Murid herpesvirus 4 strain 68 M2 protein is a B-cell-associated antigen important for latency but not lymphocytosis.
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Role of Src homology domain binding in signaling complexes assembled by the murid γ-herpesvirus M2 protein.The Gammaherpesvirus m2 protein manipulates the Fyn/Vav pathway through a multidocking mechanism of assemblyThe MHV68 M2 protein drives IL-10 dependent B cell proliferation and differentiationA single CD8+ T cell epitope sets the long-term latent load of a murid herpesvirusGammaherpesvirus latency induces antibody-associated thrombocytopenia in mice.Virus-encoded microRNAs facilitate gammaherpesvirus latency and pathogenesis in vivoThe murine gammaherpesvirus 68 M2 gene is required for efficient reactivation from latently infected B cells.Characterization of a novel wood mouse virus related to murid herpesvirus 4.Identification of viral genes essential for replication of murine gamma-herpesvirus 68 using signature-tagged mutagenesis.Global mRNA degradation during lytic gammaherpesvirus infection contributes to establishment of viral latency.Epstein-Barr virus IL-10 gene expression by a recombinant murine gammaherpesvirus in vivo enhances acute pathogenicity but does not affect latency or reactivation.Construction and characterization of an infectious murine gammaherpesivrus-68 bacterial artificial chromosome.Activation of Vav by the gammaherpesvirus M2 protein contributes to the establishment of viral latency in B lymphocytesChemokine binding protein M3 of murine gammaherpesvirus 68 modulates the host response to infection in a natural hostType I Interferons Direct Gammaherpesvirus Host ColonizationZinc finger antiviral protein inhibits murine gammaherpesvirus 68 M2 expression and regulates viral latency in cultured cellsSystematic mutagenesis of the murine gammaherpesvirus 68 M2 protein identifies domains important for chronic infection.A gammaherpesvirus-secreted activator of Vbeta4+ CD8+ T cells regulates chronic infection and immunopathology.Expression in a recombinant murid herpesvirus 4 reveals the in vivo transforming potential of the K1 open reading frame of Kaposi's sarcoma-associated herpesvirusProspects of a novel vaccination strategy for human gamma-herpesviruses.Murid Gammaherpesvirus Latency-Associated Protein M2 Promotes the Formation of Conjugates between Transformed B Lymphoma Cells and T Helper Cells.CTCF and Sp1 interact with the Murine gammaherpesvirus 68 internal repeat elements.Blimp-1-dependent plasma cell differentiation is required for efficient maintenance of murine gammaherpesvirus latency and antiviral antibody responses.Inhibition of interferon-mediated antiviral activity by murine gammaherpesvirus 68 latency-associated M2 protein.CD4 T cells specific for a latency-associated γ-herpesvirus epitope are polyfunctional and cytotoxic.
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P2860
Murid herpesvirus 4 strain 68 M2 protein is a B-cell-associated antigen important for latency but not lymphocytosis.
description
2003 nî lūn-bûn
@nan
2003年の論文
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2003年論文
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2003年論文
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2003年論文
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2003年論文
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2003年論文
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name
Murid herpesvirus 4 strain 68 ...... latency but not lymphocytosis.
@en
Murid herpesvirus 4 strain 68 ...... latency but not lymphocytosis.
@nl
type
label
Murid herpesvirus 4 strain 68 ...... latency but not lymphocytosis.
@en
Murid herpesvirus 4 strain 68 ...... latency but not lymphocytosis.
@nl
prefLabel
Murid herpesvirus 4 strain 68 ...... latency but not lymphocytosis.
@en
Murid herpesvirus 4 strain 68 ...... latency but not lymphocytosis.
@nl
P2093
P2860
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P1476
Murid herpesvirus 4 strain 68 ...... latency but not lymphocytosis.
@en
P2093
Alastair I Macrae
Anthony A Nash
David L Woodland
Edward J Usherwood
Emilio Flaño
In-Jeong Kim
Jeffery T Sample
Marcia A Blackman
S Mazher Husain
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P304
P356
10.1128/JVI.77.17.9700-9709.2003
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P577
2003-09-01T00:00:00Z