A class IA PI3K controls inflammatory cytokine production in human neutrophils. - Wikidata - awgldk - TriplyDB

A class IA PI3K controls inflammatory cytokine production in human neutrophils.

A class IA PI3K controls inflammatory cytokine production in human neutrophils.

http://www.wikidata.org/entity/Q39563488

about

The combination effects of licl and the active leflunomide metabolite, A771726, on viral-induced interleukin 6 production and EV-A71 replication Insulin modulates the inflammatory granulocyte response to streptococci via phosphatidylinositol 3-kinase.Disruption of genes encoding eIF4E binding proteins-1 and -2 does not alter basal or sepsis-induced changes in skeletal muscle protein synthesis in male or female mice.TLR4 ligand/H₂O₂ enhances TGF-β1 signaling to induce metastatic potential of non-invasive breast cancer cells by activating non-Smad pathways.Identification of a role for the PI3K/AKT/mTOR signaling pathway in innate immune cells Higher Risk of Infections with PI3K-AKT-mTOR Pathway Inhibitors in Patients with Advanced Solid Tumors on Phase I Clinical Trials.New Insights into the Pro-Inflammatory Activities of Ang1 on Neutrophils: Induction of MIP-1β Synthesis and Release.Arginine Relieves the Inflammatory Response and Enhances the Casein Expression in Bovine Mammary Epithelial Cells Induced by Lipopolysaccharide PI3K functions in cancer progression, anticancer immunity and immune evasion by tumors.Regulation of Discrete Functional Responses by Syk and Src Family Tyrosine Kinases in Human Neutrophils NF-κB feedback control of JNK1 activation modulates TRPV1-induced increases in IL-6 and IL-8 release by human corneal epithelial cells.Early tyrosine phosphorylation events following adenosine A2A receptor in human neutrophils: identification of regulated pathways.Strong inhibition of neutrophil-sperm interaction in cattle by selective phosphatidylinositol 3-kinase inhibitors.PI3K in cancer: its structure, activation modes and role in shaping tumor microenvironment.Molecular Genetic and Epigenetic Basis of Multiple Sclerosis.Probing the response of lung tumor cells to inflammatory microvascular endothelial cells on fluidic microdevice.Effects of Phytochemically Characterized Extracts From Syringa vulgaris and Isolated Secoiridoids on Mediators of Inflammation in a Human Neutrophil Model.The molecular mechanism of polygalasaponin F-mediated decreases in TNFα: emphasizing the role of the TLR4-PI3K/AKT-NF-κB pathway.Deoxynivalenol impairs the immune functions of neutrophils.Epithelial Cells Attenuate Toll-Like Receptor-Mediated Inflammatory Responses in Monocyte-Derived Macrophage-Like Cells to by Modulating the PI3K/Akt/mTOR Signaling Pathway

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P2860

A class IA PI3K controls inflammatory cytokine production in human neutrophils.

http://www.wikidata.org/entity/Q39563488

description

2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

@zh-mo

2011年論文

@zh-tw

2011年论文

@wuu

2011年论文

@zh

2011年论文

@zh-cn

name

A class IA PI3K controls inflammatory cytokine production in human neutrophils.

@en

A class IA PI3K controls inflammatory cytokine production in human neutrophils.

@nl

type

label

A class IA PI3K controls inflammatory cytokine production in human neutrophils.

@en

A class IA PI3K controls inflammatory cytokine production in human neutrophils.

@nl

prefLabel

A class IA PI3K controls inflammatory cytokine production in human neutrophils.

@en

A class IA PI3K controls inflammatory cytokine production in human neutrophils.

@nl

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P1433

European Journal of Immunology

P1476

A class IA PI3K controls inflammatory cytokine production in human neutrophils

@en

P2093

Alexandre Cloutier

http://www.w3.org/2001/XMLSchema#string

Patrick P McDonald

http://www.w3.org/2001/XMLSchema#string

Rim Bouchelaghem

http://www.w3.org/2001/XMLSchema#string

Stéphanie Sylvain-Prévost

http://www.w3.org/2001/XMLSchema#string

Thomas Z Mayer

http://www.w3.org/2001/XMLSchema#string

Thornin Ear

http://www.w3.org/2001/XMLSchema#string

P2860

Cytokine generation, promoter activation, and oxidant-independent NF-kappaB activation in a transfectable human neutrophilic cellular model

1-Phosphatidylinositol 3-kinase activity is required for insulin-stimulated glucose transport but not for RAS activation in CHO cells

Syk-mediated translocation of PI3Kdelta to the leading edge controls lamellipodium formation and migration of leukocytes

Neutrophils produce biologically active macrophage inflammatory protein-3alpha (MIP-3alpha)/CCL20 and MIP-3beta/CCL19

mTOR and S6K1 mediate assembly of the translation preinitiation complex through dynamic protein interchange and ordered phosphorylation events

ERK and the F-box protein betaTRCP target STAT1 for degradation

MAPK-activated protein kinase-2 participates in p38 MAPK-dependent and ERK-dependent functions in human neutrophils

The evolution of phosphatidylinositol 3-kinases as regulators of growth and metabolism

MatInspector and beyond: promoter analysis based on transcription factor binding sites

Neutrophils and immunity: challenges and opportunities

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1709-1719

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scholarly article

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10.1002/EJI.201040945

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6

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41

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51021508

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21469098

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