45B4-B317-9A728CE74014 Q35174677-B75DC314-68EE-4AB2-8D63-8B103A3EB114 Q35994479-4598E408-D8D9-403C-83EF-39C1146B4F5B Q36267903-42873546-4B23-4C2F-988C-ACDB137AE156 Q37167078-9EAADC66-B63A-4B89-89AD-688658CC08CB Q37433974-A16A723F-5160-45D0-9CCD-4FBD91EE9F0E Q37605343-7828F80B-5EC5-43A6-AA7C-B2A332B9CB72 Q37687002-041345D6-DC8A-4A0E-BB6E-7DE3EB90095C Q38276312-85CC8DBA-BAD5-4F52-997D-E797D47C7E2F Q38693962-6B532339-7BFC-47BD-A899-01F76F9C4DE7 Q38858553-7043D42E-4733-48AB-A392-254F24A53AE3 Q38866763-93891423-5E87-49E5-BE3C-C984C0BE02CE Q38873861-D3C98462-42BD-47D2-AF53-CBF05B9041F5 Q38958682-63F888D9-668B-42BF-9684-E191FCC0C423 Q39245631-FDAE018C-4258-457F-B5D0-D0791B6763FB Q39280748-3BE8705C-ED78-4D3C-9E2A-4EC535BE36C8 Q39679561-2F50959E-10C0-4F45-B616-6A55A1EF8EF7 Q41982345-675A51DD-BA5F-457E-A282-B56263DFA8E4 Q42287317-C78BDA69-8E36-4278-86FA-E6EE18293AB4 Q42487109-981866E3-6D15-4AF2-86B1-15154DA14E9B Q42709461-3D4A8DEC-357F-48C7-8E6D-01E75F4B1A68 Q42822391-65C1A178-9B2F-4036-92AD-8A4A33C63727 Q46243640-2C5476D0-1B17-4A7A-9132-4B4E617E1B1D Q49230399-0CE4E2A0-322C-4F3E-86EE-86A6237E504C Q55334122-A97914F1-23EF-4E14-AB67-EFA1C6C59CAD Q58798388-0764620E-AACF-4A04-884E-93B6D16CBE47

P2860

ROS-generating oxidases Nox1 and Nox4 contribute to oncogenic Ras-induced premature senescence.

http://www.wikidata.org/entity/Q39231615

description

2012 nî lūn-bûn

@nan

2012年の論文

@ja

2012年論文

@yue

2012年論文

@zh-hant

2012年論文

@zh-hk

2012年論文

@zh-mo

2012年論文

@zh-tw

2012年论文

@wuu

2012年论文

@zh

2012年论文

@zh-cn

name

ROS-generating oxidases Nox1 a ...... -induced premature senescence.

@en

ROS-generating oxidases Nox1 a ...... -induced premature senescence.

@nl

type

label

ROS-generating oxidases Nox1 a ...... -induced premature senescence.

@en

ROS-generating oxidases Nox1 a ...... -induced premature senescence.

@nl

prefLabel

ROS-generating oxidases Nox1 a ...... -induced premature senescence.

@en

ROS-generating oxidases Nox1 a ...... -induced premature senescence.

@nl

P1433

Q39231615-8B5C624F-034C-4D9D-BF15-7E487676B8CF

P1476

Q39231615-1B01938C-73CE-4BCA-BEF3-5B32045C16E7

P2093

Q39231615-31DCFCC6-1892-4CE1-8BCF-7D8C0DEA32B1

Q39231615-4F6EFB3C-4531-4748-BFEC-7ACB3C69B4FF

Q39231615-78CCD612-A173-4247-ADCD-F8A5203C111E

Q39231615-7DCF2833-D4D3-4657-9E7A-250F3870C819

Q39231615-929CB7CC-C305-4E3E-9638-7A0102D2C1DA

Q39231615-9AC7418B-484B-474B-8DCF-C5D22546EFE4

Q39231615-B64E99B0-1385-4FB1-AEFB-6018461CD94B

Q39231615-B98948DC-F07D-4448-B631-D6F2647BFCF4

Q39231615-E21589C3-B57D-474D-A05E-3C9B2A09A783

P2860

Q39231615-06DC119F-2F72-48AE-88C2-EB3FCABCE470

Q39231615-1B7F9903-40FE-48C1-86D8-B9B34FEC5768

Q39231615-1F872271-AC29-4B07-99CA-E031DD66FEB0

Q39231615-30899A2D-83B8-4F3E-9744-E7DBA206008F

Q39231615-3D834B44-EBD4-4618-A210-6E8CE672968A

Q39231615-3E821ADD-6D8A-4028-9C59-65A8D3032C8D

Q39231615-448E0B32-E720-4726-9785-D427311FB386

Q39231615-4CEC47C2-18DD-4F4B-8809-D8C50C35A0E4

Q39231615-5B767D16-2D19-4D3E-B1A6-8F41A655013D

Q39231615-5E12F2F0-84F7-4A5C-B7A2-1F97E1585737

P304

Q39231615-24718CC6-2DEF-44E6-B468-0E42DFB4F36E

P31

Q39231615-991C0FC9-A994-4D16-B956-E365CEDD7878

P356

Q39231615-C13F5BAA-B72E-46B5-9940-A37A84B3666B

P433

Q39231615-90AE13BF-2702-4480-B3A1-1455EC0A4DDB

P478

Q39231615-6CBB397A-2DB7-44EF-A28F-3122B36E64A8

P577

Q39231615-03158337-F785-437C-83BE-50468CF242D4

P5875

Q39231615-A2CAC4AB-FA95-47B1-9D27-BFF6A84070D6

P698

Q39231615-A08ACB4B-64DF-49AF-A619-6257EAF31DCA

P356

P1433

P1476

ROS-generating oxidases Nox1 a ...... -induced premature senescence.

@en

P2093

Chihiro Yabe-Nishimura

http://www.w3.org/2001/XMLSchema#string

Eiji Tanaka

http://www.w3.org/2001/XMLSchema#string

Kuniharu Matsuno

http://www.w3.org/2001/XMLSchema#string

Masayoshi Kato

http://www.w3.org/2001/XMLSchema#string

Ryo Kodama

http://www.w3.org/2001/XMLSchema#string

Shouta Ueno

http://www.w3.org/2001/XMLSchema#string

Shuichi Furuta

http://www.w3.org/2001/XMLSchema#string

Tohru Kamata

http://www.w3.org/2001/XMLSchema#string

Yugen Zhang

http://www.w3.org/2001/XMLSchema#string

P2860

Premature senescence involving p53 and p16 is activated in response to constitutive MEK/MAPK mitogenic signaling

Identification of renox, an NAD(P)H oxidase in kidney

Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a

The matricellular protein CCN1 induces fibroblast senescence and restricts fibrosis in cutaneous wound healing

Nox1 is involved in angiotensin II-mediated hypertension: a study in Nox1-deficient mice

Activation of the DNA damage checkpoint and genomic instability in human precancerous lesions

BRAFE600-associated senescence-like cell cycle arrest of human naevi

Mitogenic signalling and the p16INK4a-Rb pathway cooperate to enforce irreversible cellular senescence.

NADPH oxidase 4 is an oncoprotein localized to mitochondria.

Maximizing mouse cancer models.

P304

32-41

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1111/GTC.12015

http://www.w3.org/2001/XMLSchema#string

P433

1

http://www.w3.org/2001/XMLSchema#string

P478

18

http://www.w3.org/2001/XMLSchema#string

P577

2012-12-06T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

233875036

http://www.w3.org/2001/XMLSchema#string

P698

23216904

http://www.w3.org/2001/XMLSchema#string