Complementation of defective colony-stimulating factor 1 receptor signaling and mitogenesis by Raf and v-Src.
about
Loss of miR-200 inhibition of Suz12 leads to polycomb-mediated repression required for the formation and maintenance of cancer stem cellsA conditional form of Bruton's tyrosine kinase is sufficient to activate multiple downstream signaling pathways via PLC Gamma 2 in B cellsThe transcription factor T-box 3 regulates colony-stimulating factor 1-dependent Jun dimerization protein 2 expression and plays an important role in osteoclastogenesisNOTCH1 mediates a switch between two distinct secretomes during senescence.Stat3-mediated Myc expression is required for Src transformation and PDGF-induced mitogenesis.Induction of beta3-integrin gene expression by sustained activation of the Ras-regulated Raf-MEK-extracellular signal-regulated kinase signaling pathway.Inducible formation of breast cancer stem cells and their dynamic equilibrium with non-stem cancer cells via IL6 secretion.Inhibition of miR-193a expression by Max and RXRα activates K-Ras and PLAU to mediate distinct aspects of cellular transformation.STAT3 acts through pre-existing nucleosome-depleted regions bound by FOS during an epigenetic switch linking inflammation to cancer.MiR-27b targets PPARγ to inhibit growth, tumor progression and the inflammatory response in neuroblastoma cellsAn integrated transcriptional regulatory circuit that reinforces the breast cancer stem cell stateB-Raf activation cooperates with PTEN loss to drive c-Myc expression in advanced prostate cancerDiminished WNT -> β-catenin -> c-MYC signaling is a barrier for malignant progression of BRAFV600E-induced lung tumorsCooperative regulation of the cell division cycle by the protein kinases RAF and AKTExpression of human papilloma virus E7 protein causes apoptosis and inhibits DNA synthesis in primary hepatocytes via increased expression of p21(Cip-1/WAF1/MDA6).Regulation of c-myc expression by IFN-gamma through Stat1-dependent and -independent pathways.Helicobacter pylori-induced macrophage apoptosis requires activation of ornithine decarboxylase by c-Myc.Role for cellular Src kinase in myoblast proliferation.Protection against anoikis and down-regulation of cadherin expression by a regulatable beta-catenin protein.The mechanism of cell cycle regulation by v-Src.Regulation of the expression of c-Myc by beta1 integrins in epithelial cells.Regulation of both apoptosis and cell survival by the v-Src oncoprotein.A role for E2F1 in Ras activation of p21(WAF1/CIP1) transcription.A conditionally-active form of MEK1 results in autocrine tranformation of human and mouse hematopoietic cells.EGFR, HER-2/neu, cyclin D1, p21 and p53 in correlation to cell proliferation and steroid hormone receptor status in ductal carcinoma in situ of the breast.Cystathionase mediates senescence evasion in melanocytes and melanoma cells.c-Fms Tyrosine 559 Is a Major Mediator of M-CSF-induced Proliferation of Primary Macrophages
P2860
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P2860
Complementation of defective colony-stimulating factor 1 receptor signaling and mitogenesis by Raf and v-Src.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
1999年论文
@zh
1999年论文
@zh-cn
name
Complementation of defective c ...... mitogenesis by Raf and v-Src.
@en
Complementation of defective c ...... mitogenesis by Raf and v-Src.
@nl
type
label
Complementation of defective c ...... mitogenesis by Raf and v-Src.
@en
Complementation of defective c ...... mitogenesis by Raf and v-Src.
@nl
prefLabel
Complementation of defective c ...... mitogenesis by Raf and v-Src.
@en
Complementation of defective c ...... mitogenesis by Raf and v-Src.
@nl
P2093
P2860
P356
P1476
Complementation of defective c ...... mitogenesis by Raf and v-Src.
@en
P2093
P2860
P304
P356
10.1128/MCB.19.2.1101
P407
P577
1999-02-01T00:00:00Z