Dissection of the voltage-activated potassium outward currents in adult mouse ventricular myocytes: I(to,f), I(to,s), I(K,slow1), I(K,slow2), and I(ss).

Dissection of the voltage-activated potassium outward currents in adult mouse ventricular myocytes: I(to,f), I(to,s), I(K,slow1), I(K,slow2), and I(ss).

Item
http://www.wikidata.org/entity/Q39791465
Late Na(+) current and protracted electrical recovery are critical determinants of the aging myopathyKv4.3-Encoded Fast Transient Outward Current Is Presented in Kv4.2 Knockout Mouse Cardiomyocytes.Impact of KChIP2 on Cardiac Electrophysiology and the Progression of Heart Failure.KChIP2 genotype dependence of transient outward current (Ito) properties in cardiomyocytes isolated from male and female mice.Bisoprolol reverses down-regulation of potassium channel proteins in ventricular tissues of rabbits with heart failure.Reduction in Kv Current Enhances the Temporal Dispersion of the Action Potential in Diabetic Myocytes: Insights From a Novel Repolarization Algorithm.Molecular Basis of Functional Myocardial Potassium Channel Diversity.Modulation of voltage-gated ion channels by sialylation.Sinoatrial node cardiomyocytes derived from human pluripotent cells function as a biological pacemaker.Murine Electrophysiological Models of Cardiac Arrhythmogenesis.Loss of K+ currents in heart failure is accentuated in KChIP2 deficient mice.Stabilization of Kv4 protein by the accessory K(+) channel interacting protein 2 (KChIP2) subunit is required for the generation of native myocardial fast transient outward K(+) currents.Notch signaling modulates the electrical behavior of cardiomyocytes.Interaction of DPP10a with Kv4.3 channel complex results in a sustained current component of human transient outward current Ito.Postnatal developmental decline in IK1 in mouse ventricular myocytes isolated by the Langendorff perfusion method: comparison with the chunk method.Complex interactions in a novel SCN5A compound mutation associated with long QT and Brugada syndrome: Implications for Na+ channel blocking pharmacotherapy for de novo conduction disease.
P2860

Dissection of the voltage-activated potassium outward currents in adult mouse ventricular myocytes: I(to,f), I(to,s), I(K,slow1), I(K,slow2), and I(ss).

Item
http://www.wikidata.org/entity/Q39791465
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Dissection of the voltage-acti ...... slow1), I(K,slow2), and I(ss).
@en
Dissection of the voltage-acti ...... slow1), I(K,slow2), and I(ss).
@nl
type
label
Dissection of the voltage-acti ...... slow1), I(K,slow2), and I(ss).
@en
Dissection of the voltage-acti ...... slow1), I(K,slow2), and I(ss).
@nl
prefLabel
Dissection of the voltage-acti ...... slow1), I(K,slow2), and I(ss).
@en
Dissection of the voltage-acti ...... slow1), I(K,slow2), and I(ss).
@nl
P1433
P1476
P2093
P2860
P2888
P304
P31
P356
P433
P478
P50
P577
P698
P356
P1433
P1476
Dissection of the voltage-acti ...... ,slow1), I(K,slow2), and I(ss)
@en
P2093
P2860
P2888
P304
P31
P356
P433
P478
P50
P577
P698