Activation of cardiac Cdk9 represses PGC-1 and confers a predisposition to heart failure.
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Inhibition of Tat activity by the HEXIM1 proteinA human splicing factor, SKIP, associates with P-TEFb and enhances transcription elongation by HIV-1 Tat.PGC-1 coactivators: inducible regulators of energy metabolism in health and diseaseMonoamine oxidases (MAO) in the pathogenesis of heart failure and ischemia/reperfusion injuryRegulation of P-TEFb elongation complex activity by CDK9 acetylationThioredoxin and ventricular remodelingNew insights into PGC-1 coactivators: redefining their role in the regulation of mitochondrial function and beyondThe breast cell growth inhibitor, estrogen down regulated gene 1, modulates a novel functional interaction between estrogen receptor alpha and transcriptional elongation factor cyclin T1Transcription-dependent association of multiple positive transcription elongation factor units to a HEXIM multimerCyclin-dependent kinase-9 is a component of the p300/GATA4 complex required for phenylephrine-induced hypertrophy in cardiomyocytesA PGC-1alpha-O-GlcNAc transferase complex regulates FoxO transcription factor activity in response to glucose.The PPARGC1A Gly482Ser polymorphism is associated with left ventricular diastolic dysfunction in menRescue of cardiomyopathy in peroxisome proliferator-activated receptor-alpha transgenic mice by deletion of lipoprotein lipase identifies sources of cardiac lipids and peroxisome proliferator-activated receptor-alpha activatorsToward transcriptional therapies for the failing heart: chemical screens to modulate genesControlling cellular P-TEFb activity by the HIV-1 transcriptional transactivator TatCardiac tissue-specific repression of CELF activity disrupts alternative splicing and causes cardiomyopathy.Mutation of the HEXIM1 gene results in defects during heart and vascular development partly through downregulation of vascular endothelial growth factor.Mediator complex dependent regulation of cardiac development and disease.Transverse aortic constriction leads to accelerated heart failure in mice lacking PPAR-gamma coactivator 1alpha.Brain derived neurotrophic factor contributes to the cardiogenic potential of adult resident progenitor cells in failing murine heart.Hexim1 sequesters positive transcription elongation factor b from the class II transactivator on MHC class II promoters.CDK9 and its repressor LARP7 modulate cardiomyocyte proliferation and response to injury in the zebrafish heartCardiac lineage protein-1 (CLP-1) regulates cardiac remodeling via transcriptional modulation of diverse hypertrophic and fibrotic responses and angiotensin II-transforming growth factor β (TGF-β1) signaling axis.Transcriptional control of cardiac fuel metabolism and mitochondrial functionDnmt2/Trdmt1 as Mediator of RNA Polymerase II Transcriptional Activity in Cardiac Growth.Mitochondrial energy metabolism in heart failure: a question of balance.Increased cyclin T1 expression as a favorable prognostic factor in treating gastric adenocarcinoma.HEXIM1 controls satellite cell expansion after injury to regulate skeletal muscle regenerationThe PPARalpha-PGC-1alpha Axis Controls Cardiac Energy Metabolism in Healthy and Diseased Myocardium.Gene expression-based screening identifies microtubule inhibitors as inducers of PGC-1alpha and oxidative phosphorylation.The transcriptional coactivator PGC-1alpha is essential for maximal and efficient cardiac mitochondrial fatty acid oxidation and lipid homeostasisInducible re-expression of HEXIM1 causes physiological cardiac hypertrophy in the adult mousePositive cyclin T expression as a favorable prognostic factor in treating gastric gastrointestinal stromal tumors.Targeting PGC-1 alpha to control energy homeostasis.Telomeres and mitochondria in the aging heart.Down-regulation of cardiac lineage protein (CLP-1) expression in CLP-1 +/- mice affords.Energy metabolism in heart failure and remodelling.Apoptotic and non-apoptotic programmed cardiomyocyte death in ventricular remodelling.Positive transcription elongation factor b activity in compensatory myocardial hypertrophy is regulated by cardiac lineage protein-1.Pivotal role of cardiac lineage protein-1 (CLP-1) in transcriptional elongation factor P-TEFb complex formation in cardiac hypertrophy.
P2860
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P2860
Activation of cardiac Cdk9 represses PGC-1 and confers a predisposition to heart failure.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh
2004年學術文章
@zh-hant
name
Activation of cardiac Cdk9 represses PGC-1 and confers a predisposition to heart failure.
@en
Activation of cardiac Cdk9 represses PGC-1 and confers a predisposition to heart failure.
@nl
type
label
Activation of cardiac Cdk9 represses PGC-1 and confers a predisposition to heart failure.
@en
Activation of cardiac Cdk9 represses PGC-1 and confers a predisposition to heart failure.
@nl
prefLabel
Activation of cardiac Cdk9 represses PGC-1 and confers a predisposition to heart failure.
@en
Activation of cardiac Cdk9 represses PGC-1 and confers a predisposition to heart failure.
@nl
P2093
P2860
P356
P1433
P1476
Activation of cardiac Cdk9 represses PGC-1 and confers a predisposition to heart failure.
@en
P2093
Fernando Scaglia
George E Taffet
Keith A Youker
Lloyd H Michael
Manabu Shirai
Michael D Schneider
Motoaki Sano
Philip M Barger
Prathit A Kulkarni
P2860
P304
P356
10.1038/SJ.EMBOJ.7600351
P407
P577
2004-08-05T00:00:00Z