Stabilization of Kv4 protein by the accessory K(+) channel interacting protein 2 (KChIP2) subunit is required for the generation of native myocardial fast transient outward K(+) currents. - Wikidata - awgldk - TriplyDB

Stabilization of Kv4 protein by the accessory K(+) channel interacting protein 2 (KChIP2) subunit is required for the generation of native myocardial fast transient outward K(+) currents.

Stabilization of Kv4 protein by the accessory K(+) channel interacting protein 2 (KChIP2) subunit is required for the generation of native myocardial fast transient outward K(+) currents.

http://www.wikidata.org/entity/Q45146704

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Congestive Heart Failure Leads to Prolongation of the PR Interval and Atrioventricular Junction Enlargement and Ion Channel Remodelling in the Rabbit The potential role of Kv4.3 K+ channel in heart hypertrophy.Kcne4 deletion sex- and age-specifically impairs cardiac repolarization in mice.KChIP2 genotype dependence of transient outward current (Ito) properties in cardiomyocytes isolated from male and female mice.KChIP2 regulates the cardiac Ca2+ transient and myocyte contractility by targeting ryanodine receptor activity.Myocardial KChIP2 Expression in Guinea Pig Resolves an Expanded Electrophysiologic Role Reductions in the Cardiac Transient Outward K+ Current Ito Caused by Chronic β-Adrenergic Receptor Stimulation Are Partly Rescued by Inhibition of Nuclear Factor κB.Molecular Basis of Functional Myocardial Potassium Channel Diversity.Potassium Channel Interacting Protein 2 (KChIP2) is not a transcriptional regulator of cardiac electrical remodeling.KChIP2 is a core transcriptional regulator of cardiac excitability.Mouse models of arrhythmogenic cardiovascular disease: challenges and opportunities.Transient outward potassium channel: a heart failure mediator.Transmural gradients in ion channel and auxiliary subunit expression.A novel computational model of mouse myocyte electrophysiology to assess the synergy between Na+ loading and CaMKII.New insights into the complex effects of KChIP2 on calcium transients Loss of K+ currents in heart failure is accentuated in KChIP2 deficient mice.Development of heart failure is independent of K+ channel-interacting protein 2 expression.Notch-Mediated Epigenetic Regulation of Voltage-Gated Potassium Currents.Arrhythmia: 100 years on from George Ralph Mines.Notch signaling modulates the electrical behavior of cardiomyocytes.Differential Expression and Remodeling of Transient Outward Potassium Currents in Human Left Ventricles.Early remodeling of repolarizing K+ currents in the αMHC403/+ mouse model of familial hypertrophic cardiomyopathy.Circadian rhythm in QT interval is preserved in mice deficient of potassium channel interacting protein 2.Ventricular repolarization time, location of pacing stimulus and current pulse amplitude conspire to determine arrhythmogenicity in mice.

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P2860

Stabilization of Kv4 protein by the accessory K(+) channel interacting protein 2 (KChIP2) subunit is required for the generation of native myocardial fast transient outward K(+) currents.

http://www.wikidata.org/entity/Q45146704

description

2013 nî lūn-bûn

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2013年の論文

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2013年学术文章

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2013年学术文章

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2013年学术文章

@zh-cn

2013年学术文章

@zh-hans

2013年学术文章

@zh-my

2013年学术文章

@zh-sg

2013年學術文章

@yue

2013年學術文章

@zh-hant

name

Stabilization of Kv4 protein by the accessory K

@nl

Stabilization of Kv4 protein b ...... ansient outward K(+) currents.

@en

type

label

Stabilization of Kv4 protein by the accessory K

@nl

Stabilization of Kv4 protein b ...... ansient outward K(+) currents.

@en

prefLabel

Stabilization of Kv4 protein by the accessory K

@nl

Stabilization of Kv4 protein b ...... ansient outward K(+) currents.

@en

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JPHYSIOL.2013.255836

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The Journal of Physiology

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Stabilization of Kv4 protein b ...... ansient outward K(+) currents.

@en

P2093

Jeanne M Nerbonne

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Nicholas C Foeger

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Rebecca L Mellor

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Wei Wang

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P2860

Modulation of A-type potassium channels by a family of calcium sensors

Localization and enhanced current density of the Kv4.2 potassium channel by interaction with the actin-binding protein filamin

Conserved Kv4 N-terminal domain critical for effects of Kv channel-interacting protein 2.2 on channel expression and gating

Structural basis for modulation of Kv4 K+ channels by auxiliary KChIP subunits

Expression and function of dipeptidyl-aminopeptidase-like protein 6 as a putative beta-subunit of human cardiac transient outward current encoded by Kv4.3

Three-dimensional structure of the KChIP1-Kv4.3 T1 complex reveals a cross-shaped octamer

Traffic of Kv4 K+ channels mediated by KChIP1 is via a novel post-ER vesicular pathway

Improved monomeric red, orange and yellow fluorescent proteins derived from Discosoma sp. red fluorescent protein

A fundamental role for KChIPs in determining the molecular properties and trafficking of Kv4.2 potassium channels

A defect in the Kv channel-interacting protein 2 (KChIP2) gene leads to a complete loss of I(to) and confers susceptibility to ventricular tachycardia

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4149-4166

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scholarly article

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10.1113/JPHYSIOL.2013.255836

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3779109

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