C5a, but not C3a, increases VEGF secretion in ARPE-19 human retinal pigment epithelial cells. - Wikidata - awgldk - TriplyDB

C5a, but not C3a, increases VEGF secretion in ARPE-19 human retinal pigment epithelial cells.

C5a, but not C3a, increases VEGF secretion in ARPE-19 human retinal pigment epithelial cells.

http://www.wikidata.org/entity/Q39890415

about

Complement activation in the context of stem cells and tissue repair Complement System Part II: Role in Immunity Anaphylatoxins Activate Ca2+, Akt/PI3-Kinase, and FOXO1/FoxP3 in the Retinal Pigment Epithelium Kallistatin inhibits vascular inflammation by antagonizing tumor necrosis factor-alpha-induced nuclear factor kappaB activation Complement gene expression and regulation in mouse retina and retinal pigment epithelium/choroid.Intravitreal inhibition of complement C5a reduces choroidal neovascularization in mice.Connecting the innate and adaptive immune responses in mouse choroidal neovascularization via the anaphylatoxin C5a and γδT-cells.Retinal pigment epithelial cell death by the alternative complement cascade: role of membrane regulatory proteins, calcium, PKC, and oxidative stress.C5a and toll-like receptor 4 crosstalk in retinal pigment epithelial cells.Immunological effects and therapeutic role of C5a in cancer.Anti-complement component 5 antibody targeting MG4 domain inhibits choroidal neovascularization.Genetic and pharmacologic inhibition of complement impairs endothelial cell function and ablates ovarian cancer neovascularization.Genetic Investigation of Complement Pathway Genes in Type 2 Diabetic Retinopathy: An Inflammatory Perspective CFH Y402H polymorphism and the complement activation product C5a: effects on NF-κB activation and inflammasome gene regulation C3a Increases VEGF and Decreases PEDF mRNA Levels in Human Retinal Pigment Epithelial Cells Sub-lytic C5b-9 induces functional changes in retinal pigment epithelial cells consistent with age-related macular degeneration.Complement Component C5a Primes Retinal Pigment Epithelial Cells for Inflammasome Activation by Lipofuscin-mediated Photooxidative Damage Differential activation mechanisms of serum C5a in lupus nephritis and neuropsychiatric systemic lupus erythematosus.Effect of Anti-C5a Therapy in a Murine Model of Early/Intermediate Dry Age-Related Macular Degeneration.Correlation of complement fragment C5a with inflammatory cytokines in the vitreous of patients with proliferative diabetic retinopathy.The complement system in glioblastoma multiforme

P2860

Q26782203-8D75D7F9-F37C-44C6-B4B9-03BBA3E329FC Q28263460-B6FF613D-A493-46EF-AA02-CA6739336232 Q33802700-B2951353-6AE1-46A4-B50E-3C7E435D68A3 Q34070667-12064F09-6E94-4874-A510-18D46D23A7AC Q35068037-20DAE54C-C8C9-4208-B798-ACCA253E210C Q35633511-A2742228-9AEC-4912-B73C-97EB4C6CE965 Q35974464-F2715F0C-1B1D-4DDA-AA88-8EF31D738FB2 Q36089577-77CBC180-2CCE-4F80-9D23-A8CD2A219674 Q36107219-8E62A0F2-2ADC-4BD2-B8D0-54E7B04FD17D Q36260122-C31A9E84-C48D-451D-8E39-2E5E5A562AAF Q36364687-DFFE13E5-9479-49B6-88F8-933BB1A15D66 Q36446437-3B4A97B3-0958-4CAE-A65C-812E66AC5103 Q36632944-4508C738-ED84-4C27-9496-1E009D88F1A9 Q37088454-0D5B80EE-A8EC-4019-82D0-8E5043B24D5F Q37322563-3D6D7149-6A30-40AB-BA80-C1D5C9E0BAFB Q38543289-307EE87B-BB99-49D2-8393-13B1ACF14C28 Q38819720-39A8F2D2-6FD5-4031-8B06-69BEB7CD3D2F Q42102188-129305D4-8D52-421F-9860-BBC2CF86A36A Q49223224-085AB0A5-A63D-4ACA-8A45-3D5F5414ECF3 Q51376222-26506B7F-1A3E-490E-ABE8-E16E41217782 Q58748412-475C0AF3-BF72-4A90-A15C-A71011CBEF10

P2860

C5a, but not C3a, increases VEGF secretion in ARPE-19 human retinal pigment epithelial cells.

http://www.wikidata.org/entity/Q39890415

description

2009 nî lūn-bûn

@nan

2009年の論文

@ja

2009年学术文章

@wuu

2009年学术文章

@zh-cn

2009年学术文章

@zh-hans

2009年学术文章

@zh-my

2009年学术文章

@zh-sg

2009年學術文章

@yue

2009年學術文章

@zh

2009年學術文章

@zh-hant

name

C5a, but not C3a, increases VE ...... inal pigment epithelial cells.

@en

C5a, but not C3a, increases VE ...... inal pigment epithelial cells.

@nl

type

label

C5a, but not C3a, increases VE ...... inal pigment epithelial cells.

@en

C5a, but not C3a, increases VE ...... inal pigment epithelial cells.

@nl

prefLabel

C5a, but not C3a, increases VE ...... inal pigment epithelial cells.

@en

C5a, but not C3a, increases VE ...... inal pigment epithelial cells.

@nl

P1433

Q39890415-FC1F61FD-271C-4B6F-8B6F-D96259A11944

P1476

Q39890415-A35B545F-B6E0-4422-BDD5-0BDD7192B485

P2093

Q39890415-03307BBF-11FA-4AAC-8AE4-4B60DE6AE4C1

Q39890415-07A7E3B7-0D59-4702-A65B-1FA79B3C683B

Q39890415-311E4DBD-84ED-4A52-8505-7A057AE23405

Q39890415-8BB52C22-DC5C-4A72-B388-DD31BCD257D4

Q39890415-BDBFD3F8-5868-47E3-BDAD-7FB009800C79

P2860

Q39890415-07B755CE-DA69-4EF6-AA58-1E89885BF61A

Q39890415-13AB4832-EAFD-482C-9360-B6FD54941251

Q39890415-1C494A8F-D409-4AC6-8705-0E4EBE8C00E6

Q39890415-2C62D583-25E6-4656-9714-C18B21F4FC1F

Q39890415-4587054D-C0BE-4C92-B1EA-BE7129F1411E

Q39890415-534924B5-8B04-488A-BE0B-1D086EAFD2DD

Q39890415-563A17D3-5670-43D3-B3D3-1BB00C53E781

Q39890415-5FC817A8-BF18-49EA-893D-6877C5EE6184

Q39890415-7B9903B4-746A-4D80-A5E1-E3400DF460D7

Q39890415-83837F3D-68A5-4F5A-92AF-56E516F8FB82

P304

Q39890415-CF91D886-55D7-46FD-B802-B5BFB18BF5FA

P31

Q39890415-67845C2E-9651-4C61-B04D-ADF67EC69535

P356

Q39890415-6B6932FB-C290-419C-9DF7-7B98A3DB17B9

P433

Q39890415-B1E5A93F-B394-41AE-9EBB-6127667CA63B

P478

Q39890415-2637CAE8-C93B-47D5-949C-6F91B76991F5

P577

Q39890415-DE2FEBA9-93C1-4010-BB10-9207324D16C6

P698

Q39890415-491E9265-F878-4333-BF91-2BBB3DBDE779

P356

02713680802546658

P1433

Current Eye Research

P1476

C5a, but not C3a, increases VE ...... inal pigment epithelial cells.

@en

P2093

Bertrand L Chenard

http://www.w3.org/2001/XMLSchema#string

Daniel N Cortright

http://www.w3.org/2001/XMLSchema#string

James E Krause

http://www.w3.org/2001/XMLSchema#string

Robin Meade

http://www.w3.org/2001/XMLSchema#string

Stephen M Waters

http://www.w3.org/2001/XMLSchema#string

P2860

An animal model of age-related macular degeneration in senescent Ccl-2- or Ccr-2-deficient mice

Drusen complement components C3a and C5a promote choroidal neovascularization

Molecular cloning and characterization of the human anaphylatoxin C3a receptor

A common haplotype in the complement regulatory gene factor H (HF1/CFH) predisposes individuals to age-related macular degeneration

Function, structure and therapeutic potential of complement C5a receptors

Extended haplotypes in the complement factor H (CFH) and CFH-related (CFHR) family of genes protect against age-related macular degeneration: characterization, ethnic distribution and evolutionary implications

C5L2, a nonsignaling C5A binding protein

Corneal avascularity is due to soluble VEGF receptor-1

Inherited complement regulatory protein deficiency predisposes to human disease in acute injury and chronic inflammatory statesthe examples of vascular damage in atypical hemolytic uremic syndrome and debris accumulation in age-related macular degen

Anaphylatoxins and infectious and non-infectious inflammatory diseases.

P304

57-61

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1080/02713680802546658

http://www.w3.org/2001/XMLSchema#string

P433

1

http://www.w3.org/2001/XMLSchema#string

P478

34

http://www.w3.org/2001/XMLSchema#string

P577

2009-01-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

19172471

http://www.w3.org/2001/XMLSchema#string