Perspectives series: cell adhesion in vascular biology. Smooth muscle migration in atherosclerosis and restenosis.
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Disruption of platelet-derived growth factor-dependent phosphatidylinositol 3-kinase and phospholipase Cγ 1 activity abolishes vascular smooth muscle cell proliferation and migration and attenuates neointima formation in vivoDecreasing mitochondrial fission diminishes vascular smooth muscle cell migration and ameliorates intimal hyperplasia.Early Transcriptomic Response to LDL and oxLDL in Human Vascular Smooth Muscle CellsThe high mobility group (HMG) boxes of the nuclear protein HMG1 induce chemotaxis and cytoskeleton reorganization in rat smooth muscle cellsPeriostin mediates vascular smooth muscle cell migration through the integrins alphavbeta3 and alphavbeta5 and focal adhesion kinase (FAK) pathwayAssociation between VEGF Gene Polymorphisms and In-Stent Restenosis after Coronary Intervention Treated with Bare Metal Stent.Soluble epoxide hydrolase deficiency attenuates neointima formation in the femoral cuff model of hyperlipidemic mice.An introduction to cell migration and invasion.IL-1beta signals through the EGF receptor and activates Egr-1 through MMP-ADAM.Stem cell origins of intimal cells in graft arterial disease.A small molecule PAI-1 functional inhibitor attenuates neointimal hyperplasia and vascular smooth muscle cell survival by promoting PAI-1 cleavageROCK inhibition prevents fetal serum-induced alteration in structure and function of organ-cultured mesenteric artery.Platelet-derived growth factor (PDGF) regulates Slingshot phosphatase activity via Nox1-dependent auto-dephosphorylation of serine 834 in vascular smooth muscle cells.Hic-5 Mediates TGFβ-Induced Adhesion in Vascular Smooth Muscle Cells by a Nox4-Dependent Mechanism.Vascular smooth muscle cell migration: current research and clinical implications.Early growth response factor-1 induction by injury is triggered by release and paracrine activation by fibroblast growth factor-2The Relationships between Polymorphisms in Genes Encoding the Growth Factors TGF-β1, PDGFB, EGF, bFGF and VEGF-A and the Restenosis Process in Patients with Stable Coronary Artery Disease Treated with Bare Metal Stent.Regulation of smooth muscle by inducible nitric oxide synthase and NADPH oxidase in vascular proliferative diseasesADAMTS7 cleavage and vascular smooth muscle cell migration is affected by a coronary-artery-disease-associated variant.Endogenous cannabinoid receptor CB1 activation promotes vascular smooth-muscle cell proliferation and neointima formation.Age-related differences in insulin-like growth factor-1 receptor signaling regulates Akt/FOXO3a and ERK/Fos pathways in vascular smooth muscle cells.Mitogenesis of vascular smooth muscle cell stimulated by platelet-derived growth factor-bb is inhibited by blocking of intracellular signaling by epigallocatechin-3-O-gallate.Endothelial cells and pulmonary arterial hypertension: apoptosis, proliferation, interaction and transdifferentiationImatinib mesylate for the treatment of pulmonary arterial hypertension.High mobility group box-1 induces migration of vascular smooth muscle cells via TLR4-dependent PI3K/Akt pathway activation.Resveratrol inhibits phenotype modulation by platelet derived growth factor-bb in rat aortic smooth muscle cells.Regulator of calcineurin 1 mediates pathological vascular wall remodelingAn in vivo pilot study of a microporous thin film nitinol-covered stent to assess the effect of porosity and pore geometry on device interaction with the vessel wall.An integrated approach for the mechanisms responsible for atherosclerotic plaque regressionUnique disulfide bonds in epidermal growth factor (EGF) domains of β3 affect structure and function of αIIbβ3 and αvβ3 integrins in different manner.Perivascular delivery of resolvin D1 inhibits neointimal hyperplasia in a rat model of arterial injury.Mechanisms and consequences of affinity modulation of integrin alpha(V)beta(3) detected with a novel patch-engineered monovalent ligand.Genetic Ablation of PDGF-Dependent Signaling Pathways Abolishes Vascular Remodeling and Experimental Pulmonary HypertensionSubstrate-induced phenotypic switches of human smooth muscle cells: an in vitro study of in-stent restenosis activation pathways.Protein disulfide isomerase is required for platelet-derived growth factor-induced vascular smooth muscle cell migration, Nox1 NADPH oxidase expression, and RhoGTPase activation.MKP-1 expression and stabilization and cGK Ialpha prevent diabetes- associated abnormalities in VSMC migration.Mechanisms of vascular smooth muscle NADPH oxidase 1 (Nox1) contribution to injury-induced neointimal formation.alpha5beta1 integrin expression and luminal edge fibronectin matrix assembly by smooth muscle cells after arterial injury.Small Molecule PAI-1 Functional Inhibitor Attenuates Vascular Smooth Muscle Cell Migration and Survival: Implications for the Therapy of Vascular Disease.Platelet-derived growth factor inhibits smooth muscle cell adhesion to fibronectin by ERK-dependent and ERK-independent pathways.
P2860
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P2860
Perspectives series: cell adhesion in vascular biology. Smooth muscle migration in atherosclerosis and restenosis.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
1997年论文
@zh
1997年论文
@zh-cn
name
Perspectives series: cell adhe ...... therosclerosis and restenosis.
@en
Perspectives series: cell adhe ...... therosclerosis and restenosis.
@nl
type
label
Perspectives series: cell adhe ...... therosclerosis and restenosis.
@en
Perspectives series: cell adhe ...... therosclerosis and restenosis.
@nl
prefLabel
Perspectives series: cell adhe ...... therosclerosis and restenosis.
@en
Perspectives series: cell adhe ...... therosclerosis and restenosis.
@nl
P2860
P356
P1476
Perspectives series: cell adhe ...... therosclerosis and restenosis.
@en
P2093
Schwartz SM
P2860
P304
P356
10.1172/JCI119472
P407
P577
1997-06-01T00:00:00Z