Production of resurgent current in NaV1.6-null Purkinje neurons by slowing sodium channel inactivation with beta-pompilidotoxin.
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Peptide Toxins in Solitary Wasp VenomsThe Hodgkin-Huxley heritage: from channels to circuitsVoltage-gated sodium channel-associated proteins and alternative mechanisms of inactivation and blockResurgent current of voltage-gated Na(+) channelsControl of transient, resurgent, and persistent current by open-channel block by Na channel beta4 in cultured cerebellar granule neuronsThe Snake with the Scorpion's Sting: Novel Three-Finger Toxin Sodium Channel Activators from the Venom of the Long-Glanded Blue Coral Snake (Calliophis bivirgatus)Tetrodotoxin-resistant sodium channels in sensory neurons generate slow resurgent currents that are enhanced by inflammatory mediators.Multisite phosphorylation of voltage-gated sodium channel alpha subunits from rat brainLoss of Navβ4-Mediated Regulation of Sodium Currents in Adult Purkinje Neurons Disrupts Firing and Impairs Motor Coordination and Balance.Spontaneous Network Activity and Synaptic Development.Modulation of neuronal sodium channels by the sea anemone peptide BDS-I.Sea-anemone toxin ATX-II elicits A-fiber-dependent pain and enhances resurgent and persistent sodium currents in large sensory neurons.Regulatory Role of Voltage-Gated Na Channel β Subunits in Sensory NeuronsMolecular and functional differences in voltage-activated sodium currents between GABA projection neurons and dopamine neurons in the substantia nigraEarly painful diabetic neuropathy is associated with differential changes in tetrodotoxin-sensitive and -resistant sodium channels in dorsal root ganglion neurons in the rat.Human Nav1.6 Channels Generate Larger Resurgent Currents than Human Nav1.1 Channels, but the Navβ4 Peptide Does Not Protect Either Isoform from Use-Dependent Reduction.Subunit dependence of Na channel slow inactivation and open channel block in cerebellar neuronsAnticancer drug oxaliplatin induces acute cooling-aggravated neuropathy via sodium channel subtype Na(V)1.6-resurgent and persistent current.Navβ4 regulates fast resurgent sodium currents and excitability in sensory neuronsTime-dependent block and resurgent tail currents induced by mouse beta4(154-167) peptide in cardiac Na+ channels.Na+ channel-dependent recruitment of Navβ4 to axon initial segments and nodes of RanvierAntagonism of lidocaine inhibition by open-channel blockers that generate resurgent Na current.FGF14 regulates the intrinsic excitability of cerebellar Purkinje neurons.Interactions among DIV voltage-sensor movement, fast inactivation, and resurgent Na current induced by the NaVβ4 open-channel blocking peptideConvergence of genetic and environmental factors on parvalbumin-positive interneurons in schizophrenia.Recent developments regarding voltage-gated sodium channel blockers for the treatment of inherited and acquired neuropathic pain syndromes.Neurological perspectives on voltage-gated sodium channels.FHF2 isoforms differentially regulate Nav1.6-mediated resurgent sodium currents in dorsal root ganglion neurons.Resurgent Na currents in four classes of neurons of the cerebellum.Voltage-gated sodium channel isoform-specific effects of pompilidotoxins.Transcriptional and electrophysiological maturation of neocortical fast-spiking GABAergic interneurons.Model of very fast (> 75 Hz) network oscillations generated by electrical coupling between the proximal axons of cerebellar Purkinje cells.Human voltage-gated sodium channel mutations that cause inherited neuronal and muscle channelopathies increase resurgent sodium currents.Sodium currents in mesencephalic trigeminal neurons from Nav1.6 null mice.Identification of Persistent and Resurgent Sodium Currents in Spiral Ganglion Neurons Cultured from the Mouse Cochlea.Persistent Nav1.6 current at axon initial segments tunes spike timing of cerebellar granule cells.Sodium currents in subthalamic nucleus neurons from Nav1.6-null mice.Resurgent-like currents in mouse vas deferens myocytes are mediated by NaV1.6 voltage-gated sodium channels.Development of resurgent and persistent sodium currents in mesencephalic trigeminal neurons.Impaired motor function in mice with cell-specific knockout of sodium channel Scn8a (NaV1.6) in cerebellar purkinje neurons and granule cells.
P2860
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P2860
Production of resurgent current in NaV1.6-null Purkinje neurons by slowing sodium channel inactivation with beta-pompilidotoxin.
description
2004 nî lūn-bûn
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2004年の論文
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2004年学术文章
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2004年学术文章
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2004年学术文章
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name
Production of resurgent curren ...... tion with beta-pompilidotoxin.
@en
Production of resurgent curren ...... tion with beta-pompilidotoxin.
@nl
type
label
Production of resurgent curren ...... tion with beta-pompilidotoxin.
@en
Production of resurgent curren ...... tion with beta-pompilidotoxin.
@nl
prefLabel
Production of resurgent curren ...... tion with beta-pompilidotoxin.
@en
Production of resurgent curren ...... tion with beta-pompilidotoxin.
@nl
P1476
Production of resurgent curren ...... ation with beta-pompilidotoxin
@en
P2093
Indira M Raman
Tina M Grieco
P356
10.1523/JNEUROSCI.3807-03.2004
P407
P577
2004-01-01T00:00:00Z